ライフサイエンスコーパス: was responsible for

1 xperiments confirmed that the hematopoietic pool of miR-223 was responsible for differences in thrombosis times.

2 Consistently, miR-142-3p was responsible for the glutamatergic synaptic alterations ca

3 Instead, beta2 adrenoceptor activation was responsible for mediating gene silencing of IL-27p28 and

4 Over the course of a week, osteoclast activity was responsible for resorbing the necrotic bone, which in tur

5 ealed that disruption of apical junctional complexes (AJCs) was responsible for PH in Nestin-Cre/Llgl1(fl/fl) brains.

6 ssays indicated that 5'-nucleotidase (5NT), rather than AP, was responsible for ATP hydrolysis.

7 chanism showed a mixture of cell cycle arrest and apoptosis was responsible for the observed decrease in cell viability.

8 notype had not previously been identified in Liverpool, but was responsible for 32% (14/44) of all iGAS cases reported du

9 ction (PCR) to demonstrate that Angiostrongylus cantonensis was responsible for 67.3% of 55 cases of eosinophilic meningi

10 Individually, high BMI (544 300 cases) was responsible for twice as many cancer cases as diabetes (2

11 revealed that induction of the cell cycle inhibitor Cdkn1a was responsible for the decreased proliferation in the knocko

12 he Col1a1 gene, encoding the type I collagen, alpha1 chain, was responsible for the phenotype.

13 In 2011, CVD was responsible for 31% of all deaths, with ischemic heart di

14 In all cases, the presence of E186 was responsible for the control of host gene expression.

15 substitution (A120G) in the N-terminal region of eIF2Bbeta was responsible for the TuMV resistance.

16 gnal-regulated kinase signaling in Wolffian duct epithelium was responsible for the retention of male structures in an an

17 RIN B-LIKE PROTEIN 10 (SlCBL10) gene whose lack of function was responsible for the severe alterations observed in the sh

18 emory T (TEM) cells that secrete IL-17A, but not IFN-gamma, was responsible for early IL-17A production.

19 ther than in the hematopoietic system, and during gestation was responsible for the low-fear offspring phenotype.

20 Foam cell infiltration was responsible for 70% of false IVOCT TCFA and caused both t

21 -derived macrophages stimulated with B. burgdorferi, and it was responsible for feed-forward amplification of IFN-stimula

22 Historically it was responsible for three pandemics: the Plague of Justinian

23 Trimeric LHCII was responsible for the slowly activated quenching component

24 ds into a new light, as we cannot exclude that this lineage was responsible for the production of Acheulean or Middle Sto

25 The PE encapsulation in the liposomes was responsible for changes in the dynamics of specific regio

26 that an alternate nucleophilic aryl substitution mechanism was responsible for the rapid reaction of the faster substrat

27 Globally, cryptococcal meningitis was responsible for 15% of AIDS-related deaths (95% CI 10-19)

28 ression of the gene nfnB coding for the nitroreductase NfnB was responsible for the natural resistance of M. smegmatis ag

29 ceptional atmospheric ridge, centred over the Arctic Ocean, was responsible for a poleward shift of runoff, albedo and su

30 erents, we sought to determine whether upregulation of P2Y1 was responsible for the observed alterations in muscle affere

31 PAF1 was responsible for antirepressive activities of ENL in vitro

32 Furthermore, we demonstrated that the AhR-IDO pathway was responsible for the preferential activation of non-canoni

33 Further, we found that the ubiquitin/proteasome pathway was responsible for MeCP2 T158M degradation and that proteaso

34 The activation of this pathway was responsible for the accumulation of tau oligomers after T

35 ility of the intermediates required for protein prenylation was responsible for decreased gammaherpesvirus replication in

36 Whereas the activation of GABAA receptors was responsible for the direct inhibition of light-evoked spi

37 ncreased enkephalin tone on presynaptic mu opioid receptors was responsible for occluding the LTD.

38 crotubule dynamics, confirmed that centrosome repositioning was responsible for further cell disengagement and scattering

39 Instead, reduced activity of Rac/RhoG was responsible for the deficient macropinocytosis of proinfl

40 Scabies was responsible for 0.21% of DALYs from all conditions studie

41 Microbial enzymatic chain shortening was responsible for a shift in ethoxymer molecular weight dis

42 No single life skill was responsible for the associations we observed, nor were th

43 ntial [AP], variation potential, and system potential [SP]) was responsible for this course of action.

44 In contrast, a long receptor residence time was responsible for the prolonged effects caused by buserelin

45 ynthase, NO production, and p38 MAPK pathway, which in turn was responsible for the increased H3K9me2/3 in CIITA via up-r

46 covered that insufficient NADPH due to GLS2 underexpression was responsible for the delayed metabolism of 5-ALA and accum

47 restored by a single amino acid substitution (K186E), which was responsible for NS1 binding to the host factor CPSF30.

48 rved Ser/Thr cluster in the more proximal C-terminus, which was responsible for the beta-arrestin- and GPCR kinase-depend

49 wo cell lines, miR-155 upregulation, which is common in WM, was responsible for the inhibition of FOXO3a and Bim expressi

50 The Asian lineage of ZIKV was responsible for the recent epidemics in the Americas.