ライフサイエンスコーパス: water deprivation

1 tion), but a beneficial effect in countering water deprivation.

2 in systemic blood pressure during periods of water deprivation.

3 ges1(-/-) mice at baseline and after 12 h of water deprivation.

4 e LHA that was not evident after 24 hours of water deprivation.

5 n includes the neurons that are activated by water deprivation.

6 ed stimulus (CS) through different levels of water deprivation.

7 26A7 are differentially regulated in OMCD in water deprivation.

8 proximately 55% (P < 0.05 versus control) in water deprivation.

9 ained polyuric after DDAVP administration or water deprivation.

10 thalamo-neurohypophysial system (HNS) during water deprivation.

11 +/- 59 mOsm) did not change from that before water deprivation.

12 bolic products in T. triangulare leaves upon water deprivation.

13 food) or furosemide; or (2) antidiuretic by water deprivation.

14 le to concentrate their urine in response to water deprivation.

15 mmunohistological assessment of reactions to water deprivation.

16 leus) suppressed water intake following 24 h water deprivation.

17 egulation of AE1 and SLC26A7 was examined in water deprivation, a condition known to increase the osm

18 nents of task performance, such as handling, water deprivation, access to water used as a reward, or

19 Water deprivation also increases renal NF-kappaB-driven

20 mature kidney is appropriately stimulated by water deprivation and dDAVP.

21 These results demonstrate that water deprivation and hypertonicity activate NF-kappaB.

22 ion is enhanced further by osmotic stresses (water deprivation and salinity).

23 neurones are synaptically influenced during water deprivation, and that these neurones differentiall

24 corticoid-deficient (GD) rats in response to water deprivation as compared with control rats.

25 Physical water deprivation at the midpoint level is assessed in w

26 During water deprivation, body water preservation is ensured by

27 and heterozygous mice remained active after water deprivation, body weight decreased by 20-22%, seru

28 io, and second, characterization factors for water deprivation (CFWD) are calculated, integrating the

29 4%) RVLM-projecting PVH neurons activated by water deprivation contained VGLUT2 mRNA.

30 In response to water deprivation, COX2, but not COX1, mRNA levels incre

31 < 0.025), whereas urine osmolalities before water deprivation did not differ among the genotypes.

32 renal osmotic gradient by submitting mice to water deprivation, diuretic administration, or high-Na(+

33 uds in order to survive low temperatures and water deprivation during winter.

34 NKCC2A-/- compared with wild-type mice, but water deprivation elevated urine osmolarity to similar l

35 creased basal Purea in initial IMCD, whereas water deprivation for 1 d increased AVP-stimulated Purea

36 Water deprivation for 1 or 3 d had no effect on basal or

37 Water deprivation for 2 to 3 d, but not for 1 d, signifi

38 came severely dehydrated and lethargic after water deprivation for 36 h.

39 In response to 36-h water deprivation, GD rats demonstrated significantly gr

40 ulated Purea in the IMCD3 subsegment; 1 d of water deprivation had no effect on basal or AVP-stimulat

41 However, with 36 h of water deprivation, HT rats demonstrated significantly gr

42 e response associated with overnight food or water deprivation in male mice.

43 abeling to characterize neurons activated by water deprivation in the hypothalamic median preoptic nu

44 tion upon increased salt and water intake or water deprivation, indicating that this posttranslationa

45 nchanged in the kidney cortex and stomach in water deprivation, indicating the specificity of SLC26A7

46 In rabbits treated with SC58236, water deprivation induced apoptosis of medullary interst

47 whether the PVH-RVLM projection activated by water deprivation is glutamatergic and/or contains vasop

48 Seventy-two hours of water deprivation lead to further increases in nNOS-like

49 A period of water deprivation leads to increased ability of angioten

50 mRNA expression in rat kidney revealed that water deprivation markedly increases the relative abunda

51 One mechanism by which water deprivation may increase the sympathetic outflow i

52 determine the effects of 24- and 72-hours of water deprivation on nNOS protein expression within the

53 tive and do not have access to water, 6 h of water deprivation only slightly affected Fos-LIR.

54 Water deprivation or administration of the VR agonist dD

55 peripheral hyperosmolality caused by either water deprivation or injections of hypertonic saline.

56 s) suppressed drinking following either 22 h water deprivation or intragastric injection of hypertoni

57 urinary concentrating ability in response to water deprivation or treatment with a vasopressin analog

58 increase in urine osmolality in response to water deprivation or vasopressin administration, however

59 en these animals are normally active, 6 h of water deprivation produced near-maximal increases in the

60 Water deprivation produces a drive to seek and consume w

61 Although 48-h water deprivation resulted in comparable rises in plasma

62 In contrast, 3 d of water deprivation significantly increased both basal and

63 indicators of sugar export, measured during water deprivation, suggested sugar export maintained by

64 derive midpoint characterization factors for water deprivation taking into account downstream cascade

65 istant to high concentrations of NaCl and to water deprivation than the isogenic wild-type strains.

66 se mice have a daily cycle of sensitivity to water deprivation that is demonstrated by both behaviora

67 o-8-d-arginine-vasopressin administration or water deprivation, the AQP3 null mice were able to conce

68 three children and further increased during water deprivation to as high as 30 times normal.

69 ring the day, it required as much as 12 h of water deprivation to produce increases in Fos-LIR cells

70 Using a slow-onset water deprivation treatment in Arabidopsis (Arabidopsis

71 After 24 h of water deprivation, U(osm) (in mosM/kg H(2)O) was 2403 +/

72 Maximum urine osmolality after a 36-h water deprivation was (in mosM, n = 15) [+/+] 3,342+/-20

73 n-2 protein in inner and outer medulla after water deprivation were significantly lower in CD-KO mice

74 s response (e.g., to salt, osmotic stress or water deprivation) were the most relatively abundant gro

75 Rats were subjected to 3 d of water deprivation while having free access to food.

76 to either subcutaneous hypertonic saline or water deprivation with partial rehydration than did vehi