ライフサイエンスコーパス: was responsible for

1 ubiquitination and degradation, and hence the loss of Cav-1 was responsible for reducing the caveolae numbers.

2 Ultrasound at 7000 J g(-1) was responsible for an increase in soluble solids (16%), vita

3 wo major Salmonella pathogenicity islands, SPI-1 and SPI-2, was responsible for biofilm-induced changes in host physiolog

4 UVB exposure to promote both double-stranded RNA and LL-37 was responsible for the endothelial response to keratinocytes

5 A mechanism involving NOS activity was responsible for the observed phenomena.

6 evated inflammatory environment, and not chronological age, was responsible for the decrease in SSPC number and function.

7 To determine why aggregation was responsible for this phenomenon, the integrated stress re

8 demonstrated that only one allele was highly expressed and was responsible for the production of 2-phenylethanol.

9 t activated the beta-catenin pathway in periportal HPCs and was responsible for their expansion and de-differentiation.

10 d that catp-6, a Caenorhabditis elegans homolog of ATP13A2, was responsible for lysosomal Ca(2+) accumulation-an example

11 Subsequently, a RAD18-SLF1 axis was responsible for initiating DNA synthesis in a manner that

12 This change was responsible for increased replication and higher morbidit

13 ather than phosphorylation of other kinetochore components, was responsible for this dissociation.

14 This butyrate concentration was responsible for the maximum modulatory effects observed i

15 cial tension which was affected by the solute concentration was responsible for the velocity difference.

16 Smoking-related disease was responsible for about 41 million deaths in the United Sta

17 TRPV4-dependent sustained [Ca(2+)](i) elevation was responsible for fluid shear stress-mediated and Piezo1-me

18 e, diethyl succinate, hydroxylinalool, and 2-phenyl ethanol was responsible for describing the BRS Rubea wines as fruity/

19 -12 production, rather than inhibitory receptor expression, was responsible for inhibition of T-cell proliferation.

20 Fibrinogen was responsible for a second distinct mechanism of neutrophil

21 the reductions in disease burden, whereas population growth was responsible for most of the increases.

22 ations confirmed that the mutation at residue 217 in the HA was responsible for mediating changes to the immunological pr

23 Brain imaging revealed that the hippocampus was responsible for this interference between statistical lea

24 den of cardiovascular disease associated with HCV infection was responsible for 1.5 million DALYs, with the highest burde

25 in a MF system to the lottery they retrospectively inferred was responsible for this outcome.

26 ations, we confirmed that a temperature rise of up to ~30 K was responsible for current enhancements observed for mass tr

27 d evidence that the same causal variant at 12 of these loci was responsible for variation in both protein and neurologica

28 ether increased oxidative stress in Atg7 (Delta/Delta) mice was responsible for p53 activation, Atg7 was deleted in the p

29 To confirm that the increased Notch3 signaling in mgR mice was responsible for structure alterations in the lungs, mice

30 hat LFNG modification of O-fucose on EGF8 and -12 of NOTCH2 was responsible for enhancement of DLL1-NOTCH2 activation, si

31 We observed previously that partial oxidation was responsible for increased TK(high) activity, while low-ac

32 A cGMP-activated phosphodiesterase (AtCN-PDE1) was responsible for the UVA-induced decrease in cGMP in Arabi

33 Pneumococcus was responsible for most infections (67.3%: 66/98), followed

34 masked to the treatment group while the unmasked programmer was responsible for programming and optimisation of device se

35 Regardless of age, pyrazinamide was responsible for the majority of adverse reactions.

36 A C-terminal IQGAP1 region was responsible for its association with OX40, and TNFR-assoc

37 nic response on RV pulsatile load: the LH harmonic response was responsible for a 20% reduction of pulse pulmonary artery

38 (YI) and more specifically a sublineage (1) of this strain was responsible for the increase of serogroup Y IMD in Sweden

39 The 4-Cl substituent was responsible for higher rates of light absorption and the

40 nit attached to a biphenyl carbamate, 5-fluoro substitution was responsible for M(3) subtype selectivity over M(2), while

41 strogen caused an extension of sensitivity and testosterone was responsible for a decrease in the duration of the hyperal

42 ese four datasets identified a region in chromosome 17 that was responsible for regulating the response.

43 We observed commissural thinning that was responsible for brain dysconnectivity, recapitulating TSC

44 earning induced a lasting increase in GABA release and this was responsible for driving the change in endocannabinoid deg

45 der to determine whether this reduction in alanine turnover was responsible for the reduced rates of hepatic mitochondria

46 ic basis for this "fitness." A single mutation in the virus was responsible for greater fitness, enabling high growth of

47 NOB, while during a steady state, it was mainly FNA, which was responsible for inhibitory effects due to the accumulatio

48 ential vanilloid subfamily 4 (TRPV4) channel opening, which was responsible for the sustained elevation in intracellular

49 of confusion was an inadequately performed time out, which was responsible for nearly one third of all surgical confusio

50 The Zika virus (ZIKV) was responsible for a recent debilitating epidemic that till