ライフサイエンスコーパス: we could not

1 the number of fire vehicles on site, and building age, but we could not account for the high levels of BDE-209 observed

2 However, in those experiments we could not address the formation of mutant ribosomes, becau

3 Thus, we could not conclusively detect sequence changes resulting f

4 Because most studies measured BMI after cancer diagnosis, we could not conduct a parallel analysis to adequately evalua

5 <0.00002 methylated cytosines per nonmethylated cytosine), we could not confirm any cytosine DNA methylation in laborato

6 Although we could not confirm that methylation directly inhibits editi

7 SG classification was confirmed in our population; however, we could not confirm the primary site and the level of human

8 In addition, we could not consider haemoglobin in our analysis.

9 Using LFP frequencies <60 Hz, we could not decode the allocation of attention, but we could

10 reaction is essential to the parasite blood stages because we could not delete the Plasmodium berghei gene encoding GatA

11 cohort of patients undergoing repeated radioiodine therapy, we could not demonstrate alterations in salivary parameters.

12 ated virus (GAV), encodes the 2'-O-MTase activity, although we could not detect 2'-O-MTase activity for the homologous pr

13 In contrast, we could not detect any association between rejection and the

14 Furthermore, we could not detect any association between these two protein

15 In live-cell time-lapse microscopy experiments, we could not detect any significant phagocytosis by purified

16 Interestingly, unlike NiV-G/HeV-G, we could not detect binding of GhV-G to ephrinB3.

17 pite our large sample size and multiple datasets available, we could not detect robust associations between specific gene

18 Surprisingly, we could not detect specific antibodies against hemagglutinin

19 ing a knock-in reporter mouse line expressing tagged SUMO1, we could not detect SUMO1-conjugation of seven previously pro

20 el of CD22 monoclonal antibodies and Western blot analyses, we could not detect surface or intracellular expression of CD

21 ER- associated LCV formation was unique to CLas, as we could not detect these bodies in B. trigonica infected wit

22 Limitations of this study are that we could not determine whether TB disease was due to reactiva

23 t we did not have individual-level data on sun exposure, so we could not directly control for this in the primary analysi

24 leaved products are required for the PDI phenotype although we could not distinguish loss of function from loss of secret

25 Although, we could not establish a role of VDAC channels in the transpo

26 Because we could not exclude a small effect size due to a limited sam

27 In conclusion, we could not find any unique metabolite profile distinguishin

28 lagen (Col-I) promoter (COL-EGFP) had green stroma, whereas we could not find COL-EGFP(+) cells in tumors developing in t

29 Unexpectedly, we could not find this HoRE in any other yeast promoter.

30 of this study was that, owing to its observational nature, we could not fully exclude the effects of residual confoundin

31 After we offered telemedicine to both groups, we could not identify a difference between the groups in the

32 Because of the absence of treatment effect, we could not identify a gene signature predictive of clinical

33 Of note, we could not identify a T-stage threshold below which there w

34 In contrast to GDPDLd17, we could not identify any enzymatic activity for the GDPD-lik

35 In contrast, we could not identify major drivers of the taxonomic composit

36 Although we could not identify subassemblies of complex II, our result

37 s relies on one source of sugar industry documents and that we could not interview key actors.

38 Because we could not localize ionotropic GABA receptors on cone axon

39 -induced Akt signaling dynamics and cell cycle progression, we could not predict the fate of individual cells.

40 as high in many studies and event rates were variable, thus we could not present pooled results.

41 icroparticles, pH sensors, and 3D microfluidic devices that we could not produce using traditional 3D printing.

42 Although we could not provide a conclusive explanation for this excess

43 Strikingly, we could not reconstitute infection in permissive fibroblasts

44 Though we could not reject the hypothesis that culling up to three b

45 ed helical positioning, for the great majority of dendrites we could not reject the null hypothesis of spatial randomness

46 ater than the margin of non-inferiority of 1.08; therefore, we could not reject the null hypothesis that ibandronic acid

47 However, we could not rescue an SA11 reassortant with a human RV strai

48 Although we could not resolve the mechanism through which miR-148a-3p

49 There was no evidence of publication bias; however, we could not separate the contributions of obesity-related ca

50 In contrast, using almost identical targeting constructs, we could not truncate or C-terminally tag the SERA5 gene, or