1 With both conceptual and empirical support,
we argue that three factors-climate heterogeneity, collineari
2 We believe that utilizing registries and biobanks to reveal o
3 From these studies,
we conclude that KSHV uses PPD to fine-tune the temporal expr
4 We conclude that the DFD regimen favors persistence of functi
5 Finally,
we demonstrate that a significant fraction of PSVs in segment
6 Here,
we demonstrate that the Ras->Raf->rho kinase (ROCK) pathway i
7 Using data from patients with focal brain damage,
we demonstrate that there is a strong psychometric correspond
8 We estimate that the incidence rate of emergency department-a
9 We estimate that the neutral density required to produce the
10 We find that all three proteins exhibit restricted rates of e
11 We find that nucleation of the predominant {1 0 -1 2} twinnin
12 We find that short-term high-fat-diet (HFD) feeding of mice a
13 We found that 22 of 26 sequence type (ST) 69 isolates from th
14 Unexpectedly,
we found that 5-OHBza-R is the most favored MtBzaC substrate.
15 In this study,
we found that another key virulence regulator, ToxR, was impo
16 We found that BD CA3 neurons were hyperexcitable only when th
17 samples with annotations generated by three similar tools,
we found that Binner achieves superior performance in the num
18 In this study,
we found that CGD (CYBB-) neutrophils produced higher amounts
19 A catalytic activity did not account for DNMDP sensitivity,
we found that expression of the catalytic domain of PDE3A in
20 an and non-Bayesian methods (LSD, TreeTime, and treedater),
we found that no method performed the best in every scenario.
21 We found that TOGARAM1 variants cause JBTS and disrupt TOGARA
22 Despite the similarity in their enzymatic domain,
we found that ULK1 and ULK2 have major differences in their a
23 We highlight that millions of people around the world depend
24 We hypothesized that clearance of medroxyprogesterone acetate
25 We hypothesized that cognition would demonstrate greater HC-S
26 We identify that a fusion of the catalytic domain of TET1 to
27 We illustrated that batch-effect correction can dramatically
28 Using an inducible expression system,
we observed that this ELANE mutation diminishes enzymatic act
29 We propose that the EPFL2 signaling module evolved to control
30 We report that a conserved bromo-adjacent homology (BAH) modu
31 In the current study,
we report that inhibitors of the DNA damage response kinase A
32 Here,
we report that octopamine activity through the beta-adrenergi
33 Here
we show that beta-cells express abundant Kindlin-2 and deleti
34 Here,
we show that constrained 31-residue-peptides ('msR4Ms') desig
35 Here,
we show that cytotoxic chemotherapies induce dynamic changes
36 Here
we show that dynamically growing somatosensory neurons in the
37 We show that epimutations arise spontaneously at a rate appro
38 Here
we show that keratin intermediate filaments directly regulate
39 ing the melanoma lineage survival oncogene MITF as a model,
we show that low-affinity binding sites act as a competitive
40 We show that NVP was readily taken up by dendritic cells (DCs
41 In addition,
we show that phosphorylation of Ser(3) may be an additional m
42 variation in media markets for 771 matched rural counties,
we show that rural residents are more likely to practice soci
43 polymorphisms can affect RNA secondary structure, and here
we show that single nucleotide polymorphisms can affect RNA-p
44 Here,
we show that SOX11 confers distinct features to ER-negative D
45 We show that the increased cytokinin signaling in ARR1 gain-o
46 We show that the non-canonical tubulin Tuba8, transiently exp
47 Here,
we show that the three major classes of LCCBs activate STIM/O
48 Here,
we show that tree mortality concomitant with drought has led
49 Previously,
we showed that active K-Ras4B dimerizes in silico and in vitr
50 Finally,
we showed that interleukin-4 treatment was sufficient to incr