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1 of these Grp(+) neurons increased pain responses while itch was decreased.
2 with unmanipulated littermates, whereas crypt proliferation was decreased.
4 Fibroblast-specific collagen expression was decreased and might be due to decreased stretch and/or al
5 After CHO, muscle PDH-E1alpha Ser(300) phosphorylation was decreased, and glucose oxidation increased.
8 vely, and urinary lipid peroxidation marker malondialdehyde was decreased by 32 +/- 21% compared to baseline.
11 sor CCCTC-binding factor (CTCF) to the MSMP enhancer region was decreased by histone acetylation under hypoxic conditions
12 idal neurons; a cocaine-induced increase in PL excitability was decreased by riluzole, and a cocaine-induced decrease in
13 ED1 increased Fos expression in DH, LC, and DR, and DH Fos was decreased by systemic S-propranolol.
16 Moreover, resting-state functional coupling was decreased during atDCS compared with stDCS, most likely i
21 r RelA was enhanced, whereas nuclear translocation of c-Rel was decreased in A20-deficient thymic Treg cells.
26 Similar to the alpha7 integrin, Ptrh2 expression was decreased in laminin-alpha2 dyW null gastrocnemius muscle
28 hondrial positioning; focal adhesion assembly and stability was decreased in Miro1(-/-) MEFs compared with Miro1(+/+) MEF
30 Notch1 activation, known to promote arterial specification, was decreased in mutant DA endothelial cells (ECs), which ect
34 n brief, consistent with the results of basic studies, PDE4 was decreased in unmedicated MDD patients and increased after
35 In contrast, protein expression of ESC/E(Z) genes was decreased in untreated PMDD LCLs with MTF2, PHF19 and SIR
36 er, Scube2, a glycoprotein regulating astrocyte Shh release was decreased, inhibiting Shh delivery to brain endothelial c
38 o sodium thiosulfate; the most common serious adverse event was decreased neutrophil count: 26 episodes in 14 participant
41 ex RSFC was predictive of treatment response at 5-weeks, as was decreased parahippocampal-prefrontal cortex RSFC.
42 lphai2 was increased whereas the NDPK-C/Galphas interaction was decreased, producing a switch that may contribute to an N
44 cebo (nitrogen) or inhaled nitric oxide initiated at 20 ppm was decreased to 10 ppm between 72 and 96 hours after startin
45 Accordingly, the activity of the wild type RUNX2 promoter was decreased upon methylation treatment in vitro.
48 ssue was significantly upregulated, and the content of 5-HT was decreased which negatively correlated with the gastrointe
50 zation rates of glutamine, oxidative utilization of glucose was decreased, while the production of lactate from glutamine
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