ライフサイエンスコーパス: volume-controlled

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1 oved short-term survival after pressure- and volume-controlled bleeding.

2 ring 20-minute frequency (0.25 Hz) and tidal volume-controlled breathing after intravenous injections

3 rough 20-min periods of frequency- and tidal volume-controlled breathing.

4 donor animal prior to isolation, and chamber volume controlled by a servo-pump.

5 ation of 10 and 40 ppm to a test lung during volume-controlled (constant flow) and pressure-controlle

6 iated gas exchange with perflubron (n=10) or volume controlled continuous positive pressure breathing

7 The rats were subjected to volume-controlled hemorrhage (2.5 mL/100 g) followed by

8 al route improves survival time after severe volume-controlled hemorrhagic shock in rats without wors

9 A three-phase, volume-controlled hemorrhagic shock model was used: hemo

10 rhagic shock models and partially realistic, volume-controlled hemorrhagic shock models to more reali

11 Patients received volume-controlled mechanical ventilation according to th

12 xpiratory pressure of 1 and 10 cm H2O, under volume-controlled mechanical ventilation in the settings

13 After injury, all animals were placed on volume-controlled mechanical ventilation to achieve PaO2

14 Tracheostomy, volume-controlled mechanical ventilation, and 72 hrs of

15 Following randomization, the volume controlled positive pressure breathing group deve

16 Only two (25%) of the eight animals in the volume controlled positive pressure breathing group were

17 ereas it remained unchanged over time in the volume controlled positive pressure breathing group.

18 Animals that received volume controlled positive pressure breathing remained h

19 IEPOX uptake by pure sulfate particles is a volume-controlled process, which results in particles wi

20 ressure-controlled ventilation compared with volume-controlled ventilation (nitric oxide concentratio

21 ciency of ventilation would be greatest with volume-controlled ventilation (VCV) compared with pressu

22 GI can be used either with pressure (PCV) or volume-controlled ventilation and continuously or only d

23 ndrome, E-cadherin expression was similar in volume-controlled ventilation and variable ventilation;

24 layed was minimally altered by helium during volume-controlled ventilation but substantially decrease

25 rin expression in lung tissue was reduced in volume-controlled ventilation compared with nonventilate

26 ion and reduced lung elastance compared with volume-controlled ventilation in both acute respiratory

27 ry acute respiratory distress syndrome, only volume-controlled ventilation increased vascular cell ad

28 rdiac arrest using an original pressure- and volume-controlled ventilation strategy in rabbits.

29 After injury, the animals were placed on volume-controlled ventilation to achieve PaO2 >60 mm Hg

30 ncreased relative angiopoietin-1 expression (volume-controlled ventilation, 0.3 [0.2-0.5] vs variable

31 veolar damage (median [interquartile range]: volume-controlled ventilation, 12 [11-17] vs variable ve

32 1), and angiopoietin-2/angiopoietin-1 ratio (volume-controlled ventilation, 2.0 [1.3-2.1] vs variable

33 [8-10]; p < 0.01), interleukin-6 expression (volume-controlled ventilation, 21.5 [18.3-23.3] vs varia

34 dhesion molecule-1 messenger RNA expression (volume-controlled ventilation, 7.7 [5.7-18.6] vs nonvent

35 e randomly assigned to receive conventional (volume-controlled ventilation, n = 6) or variable ventil

36 During volume-controlled ventilation, V(T) delivered was substa

37 t was higher in variable ventilation than in volume-controlled ventilation.

38 e-controlled ventilation: 14.5 to 130.5 ppm; volume-controlled ventilation: 21.6 to 104.7 ppm; nitric

39 ure-controlled ventilation: 3.2 to 30.9 ppm; volume-controlled ventilation: 4.5 to 27.1 ppm).

40 omy was performed, sheep were connected to a volume-controlled ventilator.

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