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2 This research was cross-sectional, so we cannot be sure of the temporal sequence of exposure, but t
4 We do not know the extent of this adaptive capacity, so we cannot conclude that phytoplankton will be able to adapt t
5 ed (R = 0.7371, P = 0.0369) with overall survival, although we cannot conclude this was causally related.
6 tribute to AHR in asthma, but, because of high variability, we cannot conclude whether or not asthmatic ASM is hyperreact
8 n using expensive tests and treatments with marginal value: we cannot continue to accept novel therapeutics with very sma
9 nce with laboratory confirmation of B. pertussis infection, we cannot definitively conclude that pertussis disease is wel
10 sign leaves open the possibility of unmeasured confounding, we cannot definitively interpret these results as causal.
11 cripts does not influence measures of enhancer activity and we cannot detect evidence of purifying selection on the resul
12 In the absence of control subjects, we cannot determine whether differences observed in RBM thick
15 lable from diverse geographical areas and time periods, and we cannot discount the potential existence of other unsampled
16 hort, intense interglacials could be missed or blurred, and we cannot distinguish between a remnant ice sheet in the East
17 o-arenium ions are responsible for rearrangements; however, we cannot distinguish between arenium ion and radical cation
18 ta and methodological limitations of the available studies, we cannot draw firm conclusions to inform a population level
23 s associated with nvAMD, but the adjusted OR was small, and we cannot exclude residual confounding.
24 the fossil and archaeological records into a new light, as we cannot exclude that this lineage was responsible for the p
28 a greater degree of self-control" for which we cannot find empirical support in a large data set with dat
29 d from some common ancestor and diverged to the point where we cannot identify the similarity, or have multiple solutions
31 noise, and field of view: with current recording technology we cannot image very large neuronal populations with simultan
32 fant nociceptive processing are completely unknown, meaning we cannot infer anything about the nature of the infant pain
35 Our world is governed by hidden (latent) causes that we cannot observe, but which generate the observations we see
37 loss of classroom time, cost, and lack of lasting benefit, we cannot recommend population-based delivery of Cogmed withi
38 ion requires a 46-49% reduction in sources, indicating that we cannot reconcile the observed amplitude.
39 HGDP and HapMap that are risk alleles for type 2 diabetes, we cannot reject that their distribution is as expected from
41 associated with poor sanitation practice driving APOs, and we cannot rule out additional confounders, our results demons
43 The enrichment is strongest for schizophrenia, but we cannot rule out enrichment for other phenotypes.
44 o 1.38) or 6 years (HR 1.12, 95% CI 0.91 to 1.37), although we cannot rule out residual confounding by SEP.
46 t diagnostic factors are driving spatial patterns; however, we cannot rule out the possibility that other environmental f
47 le this association may reflect subtle confounding or bias, we cannot rule out the possibility that passive smoke exposur
49 g and interpreting motions, it has limited sensitivity, and we cannot see motions that are smaller than some threshold.
50 ill shift the position of these critical boundaries in ways we cannot yet fully predict, but landward migration will be i
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