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1                            Information about vulvovaginal and perianal condylomata acuminata and intr
2                                              Vulvovaginal and perianal condylomata acuminata or intra
3                             The treatment of vulvovaginal atrophy includes administration of estrogen
4 n, uniformly had clinical evidence of severe vulvovaginal atrophy, dyspareunia (median pain score, 8
5 promising as a therapeutic option to improve vulvovaginal atrophy.
6 ), gonorrhea (HR, 1.6; 95% CI, 1.1-2.2), and vulvovaginal candidiasis (HR, 1.5; 95% CI, 1.3-1.8).
7            Studies from women with recurrent vulvovaginal candidiasis (RVVC) and from an animal model
8 not associated with development of recurrent vulvovaginal candidiasis (RVVC) in women.
9 e and susceptibility to recurrent idiopathic vulvovaginal candidiasis (RVVC) was investigated.
10                                              Vulvovaginal candidiasis (VVC) caused by Candida albican
11                                              Vulvovaginal candidiasis (VVC) caused by Candida albican
12                                              Vulvovaginal candidiasis (VVC) caused by the commensal o
13 sseminated candidiasis (HDC) and episodes of vulvovaginal candidiasis (VVC) in humans, we found evide
14                                              Vulvovaginal candidiasis (VVC) is a common mucosal infec
15                                              Vulvovaginal candidiasis (VVC) is a high-incidence disea
16                                              Vulvovaginal candidiasis (VVC) is an insidious infection
17                                              Vulvovaginal candidiasis (VVC) is an opportunistic mucos
18  Candida test using a reference standard for vulvovaginal candidiasis (VVC) of yeast culture plus exc
19                          Acute and recurrent vulvovaginal candidiasis (VVC) remains a significant pro
20 s the causative agent of acute and recurrent vulvovaginal candidiasis (VVC), a common mucosal infecti
21 rial vaginosis (BV), 25 acquired symptomatic vulvovaginal candidiasis (VVC), and 7 acquired vaginal t
22 ion with lactobacilli may reduce the risk of vulvovaginal candidiasis (VVC), but supporting data are
23                                              Vulvovaginal candidiasis (VVC), caused by Candida albica
24                                              Vulvovaginal candidiasis (VVC), caused by Candida specie
25 opharyngeal candidiasis (OPC), as opposed to vulvovaginal candidiasis (VVC), is a common opportunisti
26 ections, including bacterial vaginosis (BV), vulvovaginal candidiasis (VVC), or Trichomonas vaginalis
27 ctions, notably bacterial vaginosis (BV) and vulvovaginal candidiasis (VVC), particularly in the sett
28 = 2) with OPC in 1 patient, whereas isolated vulvovaginal candidiasis (VVC; n = 3) was not.
29 anslocation, nor do we implicate the gene in vulvovaginal candidiasis among mice in pseudoestrus.
30 oth clinical studies of women with recurrent vulvovaginal candidiasis and a murine model of experimen
31  24 premenopausal women with acute recurrent vulvovaginal candidiasis and from 21 healthy asymptomati
32                                 In contrast, vulvovaginal candidiasis has a much weaker association w
33                         The risks of GUD and vulvovaginal candidiasis increased with progressive leve
34  women with non-antibiotic-induced recurrent vulvovaginal candidiasis suffering from acute Candida va
35 e randomly assigned 387 women with recurrent vulvovaginal candidiasis to receive treatment with fluco
36  b-), the relative risk of chronic recurring vulvovaginal candidiasis was 2.41-4.39, depending on the
37  infection, Trichomonas vaginalis infection, vulvovaginal candidiasis, and bacterial vaginosis) in HI
38                                              Vulvovaginal candidiasis, caused primarily by Candida al
39 chlamydia, bacterial vaginosis, trichomonas, vulvovaginal candidiasis, pelvic inflammatory disease, g
40 e antifungal susceptibility of yeast causing vulvovaginal candidiasis, since cultures are rarely perf
41 ypeptides in both bacterial vaginosis and in vulvovaginal candidiasis, suggesting that the abnormalit
42                                    Unlike in vulvovaginal candidiasis, the neutrophil attractant chem
43 o colonization and immunopathogenesis during vulvovaginal candidiasis.
44  protection in oral infection but exacerbate vulvovaginal candidiasis.
45 ed to fluid from healthy women or women with vulvovaginal candidiasis.
46  March 2001 from 429 patients with suspected vulvovaginal candidiasis.
47 reduce the rate of recurrence of symptomatic vulvovaginal candidiasis.
48 zole was effective in preventing symptomatic vulvovaginal candidiasis.
49 be effective for the management of recurrent vulvovaginal candidiasis.
50                Despite therapeutic advances, vulvovaginal candidosis remains a common problem worldwi
51  in both overdiagnosis and underdiagnosis of vulvovaginal candidosis.
52  that presented as the oral component of the vulvovaginal-gingival syndrome.
53 s appears to reduce the risk of cervical and vulvovaginal HPV infection.
54 patients with recurrent infections, oral and vulvovaginal isolates were identical, in 35% they were h
55 o (1%) of 341 HIV-1-negative women developed vulvovaginal or perianal lesions, resulting in an incide
56                We investigated vaginal size, vulvovaginal pathology and the presence of the main huma
57 ltaneously cultured from the oral cavity and vulvovaginal region of healthy individuals.
58                                 Cervical and vulvovaginal samples for HPV DNA testing and Papanicolao
59 questionnaire was completed and cervical and vulvovaginal samples were collected to detect HPV DNA.
60 necologic examinations included cervical and vulvovaginal specimen collection for Pap and HPV DNA tes
61 rtner was identical or highly related to the vulvovaginal strain.
62 that included HPV genotyping of cervical and vulvovaginal swab specimens and collection of colposcopy
63                  In community settings, both vulvovaginal-swab and first-catch urine specimens from w
64 ting Chlamydia trachomatis in self-collected vulvovaginal-swab and first-catch urine specimens from w
65                       We tested 2,745 paired vulvovaginal-swab and urine specimens by PCR (Roche Coba
66 I], 93.1 to 99.2%) of infected patients with vulvovaginal-swab specimens and 91.8% (86.1 to 95.7%) wi
67                              We tested 2,749 vulvovaginal-swab specimens with both a nucleic acid amp
68 tivity of an amplified enzyme immunoassay on vulvovaginal-swab specimens.
69  and behavioral information and cervical and vulvovaginal swabs for HPV DNA assay were obtained at 4-
70  musculoskeletal adverse events (MSAEs), and vulvovaginal symptoms (VVSs) in postmenopausal patients

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