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1 eded the upper boundary for noninferiority, suggesting that we cannot rule out a 20% greater risk of death with intermitt

2 However, we cannot rule out a causal effect of smoking on schizophreni

3 re dominated by pore-pressure changes in the shallow crust, we cannot rule out a longer pore-pressure transient at the de

4 mutants are deficient for only a portion of the hindbrain, we cannot rule out a role for that tissue in otic placode ind

5 associated with poor sanitation practice driving APOs, and we cannot rule out additional confounders, our results demons

6 However, we cannot rule out an association with food sensitization.

7 we do not detect cooperative binding to these sites, though we cannot rule out an independent role for Extradenticle.

8 At this point, we cannot rule out any of the known functions of APC, which i

9 Replication of these findings is needed because we cannot rule out chance or fully rule out bias.

10 rently unpolluted areas from new pollution sources, because we cannot rule out ecological impacts from even relatively sm

11 The enrichment is strongest for schizophrenia, but we cannot rule out enrichment for other phenotypes.

12 not be a net contributor to acetyl-CoA production, although we cannot rule out net reductive IDH flux in some compartment

13 ng protection and acoustic neuroma, especially among women, we cannot rule out recall bias as an alternative explanation.

14 o 1.38) or 6 years (HR 1.12, 95% CI 0.91 to 1.37), although we cannot rule out residual confounding by SEP.

15 Although we cannot rule out reverse causation, these data support the

16 Whereas we cannot rule out that Glu-166 might activate the active sit

17 Although we cannot rule out that surveillance bias has contributed to

18 Therefore, we cannot rule out that the system presented here may be suff

19 Although we cannot rule out the discovery of genes with unusual featur

20 However, we cannot rule out the possibility of there being a causal va

21 However, we cannot rule out the possibility that decreased [11C]raclop

22 As in any observational study, we cannot rule out the possibility that effects in the expose

23 Although we cannot rule out the possibility that neutrophils have an i

24 However, we cannot rule out the possibility that new therapeutic agent

25 t diagnostic factors are driving spatial patterns; however, we cannot rule out the possibility that other environmental f

26 Although we cannot rule out the possibility that other factors might l

27 o the tissue directly from the ventricular cavity; however, we cannot rule out the possibility that other mechanisms of p

28 However, as with any comparative study, we cannot rule out the possibility that our results arise fro

29 le this association may reflect subtle confounding or bias, we cannot rule out the possibility that passive smoke exposur

30 Because of this smaller sample, we cannot rule out the possibility that small numbers of dive

31 However, we cannot rule out the possibility that this association is s

32 Although we cannot rule out the possible existence of tissue-specific

33 Although we cannot rule out the presence of a approximately 100-km-thi

34 nces breakage or recombination within this region; however, we cannot rule out the presence of multiple "hot spots" withi

35 Nevertheless, we cannot rule out the theoretical possibility that such dyna

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