ライフサイエンスコーパス: water intake

1 ts of ANG II on cardiovascular functions and water intake.

2 n homozygous individuals and decreased their water intake.

3 months, which was paralleled by an increased water intake.

4 T knockdown mutant mice have higher food and water intake.

5 rtension, cardiac hypertrophy, and increased water intake.

6 % (+/-69%) increase in NAcc DA compared with water intake.

7 ssible sites were ineffective at suppressing water intake.

8 Successful NIL-D was confirmed by increased water intake.

9 the non-NMDA receptor antagonists to induce water intake.

10 tense, dose-related feeding without altering water intake.

11 along the southern shoreline past the Toledo water intake.

12 ntribute to long term adaptation to variable water intake.

13 rther structural instabilities and increased water intake.

14 tributed 12% and 10%, respectively, to total water intake.

15 t suppressed sucrose intake had no effect on water intake.

16 and sucrose solutions, but do not potentiate water intake.

17 er drinking, whereas raclopride also reduced water intake.

18 on in the NAc shell did not alter sucrose or water intake.

19 ypotensive drug that typically produces only water intake.

20 allow those prone to it to liberalize their water intake.

21 dilute urine, allowing for a broad range of water intake.

22 nhibited isoproterenol-, but not PEG-induced water intakes.

23 higher beverage and food moisture and total water intakes.

24 dy weight (7-17 g), food intake (8-13 g) and water intake (11-23 ml), while the vehicle or MS1 condit

25 ynaptic dopamine on (1) spontaneous food and water intake, (2) incentive motivation and learning to o

26 significantly increased deprivation-induced water intake (256%) over a 60 min time course.

27 n sham feeding rats, and deprivation-induced water intake (51%) in sham drinking rats.

28 corresponding controls in sucrose, chow, or water intake across a range (0.0001-20%) of sucrose conc

29 ng at baseline and a significant decrease in water intake after administration of AdCre/adenovirus en

30 creases in home-cage ambulatory activity and water intake after exposure to shock than did swim-test

31 y (PRA) and found that weight loss decreased water intake after Iso, but had no effect on PRA.

32 tremic rats exhibited a triphasic pattern of water intake after SC, with peak intakes 3 times higher

33 whereas lesions of the MnPO or LPO increased water intake after the treatment.

34 fection risk as a function of grass, soil or water intake, age of carcass sites, and the exposure req

35 responded well with the measured decrease in water intake and an increase in urine volume with surplu

36 m neurons that induce a parallel increase in water intake and arginine vasopressin (AVP) secretion to

37 Lipopolysaccharide (LPS) decreased food/water intake and body weight in ethanol-naive and ethano

38 ection of physostigmine into the SFO induced water intake and c-fos expression in the AV3V area as we

39 into the LHA on the stimulation of food and water intake and c-Fos expression.

40 ANP was found to inhibit the water intake and corticosterone release induced by i.c.v

41 on of xerostomia was determined by increased water intake and decreased salivary flow rate.

42 or antagonist, losartan, to reverse both the water intake and Fos-immunoreactivity (Fos-ir) induced i

43 rtan to assess the contribution of Ang II to water intake and Fos-ir responses to peripheral injectio

44 weight status modifies the relation between water intake and hydration status.

45 el, obesity modifies the association between water intake and hydration status.

46 ut vitamin C did not affect maternal food or water intake and led to a significant increase in matern

47 Daily water intake and plasma glucose and insulin were not cha

48 ned the effect of weight loss on Iso-induced water intake and plasma renin activity (PRA) and found t

49 Daily drinking water intake and rice consumption rate distributions wer

50 R-null mice, leading to a marked increase in water intake and salt appetite.

51 (SFO) has been suggested to be important for water intake and secretion of vasopressin (AVP).

52 rtant role in the hypothalamic regulation of water intake and the endocrine axis, in the regulation o

53 We evaluated the relation of plain-water intake and the substitution of plain water for SSB

54 roduced a marked increase in food intake and water intake and this effect was completely reversed by

55 hypohydration between subjects with adequate water intake and those with low water intake was 0.56 (9

56 VDR-null mice still maintained the increased water intake and urinary output.

57 We examined how the association between water intake and urine osmolality, which is a hydration

58 altered renal water handling, with decreased water intake and urine volume, alongside higher urine os

59 This was accompanied by reduced daily water intake and urine volume, as well as increased urin

60 , we investigated the regulation of food and water intake and weight loss following BNST PACAP infusi

61 In the brain AM inhibits water intake and, in a physiologically relevant manner,

62 ribution of iAs exposure rates from drinking water intakes and rice consumption in the U.S. populatio

63 4-h spontaneous ambulatory activity and food/water intake) and several aspects of brain catecholamine

64 BDNF significantly decreased food and water intake, and body weight gain.

65 Food intake, water intake, and body weight were monitored daily.

66 es in GLP-1-associated gene expression after water intake, and compared the effects of fluid intake t

67 act (commNTS) on body weight, daily food and water intake, and plasma glucose and insulin in rats.

68 e fluctuation, metabolism, food consumption, water intake, and renal salt and water excretion.

69 Ang II-induced increases in blood pressure, water intake, and sympathoadrenal catecholamine release;

70 The detection of water and the regulation of water intake are essential for animals to maintain prope

71 Self-reported change in water intake at 6 months was greater in the water group

72 hly diluted urine) and polydipsia (increased water intake), both features of diabetes insipidus.

73 ly in the highest quartile of total or plain water intake but did not approach the Bonferroni-correct

74 nist, losartan (1 microgram/200 nl), reduced water intake but not 0.3 M NaCl intake induced by subcut

75 II into the lateral ventricle (LV) increases water intake, but a similar response is not observed aft

76 ogen reduces angiotensin II (Ang II)-induced water intake by ovariectomized rats.

77 Similarly enhanced water intake by rats with APX also was observed when mar

78 Instead, a pattern of reduced food and water intake, combined with feces replete with lipid and

79 We used public-domain, mortality-linked water intake data from the NHANES conducted in 1988-1994

80 Although water intake decreased there was no reduction in total f

81 Non-breast milk oral water intake did not differ by group (P = 0.39) and was

82 Various contributors of total water intake differed in their association with dietary

83 d approach to identify Pennsylvania drinking water intakes downstream of wet FGD discharges and to as

84 Rats reduced water intake during 24 h HU in all conditions.

85 g sham feeding or deprivation (24 h)-induced water intake during sham drinking in rats with gastric f

86 Food and water intake, feeding microstructure, and general motor

87 ventral median preoptic nucleus) suppressed water intake following 24 h water deprivation.

88 ns involved in blood pressure regulation and water intake following dehydration.

89 Here, we show that water intake, food consumption, stool weight, urine volu

90 ke Erie, including a station proximal to the water intake for the city of Toledo.

91 The percentage of total water intake from plain water increased with age.

92 al actions, including inhibition of food and water intake, gastric emptying, and stimulation of neuro

93 Subsequent experiments showed that water intake had a selective effect on central GLP-1-rel

94 reases in bromide concentrations at drinking water intakes; however, similar low flow conditions in s

95 can be accomplished with increased salt and water intake in conjunction with fludrocortisone.

96 ge in association with higher total or plain water intake in men or women in this national cohort.

97 tions also significantly stimulated chow and water intake in nonfood-deprived rats.

98 tor subtype mediation of deprivation-induced water intake in real drinking and sham drinking conditio

99 Deprivation-induced water intake in sham drinking rats was significantly red

100 e that weight loss decreases Ang II-elicited water intake in the female rat by down-regulating the ex

101 y decreased body weight, as well as food and water intake in the first 24 h following infusion.

102 ve systematically examined the correlates of water intake in the US population.

103 ons neither altered micturition patterns nor water intake in the young adult rat.

104 The water intake induced by DNQX was also blocked by pretrea

105 rption but rather is the result of increased water intake induced by the increase in systemic and bra

106 atrial junction (SVC-RAJ) reduces sodium or water intake induced by various experimental procedures

107 Water intake induces chemical shift changes up to 2 and

108 thods were developed to study or to quantify water intake into starch-based matrices.

109 Adequate water intake is critical to physiologic and cognitive fu

110 studies provide evidence that Ang II-induced water intake is mediated via the classical G protein cou

111 How food and water intake is reciprocally regulated to maintain homeo

112 y noted in women with higher total and plain water intakes may be spurious and requires further inves

113 carbohydrates as well as deprivation-induced water intake (mu) under real-feeding and real-drinking c

114 In men, neither total water intake nor each of the individual water source var

115 Intra-vmPFC DAMGO affected neither water intake nor nonspecific oral behavior.

116 In both conditions, the increased water intake occurred within the first 15 min of the dri

117 lacked free access to water, 74% had enteral water intake of less than 1 L/d, and 94% received less t

118 ontrast, hyponatremic SC rats exhibited peak water intakes of 600 ml/24 hr, approximately 9-10 times

119 f 600 ml/24 hr, approximately 9-10 times the water intakes of sham-operated normonatremic rats.

120 ted rats with 100 mg/kg of captopril reduced water intake only during the initial 15 min after a gava

121 a fat-rich diet, with no changes in chow and water intake or body weight.

122 osses and impaired thirst or restricted free water intake or both.

123 rttin palmitoylation upon increased salt and water intake or water deprivation, indicating that this

124 ial contamination of water sources with MAP, water intake, or water treatment.

125 d significant increases in Br/Cl at drinking water intakes over the first year of the study (2009-201

126 Total water intake (P = 0.002), urine osmolality (P < 0.001),

127 Water intake partially decreased ANG I-induced Fos-ir in

128 a slight but not significant suppression of water intake, particularly after the higher dose.

129 Plain-water intake, per se, was not significantly associated w

130 SFO lesions reduced water intake regardless of hydration condition.

131 that receptor number is more critical to the water intake response than the saline intake response, o

132 e repeated injections, however, decrease the water intake response to Ang II without affecting saline

133 the G protein mediate desensitization of the water intake response.

134 cise in the heat, sweat output often exceeds water intake, resulting in a body water deficit (hypohyd

135 de facto reuse is analyzed for 2056 surface water intakes serving 1210 DWTPs across the U.S.A. that

136 despite normal activity levels and food and water intake, Smn deficiency caused constipation, delaye

137 onsumption was accompanied by an increase in water intake, so that the total volume of liquid consume

138 I in samples collected at WWTPs and drinking water intakes (source water) during one year were quanti

139 Interactions between weight status and water intake status were significant in linear (P = 0.00

140 e showed age-dependent increases in food and water intake, stomach and body weights, and decreases in

141 f brain sites implicated in hunger, salt and water intake, stress, arousal, and reward.

142 r chemistry sensors embedded in a high-speed water intake system to document spatial variability.

143 elopment of hyponatremia even in the face of water intake that can approach 20 L/d.

144 nterventions, differentiated by advice about water intake (the water group received advice to increas

145 forebrain systems mediating other aspects of water intake, the authors examined the effects of lesion

146 sertion, it minimized peptide clustering and water intake through stabilization of the bilayer struct

147 and behavioral support to increase habitual water intake to 8 cups per day.

148 (the water group received advice to increase water intake to 8 cups per day; the control group did no

149 Restriction of water intake to increase vasopressin levels also signifi

150 sin II type 1 receptor antagonist normalized water intake, urinary volume, and c-Fos expression in VD

151 Body weight, water intake, urine output, solute and urea excretion, s

152 sured, including arterial pressure, food and water intake, urine volume, and sodium and potassium exc

153 inergic receptor agonist, carbachol, induces water intake, vasopressin (VP) release and an acute incr

154 ith adequate water intake and those with low water intake was 0.56 (95% CI: 0.43, 0.73) in adults who

155 previous 24 h (in 1999-2004, estimated total water intake was 3.35 L), with plain water and beverages

156 Total water intake was determined with the use of a 24-h dieta

157 Plain water intake was inversely associated with the intake of

158 Total water intake was inversely related to energy from fat an

159 vels of ethanol in dialysates from the NAcc; water intake was negligible.

160 Plain-water intake was not associated with T2D risk in the mul

161 , a significant increase in urine volume and water intake was observed; urine volume rose from 9.5+/-

162 Plain water intake was unrelated to the intake of energy and b

163 , which is known to associate with increased water intake, was increased in the hypothalamic paravent

164 ptor function as measured by agonist-induced water intake, was significantly attenuated in these rats

165 Ad libitum water consumption, total water intake, water output through urine, total water ou

166 Alcohol intake, body weight, and water intake were measured at 24 h post-injection interv

167 Body weight and spontaneous food and water intake were monitored daily.

168 Animal weight and water intake were not altered during 10 days of EPEC inf

169 cial defeat protocol, body weights, food and water intake were recorded, depression and anxiety-like

170 Urine volume and water intake were unchanged in all other groups.

171 highest levels of genetic markers at the raw water intakes were associated with human fecal sources (

172 Body weight, kidney weight, and food and water intakes were not different from WT littermates.

173 Water intakes were unaffected by the higher dose of losa

174 Baclofen had no effect on water or sugar water intake when administered to anterior or posterior

175 rPP and NPY significantly increased food and water intake when they were administered into the LHA, a

176 AngIII produced a dose-dependent increase in water intake, whereas saline intake was equivalently inc

177 ed architecture predetermines the pattern of water intake, which sets the stage for the orchestrated

178 udies have examined the association of total water intake with all-cause mortality.

179 out the association of contributors of total water intake with dietary characteristics in US children

180 o examine the association of contributors of water intake with dietary characteristics, meal consumpt

181 ilar rate of increase in locomotion and food/water intake with time.