ライフサイエンスコーパス: voluntary movement

1 omuscular junction (NMJ) is critical for all voluntary movement.

2 sia and studied them both at rest and during voluntary movement.

3 not gate the central mechanism that produces voluntary movement.

4 lated oscillations are minimally affected by voluntary movement.

5 and another that was linked to onset of the voluntary movement.

6 cantly influencing locomotion and control of voluntary movement.

7 ts in the initiation, speed, and fluidity of voluntary movement.

8 that include cognition, reward learning, and voluntary movement.

9 is associated with and explains a change in voluntary movement.

10 striatal pathway to the striatum to regulate voluntary movement.

11 the normal sense of agency that accompanies voluntary movement.

12 ole of KMT2B in the physiological control of voluntary movement.

13 ng energy expenditure, in part by increasing voluntary movement.

14 al communication, seizures, and disorders of voluntary movement.

15 spinal cord circuits that are essential for voluntary movement.

16 ctions but is attenuated prior to and during voluntary movement.

17 as of the human medial frontal lobe prior to voluntary movement.

18 models has emerged as a leading paradigm for voluntary movement.

19 in healthy subjects and show that this slows voluntary movement.

20 he involuntary movement and during a matched voluntary movement.

21 ctions and is suppressed prior to and during voluntary movement.

22 sts modestly improved the range and speed of voluntary movement.

23 motor commands from the motor cortex during voluntary movement.

24 ty of the nervous system to plan and control voluntary movement.

25 n of primary motor cortex (MI) is to control voluntary movements.

26 movements triggered or exacerbated by sudden voluntary movements.

27 spatial representations in conjunction with voluntary movements.

28 mportantly to the muscle activity underlying voluntary movements.

29 es the direction, velocity, and amplitude of voluntary movements.

30 including locomotion, postural control, and voluntary movements.

31 n Purkinje cells and regions that coordinate voluntary movements.

32 ction and with reductions in the velocity of voluntary movements.

33 viors ranging from basic reflexes to complex voluntary movements.

34 tion of postural adjustments associated with voluntary movements.

35 estigate their role in the online control of voluntary movements.

36 insights into the adaptive control of other voluntary movements.

37 ng performance of stereotypic locomotion and voluntary movement: adaptive locomotion over obstacles a

38 nsory incomplete spinal cord injury regained voluntary movement after 7 months of epidural stimulatio

39 Voluntary movement also caused increased gamma band acti

40 the basic assumption that spasticity impairs voluntary movement and a review of the methodology of th

41 eus (STN) neurons are observed during normal voluntary movement and abnormal movement in Parkinson's

42 rone syndrome, reduce spasticity and improve voluntary movement and active function.

43 f a wide range of brain functions, including voluntary movement and behavioral processes.

44 regulate diverse brain functions, including voluntary movement and cognitive and emotive behaviors.

45 njugated fatty acids on non-exercise form of voluntary movement and lipid and glucose metabolisms for

46 The effects of involuntary, passive and voluntary movement and muscle-tendon vibration on freque

47 neurons could contribute to the decrease in voluntary movement and other dopamine-mediated behaviors

48 elucidated their contribution to generating voluntary movement and revealed their involvement in mov

49 esodiencephalic dopaminergic neurons control voluntary movement and reward based behaviours.

50 Voluntary movement and the heart beat require this calci

51 the sensations generated as a consequence of voluntary movement and those arising from events in the

52 by transcranial magnetic stimulation, rapid voluntary movements and a fatiguing exercise test of a s

53 ng motor restlessness and chorea, slowing of voluntary movements and cognitive impairment.

54 ansmission in climbing fibre pathways during voluntary movements and motor learning.

55 the effects of ipsilateral misprojections on voluntary movements and stereotypic locomotion.

56 e learning, through repeated cooccurrence of voluntary movements and their outcomes.

57 y tasks, such as reward-related behavior and voluntary movement, and excessive loss of these neurons

58 ons play a central role in the regulation of voluntary movement, and their degeneration is associated

59 of substantia nigra in the midbrain control voluntary movement, and their degeneration is the cause

60 Voluntary movements are frequently composed of several a

61 Ns) play a central role in the regulation of voluntary movement as well as other complex behaviors, a

62 scious awareness, so that subjects perceived voluntary movements as occurring later and their sensory

63 nergic neurons are essential for appropriate voluntary movement, as epitomized by the cardinal motor

64 ated during the preparation and execution of voluntary movements at both cortical and subcortical lev

65 cord can be modulated presynaptically during voluntary movement by mechanisms that depolarize afferen

66 emisphere) in the process of generation of a voluntary movement by the paretic hand in patients with

67 emisphere) in the process of generation of a voluntary movement by the paretic hand.

68 hemisphere) in the process of generation of voluntary movements by the paretic hand, a disorder corr

69 Mammalian motor circuits control voluntary movements by transmitting signals from the cen

70 Therefore, each voluntary movement carries a cost because its duration d

71 ALS patients exhibit alterations of voluntary movements caused by degeneration of motor neur

72 The control of voluntary movement changes markedly with age.

73 bstantia nigra (SN) neurons is essential for voluntary movement control.

74 del parameter averaging we found that during voluntary movements, DBS reversed the effective connecti

75 Voluntary movement difficulties in Parkinson's disease a

76 explored the pathophysiological basis of the voluntary movement disorder, in particular the role of t

77 to that observed during the performance of a voluntary movement during wakefulness.

78 ystonia, generalized athetosis, and impaired voluntary movement in all patients.

79 egy that can dramatically affect recovery of voluntary movement in individuals with complete paralysi

80 nding pathway contributing to the control of voluntary movement in mammals.

81 e neurotransmitter dopamine, which regulates voluntary movement in many organisms, can stimulate move

82 A basic EEG feature upon voluntary movements in healthy human subjects is a beta

83 olved in the preparation of externally paced voluntary movements in humans and, secondly, the degree

84 ts that disturbed proprioceptive guidance of voluntary movements in Parkinson's disease is related to

85 , posture, gait and also coordinates skilled voluntary movements including eye movements.

86 elop normally for 6-18 months, but then lose voluntary movements, including speech and hand skills.

87 Because brain function is central to voluntary movement, interventions that aim to improve mo

88 s likely reflect requirements for control of voluntary movement involving different body parts.

89 d, to help guide and improve the accuracy of voluntary movements involving motion of the head in spac

90 Voluntary movement is a result of signals transmitted th

91 Voluntary movement is accompanied by changes in the degr

92 Human voluntary movement is controlled by the pyramidal motor

93 trate that a fundamental component of normal voluntary movement is impaired in patients with function

94 ement, their involvement in externally paced voluntary movement is less clear.

95 Executive control of voluntary movements is a hallmark of the mammalian brain

96 raction of agonist/antagonist muscles during voluntary movement observed in subjects with spastic cer

97 ver, we now report in three more individuals voluntary movement occurred with epidural stimulation im

98 CBF were seen in the contralateral M1 during voluntary movement of either hand; a small ipsilateral M

99 r information plays a role in the control of voluntary movement of the upper body.

100 s pallidus interna (GPi) or scalp EEG during voluntary movements of a hand-held joystick in six awake

101 ge lesions of the primary motor cortex (M1), voluntary movements of affected body parts are weak and

102 brain activity to vibratory stimulation and voluntary movements of body parts above and below the le

103 As in voluntary movements, only a single rhythm may be easily

104 Tics are sometimes described as voluntary movements performed in an automatic or habitua

105 The application of TMS(CT) prior to control voluntary movements produced a significant delay in move

106 , involuntary movements and abnormalities of voluntary movement, psychiatric symptoms, weight loss, d

107 , involuntary movements and abnormalities of voluntary movement, psychiatric symptoms, weight loss, d

108 th healthy subjects even when suppression of voluntary movement rather than tics was required during

109 During normal voluntary movements, re-afferent sensory input continuou

110 Production of voluntary movements relies critically on the functional

111 behaviors important for survival, including voluntary movement, reward processing, and detection of

112 To test our idea, we considered elementary voluntary movements: saccades of the eye.

113 During voluntary movement, sensory activity may be globally sup

114 The voluntary movement showed activation of the putamen wher

115 movements are worsened by anxiety but not by voluntary movement, startle, caffeine, or alcohol.

116 the corticospinal tract (CST) in controlling voluntary movements, successful regeneration of large nu

117 The control of voluntary movements such as skillful reaching and graspi

118 ies in monkeys have shown that parameters of voluntary movement (such as direction) may be specified

119 When the stimulus began 2 s before the voluntary movement the response consisted of two distinc

120 In a voluntary movement, the nervous system specifies not onl

121 cal-basal ganglia network after cessation of voluntary movement: the post-movement beta synchronizati

122 fficiently for the planning and execution of voluntary movements, thereby resulting in direction-depe

123 the conditional knock-outs showed bilateral voluntary movements under conditions when single limb mo

124 The motor commands required to control voluntary movements under various environmental conditio

125 ortex plays a central role in the control of voluntary movements, which are typically guided by senso