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1 ren ages 10 to 14 years old, consistent with waning immunity.
2 d dose for outbreak control and assessed for waning immunity.
3 in highly vaccinated populations, indicating waning immunity.
4 ause of impaired initial vaccine response or waning immunity.
5 rgences of the epidemic, we go on to include waning immunity and a mechanism explicitly accounting fo
6 rces (instantaneous rates) of infection, but waning immunity and clinical consequences that depend on
7 pertussis resurgence has been attributed to waning immunity and other factors, vaccine refusal was s
8 c drift phenomenologically with continuously waning immunity, and the second epochal evolution model
10 te history of vaccination also suggests that waning immunity could have contributed to susceptibility
11 oupled with exposure to infected caregivers, waning immunity despite complete vaccination, inferior l
13 ges in sustaining high vaccination coverage, waning immunity in HIV-1-infected children, and clusteri
15 any developed countries has been linked with waning immunity induced after immunization with acellula
17 lusters of nonmedical vaccine exemptions and waning immunity may have been factors contributing to co
18 he role of nonmedical vaccine exemptions and waning immunity may have had on the resurgence of pertus
20 oncerns remain about lower effectiveness and waning immunity of rotavirus vaccines in resource-poor p
22 ine improved mumps outbreak control and that waning immunity probably contributed to propagation of t
23 rmation for both primary vaccine failure and waning immunity provide essential information for our pr
25 vaccine transmissibility reveals that higher waning immunity rates make eradication more difficult be
27 sibility, clearance of initial infection and waning immunity were derived in a Bayesian framework by
28 model for wild poliovirus that incorporates waning immunity (which affects both infection risk and t
29 porating high asymptomatic ratio and rapidly waning immunity, with infection both from human and envi
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