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1 We asked whether analysis of the genomic context of SASs can
2 We asked whether contaminating phospholipid in myosin prepara
3 We asked whether development of fundamental electrophysiologi
4 We asked whether human vision also exhibits a special sensiti
5 We asked whether mnemonic prediction errors promote hippocamp
6 We asked whether pharmacological stimulation of endogenous ne
7 We asked whether proactive and reactive mothers differed in t
8 We asked whether the same brain regions and neural codes supp
9 We asked whether this could be attributed to aberrant express
10 We asked whether this pathway had an in vivo role in mice.
13 Using the early Drosophila larval brain, we asked whether nutrient-dependent growth of neural stem cel
14 plore what features are sufficient for positional encoding, we asked whether arbitrary molecules (e.g., green fluorescent
36 Given that NKCC1 is critical for ion homeostasis, we asked whether the disruption to myelinated axons in slc12a
38 lity to vaginal colonization and resulting immunopathology, we asked whether estrogen use in the standard VVC model masks
41 xically promote cancer-a phenomenon called "Minority MOMP." We asked whether reflux-induced esophageal carcinogenesis occ
42 ans and cells yet displays a relative lack of neurotropism, we asked whether a chimeric vesicular stomatitis virus (VSV)
45 s a possible application of epidermal metabolite profiling, we asked whether metabolites extracted from the skin surface
48 ethanol requires L1 association with ankyrin-G; therefore, we asked whether NAP promotes the dissociation of ankyrin-G a
49 function and adaptive significance of amylose are unknown, we asked whether there is natural genetic variation in amylos
50 d contributors to this within-mutation disease variability, we asked whether the PRPH2 binding partner rod OS membrane pr