ライフサイエンスコーパス: was inhibited

1 s silenced in breast cancer cells, their metastatic ability was inhibited.

2 Classical complement activation was inhibited by pretreatment of complement with an anti-C1s

3 Store-operated TRPC1 channel activity was inhibited by TRPC1 and STIM1 antibodies and STIM1 short h

4 Anaphylaxis was inhibited by platelet-activating factor receptor or hista

5 Proliferation was higher in BERKO NPCs and was inhibited by LY3201.

6 In primary hepatocytes, autophagy was inhibited by 3MA or autophagy-related protein 7 (Atg7) sm

7 Upon stimulation of TLR4, the rapid induction of IFN-beta was inhibited in cells loaded with oxLDL, whereas the inducti

8 Internalization was enhanced by extracellular proteins but was inhibited by cigarette smoke extract via oxidative disrup

9 Phosphorylation of IRP1 at Ser-138 increased when CIA was inhibited and was required for iron rescue.

10 ough the morphological transition of IL1B2(-/-) conceptuses was inhibited, expression of a number of conceptus developmen

11 in both monospecies as well as multispecies broth cultures was inhibited in a dose-dependent manner while that of S. gor

12 V8-induced integrity damage was inhibited by an IL-1beta-mediated mechanism, independent

13 conversion of TCS and, in particular, the formation of DCDD was inhibited in the presence of ACs.

14 This effect was inhibited by BoNT/A, supporting a role for Gbetagamma/SNA

15 reover, Ldgp63-containing COPII vesicle budding from the ER was inhibited by LdSar1:T34N in an in vitro budding assay, in

16 naive T cells by Ag stimulation, but Egr2 and 3 expression was inhibited by Th1-inducing cytokines.

17 Interestingly, AmAChE1 expression was inhibited when bees were stimulated for brood rearing by

18 Strikingly, the number of Tmem was only reduced when FAS was inhibited during T cell priming and not during the Tmem s

19 ct on AMP activity and even with 10% serum bacterial growth was inhibited.

20 Fungal growth was inhibited on the AgBr/NPVP-modified PMMA resin compared t

21 thin the physiological range of bile salts; however, growth was inhibited at higher concentrations.

22 However, the increase was inhibited by pertussis toxin as well as by wortmannin but

23 The influx was inhibited by MK-801, a specific pore blocker of N-Methyl-

24 Interestingly, the PEX5(1-125)-DTM interaction was inhibited by a polypeptide comprising PEX5 residues 138-6

25 The effect of LTE4 was inhibited by montelukast, a CysLT1 antagonist.

26 Dvl2 recruitment to the cell membrane was inhibited by Gsc in Xenopus animal cap assays and key Wnt

27 However, the growth of MRSA was inhibited, and a significant protection in mice against t

28 When Notch1 was inhibited, this E2-mediated protection was not observed,

29 The LPS-induced increase in intestinal permeability was inhibited in MLCK(-/-) and TLR-4(-/-) mice.

30 xygenase responsible for co-metabolic transformation of PHE was inhibited by 1-octyne.

31 13 protein expression was abolished and Akt phosphorylation was inhibited.

32 This ET-1 production was inhibited by anti-IFN-gamma and/or TNF-alpha antibody.

33 Aa-induced IL-6 and IL-8 production was inhibited by rosuvastatin, particularly at higher doses.

34 NTHi-specific lung CD4(+) T cells, and cytokine production was inhibited by a HLA-DR blocking Ab.

35 Considering only the surface mixed layer, production was inhibited 7-73%.

36 evious work indicated that when in vivo Pf phage production was inhibited, P. aeruginosa was less virulent.

37 lly, the lncRNA plasmacytoma variant translocation 1 (PVT1) was inhibited by transfection of primary ASMCs with small int

38 However, promoter release was inhibited in the presence of dUTP or dUMP.

39 Finally, CXCL10-increased MMP-9 secretion was inhibited by methylprednisolone and also by compound A, a

40 Pressure-induced secretion was inhibited by the mechanosensitive ion channel antagonists

41 Stimulation also produced drinking behaviour that was inhibited as water was ingested, suggesting pre-systemic

42 trations of H2O2/Zn(2+) induced substantial cell death that was inhibited by PJ34 and DPQ, PARP inhibitors, 2-APB, a TRPM

43 e adhesion molecules by BMP9/10 in the presence of TNFalpha was inhibited by LDN193189, which inhibits ALK2 but not ALK1.

44 ntensity white light was reduced when polar auxin transport was inhibited.

45 However, migration of fs120, but not fs188 cells, in vitro was inhibited by B20-4.1.1.

46 f HIPK2 along with an increase in the protein amount, which was inhibited by p300 RNAi.

47 reases in cytosolic Ca(2+) concentration ([Ca(2+)]c), which was inhibited by PJ34, a PARP inhibitor, and abolished by TRP

48 y at 24 h in renal proximal tubular epithelial cells, which was inhibited by sodium hydrosulfide (NaHS), a source of H2S.

49 effect on response to TNFalpha stimuli was detected, which was inhibited by vitamin D.

50 In this in vitro system, cross-linking of P-X was inhibited by itraconazole, but not by U18666A.