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1 ty (three), mild FXI deficiency (four), mild von Willebrand's disease and FXI deficiency (one), combi
2                             The frequency of von Willebrand's disease and FXI deficiency were 13% (95
3  investigated for these disorders-especially von Willebrand's disease-before invasive procedures are
4 s disease and FXI deficiency (one), combined von Willebrand's disease, FXI deficiency, and factor X d
5 ctor IX (haemophilia B) are well recognised, von Willebrand's disease is much more common.
6 sed in 26 (17%) patients: the disorders were von Willebrand's disease of mild (15) or moderate severi
7 for delta-SPD; it was not observed in either von Willebrand's disease or thrombasthenia.
8 rder compared with 13 (65%) of 20 women with von Willebrand's disease (p=0.001) and four (66.7%) of s
9 s in the naturally occurring variant form of von Willebrand's disease-type 2M underscoring the physio
10       Mutations causing types 2B, 2M, and 2A von Willebrand's disease (vWD) are located in the A1 and
11                                              von Willebrand's disease (vWD) arises from abnormalities

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