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1 /MAPK by the inductive signal that initiates vulval development.
2 dentify attenuators of RAS signalling during vulval development.
3 s fos-1 has been shown to act in uterine and vulval development.
4  required for neuronal migrations and during vulval development.
5 of let-7 results in irregular hypodermal and vulval development.
6  at the O. tipulae cov-3 locus, which affect vulval development.
7 lin-3, which encodes the signal that induces vulval development.
8 erus is not due to precocious or accelerated vulval development.
9 parallel to LIN-17/Frizzled (Fz) during worm vulval development.
10 as signaling pathway that induces C. elegans vulval development.
11 mote cell proliferation at multiple steps of vulval development.
12 gonad, the critical organizer of uterine and vulval development.
13 onstrate multiple functions of lin-11 during vulval development.
14 iptional targets of intercellular signals in vulval development.
15 scription in the AC and thus are involved in vulval development.
16 he AC must be precisely regulated for proper vulval development.
17 c postembryonic defects in gonadogenesis and vulval development.
18 /Notch pathway during Caenorhabditis elegans vulval development.
19 e role of Rac signaling during each phase of vulval development.
20 thways in specific cellular processes during vulval development.
21 ssion to antagonize RTK/Ras signaling during vulval development.
22 hat a Wnt signaling pathway is acting during vulval development.
23 ific to the role of LIN-35 in intestinal and vulval development.
24 g-14, egl-18, and sem-4 also function during vulval development.
25 egans, starting with 27 proteins involved in vulval development.
26 Caenorhabditis elegans protein essential for vulval development.
27 riation in the role of the gonad in nematode vulval development.
28 diated signal transduction during C. elegans vulval development.
29 olutely required for Ras signaling to induce vulval development.
30 ion during cell fate decisions important for vulval development.
31  growth factor receptor (EGFR) necessary for vulval development.
32 in-3 EGF to negatively regulate Ras-mediated vulval development.
33  obtain cell-cycle statistics for C. elegans vulval development, a paradigm for organogenesis.
34 legans ROR homolog, cam-1, during C. elegans vulval development, a Wnt-regulated process.
35                During Caenorhabditis elegans vulval development, activation of receptor tyrosine kina
36 ulval-uterine coordination due to precocious vulval development also leads to uv1 cell differentiatio
37 ene, which plays a role in gonadogenesis and vulval development and encodes a Notch ligand, is derepr
38 rstanding of how these genes function during vulval development and have also identified roles for th
39     The hda-1(cw2) mutation causes defective vulval development and reduced fertility, like hda-1(e17
40 ated MAGUK in Caenorhabditis elegans, blocks vulval development, and mutation of the postsynaptic den
41 ns as heterochronic regulators of larval and vulval development but have recently been implicated in
42  NuRD-like complex and HP1 homologs regulate vulval development, but no HMT is known to act in this p
43                       Caenorhabditis elegans vulval development culminates during exit from the L4-to
44 d to establish patterns of cell fates during vulval development have also been conserved.
45                        Studies of C. elegans vulval development have illuminated mechanisms underlyin
46 ur results suggest that in the inhibition of vulval development, homologs of SETDB1, HP1 and the NuRD
47                                    Analyzing vulval development in a let-341 null mutant, we find an
48                                 Ras-mediated vulval development in C. elegans is inhibited by the fun
49 s (VPCs) by LIN-3/EGF from the gonad induces vulval development in C. elegans.
50      To illustrate, a geometric model fit to vulval development in Caenorhabditis elegans, implies a
51                                       During vulval development in Caenorhabditis elegans, six precur
52 al for viability, seam cell development, and vulval development in Caenorhabditis elegans.
53                                              Vulval development in Caenorhabiditis elegans is inhibit
54                              Our analysis of vulval development in cye-1 mutants suggests that a timi
55    Heterochronic genes control the timing of vulval development in the C. elegans hermaphrodite.
56                                              Vulval development in the nematode Caenorhabditis elegan
57                                              Vulval development in the nematode Caenorhabiditis elega
58                       Caenorhabditis elegans vulval development is a paradigmatic example of animal o
59                                              Vulval development is initiated by a signal from the anc
60 lass B synMuv genes act together to regulate vulval development, lin-61 functions separately from som
61                                          The vulval development of Caenorhabditis elegans provides an
62  Cel-mig-13 mutants also have an unsuspected vulval-development phenotype.
63 gh ptp-2 function is not required for normal vulval development, ptp-2(op194) altered significantly t
64     A recent study of Caenorhabditis elegans vulval development reports a novel invasive mechanism in
65  signaling network underlying Caenorhabditis vulval development specifies three fates in a row of six
66 fications converge to inhibit Ras-stimulated vulval development: sumoylation, histone tail deacetylat
67  elegans gene lin-9, a negative regulator of vulval development that acts in the same SynMuvB genetic
68                            During C. elegans vulval development, the anchor cell (AC) in the somatic
69 e NURD chromatin remodelling complex inhibit vulval development through both the synMuvA and synMuvB
70             To identify novel loci acting in vulval development, we carried out a genetic screen for
71  network topology for cell patterning during vulval development, we developed a mathematical model of
72 utations of the same class undergo wild-type vulval development, whereas animals with mutations of an
73 NURD) complex [1] [2] [3] [4] [5] antagonise vulval development, which is induced by the Ras signal t

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