ライフサイエンスコーパス: walker

1 modified procedure described by Robinson and Walker.

2 ree', and autonomous locomotion of a bipedal walker.

3 ith substrate (or "track") for the molecular walker.

4 d displacement (OSD) reaction to release the walker.

5 facilitate the development of functional DNA walkers.

6 inetics and processivity of DNA enzyme-based walkers.

7 design of small-molecule synthetic molecular walkers.

8 hose that come closest to being optimal Levy walkers.

9 s are known to enhance exploration of random walkers.

10 gnitude more efficiency than Brownian random walkers.

11 to real-life race performance in elite race walkers.

12 neously be induced for forwards and reversed walkers.

13 st walkers (<0.5 m/s) constituting 28%, slow walkers (0.5-0.83 m/s) making up 48%, and normal walkers

14 ling was attenuated in slow compared to fast walkers (2.8%; 95% confidence interval [CI], -0.9 to 6.6

15 istar rats were inoculated with 2 x 10(7) of Walker 256 tumor cells [tumor bearing (TB) rats].

16 ines that lie close to the LSGGQ (P517C) and Walker A (I1050C) sites of NBD1 and NBD2, respectively,

17 we have mutated conserved residues in Fml1's Walker A (K99R) and Walker B (D196N) motifs to determine

18 activity and include the residues from both Walker A and B motifs and the lid.

19 r mutagenesis analyses indicated that Rrp2's Walker A and B motifs are required for borrelial growth;

20 ctive sites are formed combinatorially, with Walker A and B motifs contributed by one subunit and a c

21 uivalent mutation at ABP1 (consisting of the Walker A and B motifs of NBD1 and the signature sequence

22 dies have indicated that ABP2, formed by the Walker A and B motifs of NBD2 and the signature sequence

23 Mutation of amino acids in the Walker A and B motifs of NS4B resulted in decreased affi

24 and hydrolysis activities engendered by the Walker A and B motifs respectively.

25 sociated with binding and hydrolysis of ATP (Walker A and B motifs).

26 Here, we use Walker A and B mutants to demonstrate that D1 is capable

27 and NMS-873) have differential responses to Walker A and B mutations, to disease-causing IBMPFD muta

28 Alanine substitution mutants in the deviant Walker A and Signature motifs retain significant, albeit

29 The combined data suggest that the Walker A and Walker B motifs of Rrp2 are involved in the

30 ndent on ATP and have identified ATP-binding Walker A and Walker B motifs that are present in Escheri

31 including the G239 residue and the putative Walker A and Walker B motifs.

32 ins contain the nucleotide-binding sequences Walker A and Walker B, respectively.

33 D-loop aspartate functions to stabilize the Walker A asparagine in a position favorable for catalysi

34 tation of either the D-loop aspartate or the Walker A asparagine results in dramatic reductions in AT

35 This interaction depends on the Walker A ATP binding motif of SpoIVA and the LysM domain

36 Inducible expression of an Atcdc48A Walker A ATPase mutant in planta results in cytokinesis

37 prior work using mutations in the conserved Walker A box ATPase structural motif strongly suggests t

38 ly, it appears that two sequence motifs, the Walker A box involved in ATP binding and an iron-sulfur-

39 ation of the conserved Lys-669 to Met in the Walker A box of the first nucleotide-binding domain (Ycf

40 We show that SpoIVA harbors a Walker A box that is required for the proper deployment

41 Further, a divergent Walker A glutamic acid residue acts synergistically with

42 partate and an asparagine residue located in Walker A loop of the opposing subunit.

43 ries of point mutants, each lacking a single Walker A Lys residue, was generated to study the effects

44 Loss-of-ATP binding mutations at the Walker A motif (K83A) or gain-of-ATP binding mutations a

45 including a conserved Lys located within the Walker A motif (or P-loop).

46 e conserved lysine to alanine (K629A) in the Walker A motif abolished ATP hydrolysis and substrate tr

47 It is an ATPase in the MinD/ParA/Mrp deviant Walker A motif family which is within the P loop GTPase

48 Interestingly, the Walker A motif is located in the C-terminal region of RN

49 all possible combinations of wild-type or a Walker A motif K51E variant tau or gamma have been recon

50 Confirming this, a FleQ Walker A motif mutant failed to bind c-di-GMP.

51 The Walker A motif of FleQ is perfectly conserved, opening u

52 -di-GMP competes with ATP for binding to the Walker A motif of FleQ.

53 e or alanine for the conserved lysine in the Walker A motif of SecA2 eliminated ATP binding.

54 ParA that are altered in a key lysine in the Walker A motif of the ATP binding site.

55 Amino acid substitutions in the Walker A motif of TssM caused reduced ATP binding and hy

56 position is conserved in four of the deviant Walker A motif subfamilies (MinD, chromosomal ParAs, Get

57 maS adopts an extended conformation, and the Walker A motif undergoes substantial conformational chan

58 Site-directed mutagenesis showed that a Walker A motif was vital to the function of GguA.

59 Members of this subgroup have a deviant Walker A motif which contains a signature lysine that is

60 Here we show that the putative Zn finger, Walker A motif, KNRXG motif and Lon protease homology do

61 huttling and function depend on a functional Walker A motif.

62 with the gamma-phosphate-binding site of the Walker A motif/P-loop, potentially explaining the limite

63 onstrate that mutations in Cdc6 sensor-2 and Walker A motifs, which are predicted to affect ATP bindi

64 hat differences in the binding affinities of Walker A mutants stem from differences in stabilization

65 Walker A mutations had a smaller effect on DNA binding t

66 Interestingly, the VirB4 ATPase, but not a Walker A nucleoside triphosphate (NTP) binding motif mut

67 Despite the conservation of its Walker A nucleotide-binding motif, the NTPase activity o

68 Hsp104(A503V) variants bearing inactivating Walker A or Walker B mutations in both NBDs are inoperat

69 ing bonds between adenine, triphosphate, and Walker A P-loop (Y142, Q143, and R140).

70 a member of the ParA subfamily of "deviant" Walker A proteins.

71 nalysis of human MRP1-NBD1 revealed that the Walker A S685 forms a hydrogen bond with the Walker B D7

72 A unique "closed to open" movement of Walker A, assisted by trans-acting "Glu switch" Glu-286,

73 The NBD is defined by six motifs: the Walker A, Q-loop, Signature, Walker-B, D-loop, and H-loo

74 with sterol transport, whereas swapping the Walker A, Walker B, and signature motifs together result

75 istic, deviant ATP-binding site with altered Walker A, Walker B, Signature (C-loop), and Q-loop resid

76 dings support the hypothesis that a deviant "Walker A-like" phosphate-binding motif lies adjacent to

77 ibuted to the binding of ATP to glycine rich Walker A-type motifs present in the regulatory domain of

78 A Walker-A "P-loop" motif is proposed to coordinate ATP bi

79 aired translocation phenotypes and show that Walker-A residues play important roles in determining mo

80 le microscopy observations confirm that such walkers achieve directional movement by sensing and modi

81 had 35% higher 30-day mortality than normal walkers (adjusted odds ratio, 1.35; 95% confidence inter

82 e show that the movement of a small-molecule walker along a five-foothold track can be monitored cont

83 hydrolysis of the rear foot of macrocyclized walkers (an information ratchet mechanism), the rear foo

84 ote formation of the ester bonds between the walker and the track.

85 allowing its cargo to be transferred to the walker, and an 'OFF' state, in which no transfer occurs.

86 e their exquisite programmability, DNA-based walkers are challenging to work with because of their lo

87 The fittest composite correlated random walkers are found to be those that come closest to being

88 DNA based synthetic molecular walkers are reminiscent of biological protein motors.

89 ales, but not females, toward perceiving the walkers as more feminine.

90 emales, but not males, toward perceiving the walkers as more masculine.

91 ibution significantly biased in favor of the walker at that site.

92 ParA Walker ATPases form part of the machinery that promotes

93 erved residues in Fml1's Walker A (K99R) and Walker B (D196N) motifs to determine whether its activit

94 ns the key residues of the catalytic motifs, Walker B and C, in the ATPase center and is structurally

95 loading and Cdt1 release, similar as a Cdc6 Walker B ATPase mutant.

96 bution that two other structural motifs, the Walker B box and arginine finger, make to each Mcm2-7 AT

97 ir, a predicted inactivating mutation of the Walker B box domain has no impact on FA pathway associat

98 Walker A S685 forms a hydrogen bond with the Walker B D792 and interacts with the Mg (2+) cofactor an

99 ecause they can alternately contact ptDNA or Walker B glutamate in the ATPase site and lie close to t

100 83A) or gain-of-ATP binding mutations at the Walker B motif (D299N) resulted in Reptin mutants with a

101 at could still bind nucleotide represent the Walker B motif (D478 and D479) and Sensor 1 (N523), thre

102 e third, D290N mutation in the conserved NS3 Walker B motif appeared >/=16 days post-transfection.

103 riant carboxylates, Asp Box motif Glu163 and Walker B motif Glu204, have been assigned to Mg(2+) bind

104 ents that a mutation T194M downstream of the Walker B motif in the phage lambda gpA packaging motor c

105 he C-terminal region of RNase R, whereas the Walker B motif is in its N-terminal region implying that

106 Mutation of the Walker B motif of RPT6 prevented XopJ-mediated degradati

107 Importantly, we discovered the Walker B motif of TssM and demonstrated that it is criti

108 otein F (PspF), by specifically altering the Walker B motif sequence required in catalyzing ATP hydro

109 ite with a non-catalytic residue next to the Walker B motif.

110 tly connected to the catalytically important Walker B motif.

111 ag with a single residue substitution in the Walker B motif.

112 combined data suggest that the Walker A and Walker B motifs of Rrp2 are involved in the control of a

113 and have identified ATP-binding Walker A and Walker B motifs that are present in Escherichia coli RNa

114 riant (ClpC(trap)) with mutations within the Walker B motifs to identify the direct substrates of Clp

115 e G239 residue and the putative Walker A and Walker B motifs.

116 V) variants bearing inactivating Walker A or Walker B mutations in both NBDs are inoperative.

117 of the noncanonical methionine residue M584 (Walker B sequence of nucleotide binding site 1) by gluta

118 somerization of a conserved cis peptide near Walker B to the trans configuration, which appears to pr

119 ol transport, whereas swapping the Walker A, Walker B, and signature motifs together resulted in fail

120 he nucleotide-binding sequences Walker A and Walker B, respectively.

121 f three motifs implicated in ATP hydrolysis (Walker B, sensor 1, and sensor 2) are mutated.

122 iant ATP-binding site with altered Walker A, Walker B, Signature (C-loop), and Q-loop residues.

123 six motifs: the Walker A, Q-loop, Signature, Walker-B, D-loop, and H-loop.

124 approximately 0.7 s per step) than previous walkers based on covalent chemistry and is weakly proces

125 entirely to autonomous decisions made by the walker, behaviour analogous to amorphous chemical reacti

126 g protein ParG, the parH centromere, and the Walker box ATPase ParF.

127 jP protein (Soj from pXO1) contains putative Walker box motifs and belongs to the ParA family of ATPa

128 Walker box mutations in RFS-1, which abolish filament re

129 e insight into a unique mechanism by which a Walker box protein forms polymers that involves the gene

130 Moreover, it is unknown how any Walker box protein polymerizes in an ATP-dependent manne

131 most common plasmid-encoded NTPases contain Walker-box and actin-like folds.

132 , and an assembling protein that is either a Walker-box ATPase (type I) or an actin-like ATPase (type

133 ly mediates dimerization, a winged-HTH and a Walker-box containing C-domain.

134 Well-studied plasmid Walker-box partition modules require ParA, centromere-DN

135 Walker-box partition systems are ubiquitous in nature an

136 induced propagation both below and above the Walker breakdown field it is shown that all experimental

137 It is also noted that Walker breakdown occurs at lower fields and peak DW velo

138 th dopants such as holmium, acts to suppress Walker breakdown phenomena.

139 rvation and the piezoelectric control of the Walker breakdown separating two regimes with different m

140 aids the velocity enhancement by preventing Walker breakdown.

141 ith increasing applied field associated with Walker breakdown.

142 pplications of this SNA-based stochastic DNA walker by exploiting movement-triggered cascade signal a

143 el microparticles and the detection of those walkers by substrate-coated microparticles.

144 on reactions, the nanoscale movements of the walker can lead to the generation of a single-stranded p

145 The walking behaviour is robust and the walker can take more than 30 continuous steps.

146 ticle, we show that the presence of multiple walkers can be beneficial for a procedure to estimate th

147 show that the translocation kinetics of DNA walkers can be effectively controlled by external light

148 onvergence Zone and weakening of the Pacific Walker circulation (PWC) between approximately 1000 and

149 In addition to the anomalous SST, the Walker circulation anomalies at different ENSO phases al

150 o such as the rising branch of the anomalous Walker circulation being shifted to the central Pacific

151 ey do show that the observed slowdown in the Walker circulation is presumably driven by oceanic rathe

152 patterns are the main cause of the weakened Walker circulation over the past six decades (1950-2009)

153 AMO state, there is an anomalously weakened Walker circulation over the tropical Pacific that transp

154 simulate the observed changes, including the Walker circulation slowdown and the eastward shift of at

155 lation above the tropical Pacific Ocean (the Walker circulation) over the twentieth century.

156 ics of the hiatus, including the intensified Walker circulation, the winter cooling in northwestern N

157 n rainfall variability by altering the local Walker circulation, whereas the influence of the Pacific

158 ia global reorganizations of the atmospheric Walker Circulation.

159 to insolation forcing through changes in the Walker circulation.

160 Zone (ITCZ) and intensification/slowdown of Walker circulation.

161 risingly, the NHSM as well as the Hadley and Walker circulations have all shown substantial intensifi

162 Dynamic DNA enzyme-based walkers complete their stepwise movements along the pres

163 follows from this that vast numbers of Levy walkers could be hiding in plain sight.

164 per day (-813 steps, 95%CI,-1043,-582) than walkers/cyclists.

165 lic transport had similar activity levels to walkers/cyclists.

166 d no aftereffect, yet when adapting and test walkers differed in size or appeared on opposite sides o

167 In this issue of Blood, Walker et al investigate the preclinical potential of KP

168 of Blood, Keats et al,(1) Egan et al,(2) and Walker et al(3) provide a genome-wide snapshot of the cl

169 ncorrectly placed to tribe and that Sophonia Walker, Evacanthus Le Peletier &Serville, Bundera Distan

170 The "walker" features a palladium(II) complex "foot" that can

171 We report the release of catalytic DNA walkers from hydrogel microparticles and the detection o

172 ers (0.5-0.83 m/s) making up 48%, and normal walkers (>0.83 m/s) constituting 24% of the population.

173 The slowest walkers had 35% higher 30-day mortality than normal walk

174 The bipedal walker has identical carboxylic acid feet, and "steps" a

175 n, a variety of DNA-based and small-molecule walkers have been created, but observing the translation

176 al Runners' (17,201 male, 16,173 female) and Walkers' Health Studies (3,434 male, 12,384 female).

177 olled three isoenergetic diets in elite race walkers: high CHO availability (g kg(-1) day(-1) : 8.6 C

178 of finite length and the protein as a random walker in the free energy landscape.

179 Twenty-two fast and 20 slow walkers in the lowest quartile of cerebral vasoreactivit

180 of the intrinsic quantum nature of multiple walkers, in order to achieve the full computational powe

181 aining and mild energy deficit in elite race walkers increases peak aerobic capacity independent of d

182 ssibility of gathering information about the walkers indistinguishability from the observation of bun

183 The walker is an organoarsenic(III) molecule with exchangeab

184 The random walker is designed to make random moves to adjacent node

185 The navel orangeworm, Amyelois transitella (Walker), is an agricultural insect pest that can be cont

186 ., Miller, S., Ho, S-Y., Wang, W., Chen, Q., Walker, K., Wypych, J., Narhi, L., and Gunasekaran, K.

187 hen the alanine residue (A6) in the atypical Walker-like A box of AaTadZ was converted to lysine, the

188 at position 6 (K6) present in the canonical Walker-like A box.

189 at all TadZ proteins are predicted to have a Walker-like A box.

190 percentile, 0.47-0.79 m/s), with the slowest walkers (<0.5 m/s) constituting 28%, slow walkers (0.5-0

191 ng the anti-inflammatory actions of statins.-Walker, M.

192 frequency of the transitions that the random walker makes - between nodes in different PPI networks,

193 FOXC1 loss contributes to Dandy-Walker malformation (DWM), a common human cerebellar mal

194 Dandy-Walker malformation (DWM), the most common human cerebel

195 ith the common cerebellar birth defect Dandy-Walker malformation (DWM).

196 chromosome 3q25.1 are associated with Dandy-Walker malformation of the cerebellum, and loss of the o

197 posterior fossa defects (including the Dandy-Walker malformation), and renal cystic dysplasia.

198 rain abnormalities (e.g., heterotopia, Dandy-Walker malformation), pituitary insufficiency, and/or sy

199 Loss of Foxc1 is associated with Dandy-Walker malformation, the most common human cerebellar ma

200 tions of the genes implicated in human Dandy Walker Malformation, Zic1 and Zic4.

201 Habitual runners and walkers may quantify exercise in terms of distance (km/d

202 : First, we develop a fast, automatic random walker method for cell segmentation.

203 tation is faster than the traditional random walker method or the level set method, and performs bett

204 The dynamics of the walker migration is well described by the random walk of

205 In 2009, Walker, Miles, and Davis proposed a model of amygdala-BN

206 idence interval [0.19, 0.78]) under the Eyre-Walker model.

207 The diffusion temperature of molecular 'walkers', molecules that are capable of moving unidirect

208 e ParA motor, which contains a bacteria-like Walker motif.

209 RNase R due to mutations in its ATP-binding Walker motifs exhibit growth defects at low temperatures

210 rporate these characteristics, the idea that walker motion is encoded directly, such that viewpoint a

211 to look for the co-encoding of viewpoint and walker motion, a hallmark of motion template analysis.

212 enzyme, leading to high processivity of the walker movement along the track.

213 esign parameters enables us to demonstrate a walker movement near 5 mum at an average speed of approx

214 The walker moves considerably faster ( approximately 0.7 s p

215 we show that a DNA mechanical device--a DNA walker moving along a DNA track--can be used to perform

216 predictions derived from this strategy: (1) Walkers must have information about upcoming footholds d

217 ution of crosslinks and consumes neither the walker nor the track irreversibly.

218 ling microscopy shows that the motion of the walker occurs along the [110] direction of the Cu surfac

219 On the longest track (n = 9) the fraction of walkers on each end-foothold can be quantified with resp

220 ikely to require assistive devices such as a walker or wheelchair for mobility (OR=23.00; p=0.007).

221 e-body fat oxidation during exercise in race walkers over a range of exercise intensities.

222 rrent, atmospheric wind, a preference of the walker owing to prior experience, or a general bias in a

223 ) for males and P=0.02 for females) and male walkers (P=0.01 for males and P=0.08 for females) and fo

224 P<10(-5) for males and P=0.003 for females; walkers: P=0.03 for males and P<10(-4) for females), hig

225 Herein we review recent progress on DNA walker principles and characterization methods, and eval

226 The walker randomly and processively takes zero or one step

227 field effect is found to change sign in the Walker regime.

228 e track via reversible protonation while the walker remains attached to the track throughout by means

229 , and 5.4% for the slowest, slow, and normal walkers, respectively (P<0.001).

230 r's areas 11 and 13 and the other centers on Walker's area 14.

231 One subregion encompasses Walker's areas 11 and 13 and the other centers on Walker

232 sensitive to lesions of the PFo as a whole (Walker's areas 11, 13, and 14).

233 tricted excitotoxic lesions targeting either Walker's areas 11/13 or area 14.

234 so the resulting ballistic trajectory of the walker's center-of-mass will facilitate stepping on targ

235 trotter" was sufficient to bias the bistable walker's direction, as were full-body adaptors.

236 ile varying the presentation duration of the walker selectively affected body motion discriminations.

237 mple model for a chemically driven molecular walker shows that the elastic energy stored by the molec

238 demonstrate that previously developed random walkers-so-called molecular spiders that comprise a stre

239 ryos had cerebellar aplasia similar to Dandy-Walker spectrum malformations observed in human patients

240 aligning the ellipses that made up a dynamic walker stimulus selectively disrupted body form discrimi

241 3), distal arthrogryposis 5 (DA5), or Marden-Walker syndrome (MWKS), which encompass contractures of

242 istal arthrogryposis type 5 (DA5) and Marden-Walker syndrome (MWS).

243 construct highly processive, autonomous DNA walker systems and to regulate their translocation kinet

244 The functionalities of state-of-the-art DNA walker systems can thus be analyzed for various applicat

245 easures the average number of steps a random walker takes to reach the ith node.

246 tion and hydrolysis reactions lead to 68% of walkers taking two steps directionally along a three-foo

247 uclease III (Exo III)-powered stochastic DNA walker that can autonomously move on a spherical nucleic

248 as been used in a remote-controlled inchworm walker that can directly couple a color-changing skin wi

249 re attached in series to the tile, and a DNA walker that can move on the track from device to device

250 e have constructed an autonomous DNA bipedal walker that coordinates the action of its two legs by cy

251 of a model system comprised of a bipedal DNA walker that strides on a DNA origami track powered by in

252 Here, we report a DNA walker that uses hybridization to drive walking on DNA-c

253 been developed including self-assembled DNA walkers that can make stepwise movements on RNA/DNA subs

254 be treated essentially as many-legged random walkers that can pass through a cytoskeletal barrier by

255 inspired the construction of rudimentary DNA walkers that run along self-assembled tracks.

256 We describe a small-molecule "walker" that uses enzyme catalysis to discriminate betwe

257 By covalently cross-linking aliquots of the walker to its track in successive walking states, we dem

258 des of control work synergistically to allow walkers to negotiate complex terrain with efficiency, st

259 Only macrocyclic walker-track conjugates are efficiently hydrolyzed by th

260 ding molecular muscles, synthesizers, pumps, walkers, transporters and light-driven and electrically

261 As the walker traverses the pathway prescribed by the origami t

262 Sackler Foundation, and Sir David and Isobel Walker Trust.

263 ions, we show dynamic chromosome binding and Walker-type ATPase activity are essential for cluster se

264 inding protein, typically called ParB, and a Walker-type ATPase, typically called ParA, which also bi

265 ital encephalocele, hydrocephalus, and Dandy Walker-type cerebellar anomalies).

266 SegA is an ortholog of bacterial, Walker-type ParA proteins, whereas SegB is an archaea-sp

267 fficients is used to form a generalized Yule-Walker-type system of equations.

268 We propose that walkers use visual information to initialize the mechani

269 Auerbach S, Filer D, Reif D, Walker V, Holloway AC, Schlezinger J, Srinivasan S, Svob

270 s more than median walking distance, or slow walker versus fast walker) was also assessed.

271 Walker-Warburg syndrome (WWS) is clinically defined as c

272 o identify genetic mutations responsible for Walker-Warburg syndrome (WWS), a genetically heterogeneo

273 linked to a variety of phenotypes including Walker-Warburg syndrome (WWS), limb girdle muscular dyst

274 y (FCMD), Muscle-Eye-Brain disease (MEB) and Walker-Warburg syndrome (WWS), which are associated with

275 th predominantly muscle phenotypes to severe Walker-Warburg syndrome and muscle-eye-brain disease wit

276 congenital muscular dystrophy 1C (MDC1C), to Walker-Warburg Syndrome and Muscle-Eye-Brain disease.

277 stroglycan, which not only causes the severe Walker-Warburg syndrome but is also a common cause of th

278 with brain and eye anomalies and range from Walker-Warburg syndrome to Fukuyama congenital muscular

279 congenital muscular dystrophies that include Walker-Warburg syndrome, muscle-eye-brain disease, and F

280 e congenital muscular dystrophies, including Walker-Warburg syndrome, muscle-eye-brain disease, Fukuy

281 fferent clinical manifestations ranging from Walker-Warburg syndrome, the most severe form of dystrog

282 vere of the congenital muscular dystrophies, Walker-Warburg syndrome, to mild forms of adult-onset li

283 ing (ISPD), are a relatively common cause of Walker-Warburg syndrome.

284 nging from limb girdle muscular dystrophy to Walker-Warburg syndrome.

285 uman conditions muscle-eye-brain disease and Walker-Warburg syndrome.

286 The odds ratio of being a slow walker was 6.4 (95% CI, 1.7-24.9; p = 0.007) if there wa

287 Mary Edwards Walker was a gallant woman who stood for women's rights,

288 ch suggested that the performance of the DNA walker was critically dependent upon the DNA density and

289 The operation of this Exo III-propelled DNA walker was monitored in real time and at the single-part

290 walking distance, or slow walker versus fast walker) was also assessed.

291 tory conventional metadynamics, and multiple-walkers well-tempered metadynamics calculations.

292 nced a consistent aftereffect: a bistable PL walker, which could be perceived in the adapted orientat

293 ore reflect the individual steps of a single walker, which require the making and breaking of As-S bo

294 in this direction was the development of DNA walkers, which have developed from being non-autonomous

295 Baseline slow walkers who were able to improve the 6MWT distance prese

296 int-light displays that portray the gaits of walkers whose gender is digitally morphed from male to f

297 After exposure to a PL walker with a particular stereoscopically defined headin

298 l ring of annihilating and coalescing random walkers with deterministic biases due to selection.

299 ques enable direct observation of individual walkers with high temporal and spatial resolution.

300 this problem by modelling animals as random walkers with scent-mediated interaction processes.