ライフサイエンスコーパス: wave front

1 er, maintains VF by generating new reentrant wave front.

2 sary to propagate a mechanically coordinated wave front.

3 was activated predominantly by the superior wave front.

4 the concave shape of the Ca2+ fertilization wave front.

5 mapped area displacing the entire reentrant wave front.

6 lided with the leading edge of the reentrant wave front.

7 sed current load experienced by the pivoting wave front.

8 e border or by collision with an outside new wave front.

9 due to higher rates of genetic drift on the wave front.

10 of these 3 patterns could occur in the same wave front.

11 magnetic susceptibility gradient across the wave front.

12 lion cell genesis behind the differentiation wave front.

13 quency of deleterious mutations on expanding wave fronts.

14 fronts and multiple simultaneous-activation wave fronts.

15 t role in re-formation of unstable reentrant wave fronts.

16 erns, or varying orientations of circulating wave fronts.

17 functional conduction block and circulating wave fronts.

18 cycle length, or core size of the reentrant wave fronts.

19 ained by both focal and incomplete reentrant wave fronts.

20 but broad spectra in AF, reflecting multiple wave fronts.

21 greater negative curvature of the activation wave front (-1.86+/-0.40 mm in chimeric mice versus -0.8

22 low waves, propagated without decrement as a wave front (2-5 mm/s) through the ICC-SM network lying a

23 second wave front met the tail of the first wave front 69 +/- 11 ms (range, 40 to 90 ms) after its l

24 inear relationship exists between activating wave-front acceleration and deceleration in the SCZ and,

25 lation showed neither reentrant circuits nor wave fronts activating the right atrium via BB.

26 In 342 wave fronts, activation was mapped arising focally.

27 In 142 wave fronts, activation was mapped propagating from PF t

28 In 534 wave fronts, activation was mapped propagating from work

29 The length of the pathway of each wave front along the catheter was 6.5 +/- 4.5 cm.

30 Activation times were grouped into wave fronts along the catheter, and the lengths of the w

31 New technologies reported include wave-front analysis for amblyogenic factors and a visual

32 ions were determined using the Topcon KR-1 W wave-front analyzer.

33 odes were associated with a single reentrant wave front anchored to the PM.

34 n the origin and direction of the excitation wave front and are responsible for polymorphic arrhythmi

35 tex, which means that it possesses a helical wave front and carries orbital angular momentum.

36 side new wave front merged with the original wave front and excited the core.

37 A fraction of mutations travel with the wave front and generate mutant populations that are on a

38 ose ancestors lived mostly on the colonizing wave front and individuals whose ancestors remained in t

39 We experimentally observe the wave front and the distortion of modulated wave packets

40 progression of ganglion cell differentiation wave front and to induce its own expression.

41 cities leading to the self-steepening of the wave front and to the formation of a shock.

42 The number of wave fronts and approximate entropy were significantly (

43 s independent of the rate of invasion of new wave fronts and epicardial breakthroughs.

44 ation were seen, including single-activation wave fronts and multiple simultaneous-activation wave fr

45 sing evidence that both functional reentrant wave fronts and multiple wavelets are present during ven

46 human hearts with DCM, epicardial reentrant wave fronts and transmural scroll waves were present dur

47 aps showed incessantly changing beat-to-beat wave fronts and varying spatiotemporal behavior of drive

48 inal phonon cooling effect on the molten COD wave front, and the formation of patterns due to laser l

49 excitation caused disruption of the original wave front, and the newly formed wave front(s) vanished

50 ent activated first by the paced orthodromic wave front, and were mainly due to local pacing latency,

51 globally synchronized activity, propagating wave fronts, and spiral waves that have been previously

52 quations are used only near action potential wave fronts, and the macroscopic equations are used ever

53 , both the amplitude and phase of an optical wave front are acquired with a dual-plane construct.

54 (2) New reentrant wave fronts are generated when one wave front interacts

55 tablishing segmentation, including clock and wave front, are not perturbed.

56 d to rotate as a single reentrant activation wave front around the TA, and the role of LLR in those p

57 ls of Shh expression are detected behind the wave front as ganglion cells accumulate, while the Patch

58 f naturally occurring unshocked fibrillation wave fronts, at comparable CIs and Vms.

59 tained VT always occurred when the reentrant wave front blocked in the central common pathway in reen

60 ion always occurred when the two circulating wave fronts blocked in the central common pathway (CCP).

61 The distally initiated activation wave front blocks near the S2 site because the same S2-i

62 AFL, reflecting its single macro-re-entrant wave front, but broad spectra in AF, reflecting multiple

63 A single meandering functional reentrant wave front can be induced in human atrial tissues and pr

64 hat a single meandering functional reentrant wave front can result in rapid and irregular electrogram

65 tion was caused by two successive activation wave fronts circulating in the same direction along the

66 ing in a direction opposite to the reentrant wave front collided with the leading edge of the reentra

67 ested by early breakthrough in the lower RA, wave-front collision in the high lateral RA or septum, a

68 ise orientation with early annular break and wave-front collision over the isthmus.

69 Intercycle interval (ICI) and wave front conduction time (WCT) were determined for the

70 he heart', Mines proposed that an activation wave front could propagate repeatedly in a circle, initi

71 Density of wave fronts decreased during both global ischemia (P<0.0

72 As tissue mass was decreased, the number of wave fronts decreased, the life-span of reentrant wave f

73 eterogeneities of repolarization may lead to wave-front destabilizations and initiation of ventricula

74 out of focus light, minimal aberrations and wave-front distortions.

75 The distortion of the shape of the surface wave fronts due to the curvature is corrected with a sui

76 d when one wave front interacts with another wave front during its vulnerable period.

77 We sought to evaluate the characteristics of wave fronts during ventricular fibrillation (VF) in huma

78 The effects of PA on cellular properties and wave front dynamics were reversed during 60 minutes of d

79 areas in which the cathode-break excitation wave fronts easily invaded the spatial excitable gap via

80 All wave fronts emanated from foci and breakthrough sites, a

81 During persistent and LSP AF in 12 patients, wave fronts emanating from foci and breakthrough sites m

82 ventricles at such high frequencies that the wave fronts emanating from them breakup at varying dista

83 If wave fronts ever arise focally during VF, it should be p

84 (1) The reentrant wave front for each morphology rotated around the same l

85 spontaneous wave break and by new reentrant wave front formation.

86 that occur before the arrival of the seismic wave front from the mainshock, which violates causality.

87 s such as rigid gas permeable lenses, custom wave front-guided soft contact lenses, hybrid lenses and

88 These wave fronts had a mean cycle length of 229+/-45 ms (160

89 The roles of Purkinje fibers (PFs) and focal wave fronts, if any, in the maintenance of ventricular f

90 conduction was equal to the native reentrant wave front in completely excitable regions and slower th

91 ented settlement of pioneers directly on the wave front in North America has contributed to low level

92 itable regions and slower than the reentrant wave front in partially excitable regions.

93 (2) In 5 VT episodes, the activation wave front in the CCP abruptly broke across a stable arc

94 onal maps revealed the presence of reentrant wave fronts in 17 of 33 runs of VF in ablated ventricles

95 Knowledge of the pathway common to both wave fronts in figure-8 reentrant circuits (ie, the isth

96 tylcholinoceptors activates propagating Ca2+ wave fronts in oligodendrocytes and that the characteris

97 ) induces nonstationary meandering reentrant wave fronts in the atrium.

98 uent intra-PV conduction blocks and multiple wave fronts in the PVs were recorded during 10 AF episod

99 Interaction between wave fronts in the right and left atrium may be importan

100 In conclusion, a decrease in the number of wave fronts in ventricular fibrillation by tissue mass r

101 lesion (n = 8), the induced single reentrant wave front, in the form of a spiral wave, meandered irre

102 fronts decreased, the life-span of reentrant wave fronts increased, and the cycle length, the diastol

103 showed that the PV was activated by a focal wave front independent of left atrial (LA) activation in

104 Postshock activation times and wave-front interaction patterns were determined with an

105 reentrant wave fronts are generated when one wave front interacts with another wave front during its

106 ocated by the site of entry of the premature wave front into the circuit.

107 the translocation of Ca2+ from the spreading wave front into the SR.

108 gesting that the leading edge of the reentry wave front is not always at the TA.

109 amic strategy treating sites on the epidemic wave front leads to optimal performance.

110 whether excitation of the core by an outside wave front leads to termination of the reentry in the at

111 o provides a natural anchor to the reentrant wave front, lengthening the life span of reentry.

112 odes, reentry terminated when an outside new wave front merged with the original wave front and excit

113 The second wave front met the tail of the first wave front 69 +/- 1

114 uring rapid ventricular stimulation by AC, a wave front might encounter the refractory tail of an ear

115 um) observed during AF were reactivated by a wave front most often coming from the atrial septum via

116 as the result of interference by an invading wave front (n = 19 or meandered off the mapped region (n

117 vations because of interference with outside wave fronts (n=5) or spontaneous separation of waves fro

118 posite polarities, we detected a propagating wave front of activity that originated at the cortical l

119 lates the fate of mutations occurring at the wave front of an expanding population.

120 A broad wave front of atrial activation was recorded in 17 patie

121 neuroepithelium peripheral to the expanding wave front of differentiation.

122 s from the central to peripheral retina as a wave front of differentiation.

123 During the third instar, a wave front of enhanced Notch activity progressing over t

124 anterior barrier constraining the reentrant wave front of human counterclockwise atrial flutter.

125 design of artificial meta-atoms to shape the wave front of light by optimal control of both its phase

126 of data that were analyzed, 1018 PF or focal wave fronts of activation were identified.

127 elimination of most, if not all, propagating wave fronts of electrical activation by a shock constitu

128 ing ventricular fibrillation (VF), reentrant wave fronts often transiently anchored to the PM.

129 tation, VF consists primarily of a few large wave fronts on the endocardium.

130 humans, consisting primarily of a few large wave fronts on the order of 6 to 9 cm.

131 front velocity and could be used to predict wave front orientation with respect to the surface.

132 rior to superior regions, whereas 2 opposing wave fronts originated on the left septum in both the su

133 In acute AF, a single broad wave front originating from the posterior and medial atr

134 eentrant impulse circulated or was caused by wave fronts originating outside the reentrant circuit.

135 apex, V(m) was expected to exhibit circular wave front patterns and B(z) to reflect the circular com

136 and advance for many millimeters as a sharp wave front perpendicular to the pial surface, at speeds

137 (32.8 +/- 6.5 mm, P < .02), indicating that wave-front pivoting was primarily responsible for shorte

138 Abrupt changes in loading during wave-front pivoting, rather than membrane ionic properti

139 quences expected to result in differentiated wave-front populations with low genetic variation and po

140 The increase in [Ca(2+)](i) at the wave front preceded depletion of the SR at each point al

141 , activation of the septum was discordant; 1 wave front propagated rapidly on the right septum from i

142 nus rhythm and superior right septal pacing, wave fronts propagated predominantly from superior to in

143 Although caffeine-evoked Ca(2+) wave fronts propagated throughout the cell, in most cell

144 The Ca(2+) wave fronts propagated with constant amplitude; the spre

145 ntry terminated abruptly when an outside new wave front propagating in a direction opposite to the re

146 VF maintenance, it should be possible to map wave fronts propagating from PFs into the working ventri

147 patially distributed reaction can support pH wave fronts propagating with a speed of the order of 0.1

148 ropagating in a negative-index material have wave-front propagation (wavevector, k) opposite in direc

149 orientation in the pulmonary veins (PVs) in wave-front propagation are poorly understood.

150 f shock-depolarized areas (BSDAs) from which wave-front propagation could have arisen were identified

151 We conclude that the incidence of postshock wave-front propagation decreases with increasing refract

152 Furthermore, incidences of wave-front propagation following shocks were consistentl

153 ired to guarantee the cessation of continued wave-front propagation in defibrillation.

154 w that plasmonic nanoparticles can produce a wave-front reconstruction when they are sampled on a dif

155 The number of wave fronts recorded by the catheter was 9.2 +/- 2.9 wav

156 The reentrant wave fronts remained stationary and rotated around these

157 Coordinating this wave front requires fast and robust signaling mechanisms

158 encounter the refractory tail of an earlier wave front, resulting in the formation of a wave break a

159 The central core around which the wave front rotated became smaller, which caused shorteni

160 The central area (core) around which the wave fronts rotated had a mean surface area of 12 +/- 3

161 by decreasing the core size around which the wave front rotates.

162 ed by an interaction between two propagating wave fronts roughly perpendicular to each other.

163 he original wave front, and the newly formed wave front(s) vanished at the tissue border within 77 +/

164 nts recorded by the catheter was 9.2 +/- 2.9 wave fronts/s.

165 When clearly seen, the AFL wave front split (n = 3) or turned in the region of the

166 eralization of this strategy to more complex wave-fronts, such as vortex beams that carry orbital ang

167 The integration of wave-front technology and lens-based surgery represents

168 (3) The reentrant wave fronts terminate spontaneously or as the result of

169 The reentrant wave fronts terminated spontaneously (n = 7), as the res

170 n analysis of an expanding population with a wave front that advances at a constant slow rate.

171 ansported in the form of a rapidly advancing wave front that progressed along axons, in a microtubule

172 ion, followed 22+/-15 ms later by propagated wave fronts that arose from the same site.

173 arily by propagation of the stimulus-induced wave fronts that closed up the excitable gap.

174 coordinated fashion, generating contractile wave fronts that propagate through the heart with each b

175 te the propagation of a number of activation wave fronts that reenter to maintain the arrhythmia.

176 ithin the measured range of the speed of the wave fronts, the length of breaking fronts per unit area

177 The "broken" wave front then circulates around both sides of the bloc

178 ering by causing attachment of the reentrant wave front tip to the obstacle.

179 attachment, both conduction velocity of the wave-front tip and wavelength near the obstacle adapted

180 onverts a nonstationary meandering reentrant wave front to a stationary one.

181 e attachment and detachment of the reentrant wave front to and from the ridge determine "flutter-like

182 egative regulator behind the differentiation wave front to control ganglion cell genesis from the com

183 ct excitation of the core that displaces the wave front to the tissue border or by collision with an

184 Wave fronts traveling from one atrium to the other and/o

185 Tumor wave front velocities determined via a marginal stabilit

186 hly sensitive to the transmural component of wave front velocity and could be used to predict wave fr

187 between the interfacial morphology and tumor wave front velocity provides an explicit, testable, clin

188 aps of reentry were constructed to determine wave-front velocity, and piecewise linear adaptive templ

189 n all samples, a single meandering reentrant wave front was induced, causing irregular and rapid bipo

190 t (78.6%) terminated after one of the double wave fronts was blocked in the TA-ER isthmus.

191 By analyzing the wave fronts, we found that the cycle length, refractory

192 Although the circular V(m) wave fronts were detected, the B(z) maps were not as sim

193 s along the catheter, and the lengths of the wave fronts were estimated.

194 ted sinusoidal packets with non-superluminal wave fronts were observed.

195 had their subendocardium ablated, reentrant wave fronts were present in 6 of the 108 runs of VF at b

196 In all episodes, the reentrant wave fronts were spontaneously initiated by an interacti

197 The ECL "wave fronts" were visualized and imaged in space and tim

198 be observed among mutations arising in this wave front where extreme population bottlenecks arise an

199 of a larger number of invader alleles at the wave front, where effective population size is thus incr

200 distance between the head of the circulating wave front, which could be located on the activation map

201 peaks and oscillations were found to be Ca2+ wave fronts, which propagate via distinct amplification

202 set was defined as SVE advanced by the paced wave fronts while atrial signal continued at the tachyca

203 on of the SR at each point along the calcium wave front, while during this latency period a transient

204 ime asymptotic behaviour, where a travelling wave front with a constant speed has been established.

205 correlates with position along the advancing wave front with more divergent lineages near the origin

206 ely caused by the interaction of propagating wave fronts with surface topography.

207 and spectral analysis to identify sequential wave fronts with temporal periodicity and similar spatia

208 thus the conduction velocity (CV) of the AFL wave front within the tricuspid annulus-inferior vena ca

209 ons in the PVs and the VOM, and with complex wave fronts within the PVs.