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1 ith higher roasting temperatures (17-fold in walnuts).
2 ol in studies aimed at genetically improving walnut.
3 loping repeat DNA-based molecular markers in walnut.
4 based on CTAB-phenol-chloroform was best for walnut.
5 cashew, hazelnut and/or pistachio but not to walnut.
6 erapy approach may not be most effective for walnut.
7 ts as an indirect regulator of cell death in walnut.
8 ds and tocopherols especially in almonds and walnuts.
9  more efficiently than high pressure treated walnuts.
10 sive identification of phenolic compounds in walnuts.
11 idant capacity and arbutin levels than amber walnuts.
12 entially health-relevant phytochemicals from walnuts.
13 uts and tree nuts (2 or more times/week) and walnuts (1 or more times/week) was associated with a 13%
14 ts (1.4-fold), macadamia nuts (1.7-fold) and walnuts (3.7-fold).
15 he total antioxidant capacity was highest in walnuts (3689.7muM trolox equivalents 100g(-1)) followed
16 ergy in sensitized patients was observed for walnut (74.6%) and cashew (65.6%).
17 nguished allergic from tolerant patients for walnut (87%), pecan (66%), cashew (71%), and pistachio (
18  of this study was to investigate changes in walnut allergenicity after processing treatments by in v
19  examine CD4(+) T-cell reactivity toward the walnut allergens Jug r 1, Jug r 2, and Jug r 3.
20 ittle information is available regarding how walnut allergens respond to thermal processing.
21 ns of reactivity observed for human IgE from walnut-allergic individuals.
22  in nonasthmatic and asthmatic patients with walnut allergy.
23 ate walnut T-cell responses in patients with walnut allergy.
24 of consuming single or mixed nuts (including walnuts, almonds, pistachios, cashews, hazelnuts, macada
25             Lack of cross-reactivity between walnut and cashew suggests that cashew peptide immunothe
26 pistachio-allergic patients were allergic to walnut and cashew, respectively.
27                  The method was positive for walnut and pecan respectively, and negative for all othe
28  exogenous application elicits cell death in walnut and several other plant species.
29 first time that arbutin has been reported in walnuts and could provide the first insight into how enz
30                                    Including walnuts and fatty fish in a healthy diet lowered serum c
31 ation; it is also present in high amounts in walnuts and flaxseed.
32 sources including flaxseed and flaxseed oil, walnuts and walnut oil, and canola oil are recommended.
33 ella, is a worldwide pest of apple, pear and walnut, and behavior-modifying semiochemicals are used f
34 mothy, pigweed, Russian thistle, cottonwood, walnut, and CDE stimulated MD2-dependent CXCL secretion.
35 ific types of nuts, specifically peanuts and walnuts, and cardiovascular disease remain unclear.
36 ound mostly in plant foods such as flaxseed, walnuts, and vegetable oils, including canola and soybea
37                                Two-thirds of walnut- and cashew-allergic patients were also allergic
38 oreactivity (using anti-raw and anti-roasted walnut antisera), with similar patterns of reactivity ob
39                 The samples studied included walnuts, Brazil nuts, Macadamia nuts, pecans, hazelnuts,
40                        Allergic reactions to walnut can be life-threatening.
41  foundation for in depth characterisation of walnut compounds and offered an efficient strategy for i
42  trials are needed to address the effects of walnut consumption on cardiovascular risk and body weigh
43 als have demonstrated consistent benefits of walnut consumption on coronary heart disease risk and ot
44 ractical detection limit of 0.1 mg kg(-1) of walnut content was estimated.
45          Cashew and pistachio, and pecan and walnut cross-react the most.
46  growing areas and four different colours of walnuts cv.
47 ean/low-carbohydrate (MED/LC) diet plus 28 g walnuts/d with a calorically equal low-fat (LF) diet amo
48 erranean/low-carbohydrate (MED/LC) diet+28 g walnuts/day with/without added moderate physical activit
49  1.19 +/- 0.05 mmol/L, respectively) and the walnut diet (1.11 +/- 0.11 mmol/L, P < 0.05, and 1.18 +/
50 ol concentrations in adults who followed the walnut diet (4.87 +/- 0.18 and 2.77 +/- 0.15 mmol/L, res
51 e lower (P < 0.05) in those who followed the walnut diet compared with those who followed the control
52 lycerides were not significantly affected by walnut diets more than with control diets (HDL cholester
53  The spring phenological behavior of 'Payne' walnut differed depending on bud type.
54 sure (HHP) did not produce any effect on the walnut DNA amplification.
55 and amplification (integrity and quality) of walnut DNA.
56 nce, and the limit of detection was 2.5pg of walnut DNA.
57                                Overall, high-walnut-enriched diets significantly decreased total and
58    The highest phenolic content was found in walnuts, followed by pistachios extracts (596.9 and 410.
59 d antiradical capacity of different parts of walnut fruit among six genotypes of Juglans regia L.
60                    The density of SSR in the walnut genome analyzed was also slightly higher than tha
61  study is an initial characterization of the walnut genome and provides the largest genomic resource
62 Ss indicated that approximately 11.5% of the walnut genome represents a coding sequence.
63                                          The walnut genome sequence provides important tools and meth
64 ight into the structure and evolution of the walnut genome, we constructed two bacterial artificial c
65 2 Mb, representing approximately 5.1% of the walnut genome.
66              These results demonstrated that walnut genotypes have different phenolic compounds and p
67 t were determined and methanolic extracts of walnut genotypes were considered by the reducing power,
68 r ago, probably reflecting the time when the walnut genus last shared a common ancestor.
69                                              Walnuts grown in the zone with Andes Mountains influence
70                                  Extra light walnuts had higher (p<0.05) total phenolic compounds, an
71 reover, high consumption of nuts (especially walnuts) has been associated with lower diabetes risk.
72                     Although IgE epitopes of walnut have been studied, CD4(+) T cell-specific epitope
73 ildren, peanut (a legume) and tree nuts (ie, walnut, hazel nut, Brazil nut, pecan) have attracted con
74  to represent peanut, almond, pecan, cashew, walnut, hazelnut, pine nut, Brazil nut, macadamia nut, p
75 g a series of model samples with defined raw walnut in wheat flour and heat-treated walnut in wheat f
76 d raw walnut in wheat flour and heat-treated walnut in wheat flour with a range of concentrations of
77                                              Walnuts in particular have a unique profile: they are ri
78                 We determined the effects of walnut intake on plasma fatty acids, lipoproteins, and l
79                          The light colour of walnuts is a quality attribute that leads to consumer pr
80  dynamics of N remobilization was studied in walnut (Juglans nigra x regia) trees growing in sand cul
81                                      Persian walnut (Juglans regia L.) is an economically important t
82 latile compounds in the kernel oils from six walnut (Juglans regia L.) varieties.
83                                  The Persian walnut (Juglans regia L.), a diploid species native to t
84 reaction (PCR)-based assays for detection of walnut (Juglans regia) and pecan (Carya illinoinensis) t
85 for timing of leaf-out and male flowering of walnut (Juglans regia) cultivar 'Payne' to examine this
86                                              Walnut (Juglans regia) produces a rich array of phenolic
87                                              Walnuts (Juglans regia L.) are well known for their flav
88                                              Walnuts (Juglans regia L.) have been described previousl
89                                      English walnuts (Juglans regia) are among the most commonly alle
90                                              Walnuts (Juglans regia) have been associated with foodbo
91 enerated a series of PPO-silenced transgenic walnut lines that display less than 5% of wild-type PPO
92 g r 3 primers detected up to 100mg/kg of raw walnut (LOD 0.01%, LOQ 0.05%).
93 and insoluble protein fractions from roasted walnuts maintained substantial amounts of IgG immunoreac
94 ted by IgE-immunoblot and antibodies against walnut major allergen Jug r 4.
95 hildren were challenged with soy (n = 10) or walnut (n = 13).
96 uding flaxseed and flaxseed oil, walnuts and walnut oil, and canola oil are recommended.
97 t variables: ultrasonic time (UT, 5-15 min), walnut-oil content (WO, 4-10% (w/w)) and Span 80 content
98 several trials and to estimate the effect of walnuts on blood lipids.
99 nella), an important pest of apple, pear and walnut orchards worldwide.
100 ative weight loss for yogurt, peanut butter, walnuts, other nuts, chicken without skin, low-fat chees
101 ted and strongly correlated with IgE against walnut, peach, and apple and against Chenopodium, plane
102 in hazelnut, almond, cashew nut, Brazil nut, walnut, pecan nut and pistachio nut show heat stability
103 y-three patients prospectively evaluated for walnut, pecan, cashew, pistachio, hazelnut, and almond a
104        We examined the effects of tree nuts (walnuts, pistachios, macadamia nuts, pecans, cashews, al
105       The objective was to determine whether walnuts (plant n-3 fatty acid) and fatty fish (marine n-
106 lation of complex mixtures of compounds from walnut polar extracts was established by a combination o
107 t common causes of pollen allergy) and black walnut pollen (Juglans nigra) were used as model bioaero
108                       A dramatic decrease in walnut protein solubility was observed following dry roa
109                                        Thus, walnut proteins are relatively stable under certain ther
110 ceptibility to digestion of pressure treated walnut proteins was observed.
111 n) on the solubility and immunoreactivity of walnut proteins.
112 ted walnut-specific IgE upon oral feeding of walnut proteins.
113 esults reported in the trials indicated that walnuts provided significant benefits for certain antiox
114                    Diets lasted 4-24 wk with walnuts providing 10-24% of total calories.
115 o provide a thorough phenotypic analysis for walnut-reactive T cells in allergic and nonallergic subj
116 uts, almonds, macadamia nuts, pistachios and walnuts regarding health-promoting and potentially harmf
117                          Previous studies in walnut relying on tissue-specific methods have only iden
118 studied, CD4(+) T cell-specific epitopes for walnut remain uncharacterized.
119  with control diets, diets supplemented with walnuts resulted in a significantly greater decrease in
120 tachio (rho = 0.66; P < 0.001) and pecan and walnut (rho = 0.65; P < 0.001) correlated the strongest.
121 protection), IgE against Cor a 14 (risk) and walnut (risk) increased the AUC to 0.91.
122 TRIM elements represent the first identified walnut short interspersed element (SINE) and terminal-re
123 ainous regions of Central Asia, is the major walnut species cultivated for nut production and is one
124        The population histories of temperate walnut species were not driven by extrinsic environmenta
125        Cashew-allergic mice develop elevated walnut-specific IgE upon oral feeding of walnut proteins
126 ationship of both phenotype and frequency of walnut-specific T cells to the disease have not been exa
127 elationship of phenotypes and frequencies of walnut-specific T cells with the disease.
128 polyphenols that have never been reported in walnuts: stenophyllanin C, malabathrin A, eucalbanin A,
129                         The allergenicity of walnuts subjected to high hydrostatic pressure and therm
130                      The results showed that walnuts subjected to pressure treatment at 256 kPa, 138
131 al women and 5 men ( +/- SD age 60 +/- 8 y), walnut supplementation did not increase body weight desp
132  subclasses and particle size suggested that walnut supplementation lowered cholesterol preferentiall
133                                              Walnut supplementation may beneficially alter lipid dist
134 ate the distribution of bacteria on in-shell walnut surfaces from six representative provinces in Chi
135 on and abundance of bacterial consortiums on walnut surfaces varied among the geographical sites wher
136                 The bacterial populations on walnut surfaces were investigated by high-throughput seq
137 bacterial communities' diversity on in-shell walnut surfaces.
138          Jug r 2-specific responses dominate walnut T-cell responses in patients with walnut allergy.
139                     The ability of processed walnut to cross-link IgE on effector cells was evaluated
140 g capacity could be a potential strategy for walnut tolerance induction.
141 , 98.6 +/- 6.4%, and 67.0 +/- 4.5% for beer, walnut, tomato and sour cherry samples, respectively.
142 sitivity and reliability in the detection of walnut traces in commercial foodstuffs compared with ELI
143 nome consists of known repetitive DNA, while walnut-unique repetitive DNA identified in this study co
144                                    Among the walnut-unique repetitive DNA, Julia SINE and JrTRIM elem
145 zelnuts, peanuts, pine nuts, pistachios, and walnuts) using a QuEChERS-LC-ESI-MS-Triple Quadrupole ap
146 ion to foods (milk, egg, peanut, soy, wheat, walnut) was assessed.
147 tocopherol equivalents) while pistachios and walnuts were rich in gamma-tocopherol.
148 n D (4) was isolated for the first time from walnut, while praecoxin A methyl ester (5) and glansregi
149 Bosnian pine, cherry, common juniper, common walnut, white mulberry, black locust and apricot).
150                         The use of processed walnuts with decreased IgE binding capacity could be a p
151                 Concurrently sensitized PN-, walnut- (WN) and cashew (CSH)-allergic mice received 1-d

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