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1  over a 0.20-0.50 hct range) for the hemaPEN devices, which we could not attribute to the analytical procedure.
2                                                       Thus, we could not conclusively detect sequence changes resulting f
3                        Also, using the inhibitor erlotinib, we could not confirm a role for the epidermal growth factor r
4                                                    Although we could not confirm that methylation directly inhibits editi
5 cohort of patients undergoing repeated radioiodine therapy, we could not demonstrate alterations in salivary parameters.
6             In live-cell time-lapse microscopy experiments, we could not detect any significant phagocytosis by purified
7                                                In contrast, we could not detect auxilins in abortive pits or at any time
8 s, etoposide robustly induced TOP2B covalent complexes, but we could not detect doxorubicin-induced TOP2-DNA complexes, a
9                      Overall, in healthy adult C57BL/6 mice we could not detect male/female differences in glymphatic inf
10 pite our large sample size and multiple datasets available, we could not detect robust associations between specific gene
11 ing a knock-in reporter mouse line expressing tagged SUMO1, we could not detect SUMO1-conjugation of seven previously pro
12         ER- associated LCV formation was unique to CLas, as we could not detect these bodies in B. trigonica infected wit
13                                               Surprisingly, we could not detect yuzunone by GC-MS in any of our samples.
14                                                In 269 (80%) we could not determine the cause of death.
15                                 We removed papers for which we could not determine the gender of either the first or last
16                          Limitations of this study are that we could not determine whether TB disease was due to reactiva
17 t we did not have individual-level data on sun exposure, so we could not directly control for this in the primary analysi
18 leaved products are required for the PDI phenotype although we could not distinguish loss of function from loss of secret
19  the duration of systemic corticosteroid exposure, although we could not entirely exclude harm on some secondary outcome
20                                                   Although, we could not establish a role of VDAC channels in the transpo
21                                                     Because we could not exclude a small effect size due to a limited sam
22 s to A(H7N2) virus, indicating possible infection; however, we could not exclude cross-reactive antibody responses to sea
23                              The main limitations were that we could not explore the consistency of our results using a m
24                                              In conclusion, we could not find any unique metabolite profile distinguishin
25 lagen (Col-I) promoter (COL-EGFP) had green stroma, whereas we could not find COL-EGFP(+) cells in tumors developing in t
26                                                       While we could not formally assess the association of the CR1 frame
27  of this study was that, owing to its observational nature, we could not fully exclude the effects of residual confoundin
28         If a STEPS dataset was not available for a LMIC (or we could not gain access to it), we conducted a systematic se
29                 Because of the absence of treatment effect, we could not identify a gene signature predictive of clinical
30             It also exposes a diversity of network effects: we could not identify a single sub-network that is perturbed
31                                    In contrast to GDPDLd17, we could not identify any enzymatic activity for the GDPD-lik
32                                                In contrast, we could not identify major drivers of the taxonomic composit
33                        The main study limitations were that we could not investigate certain characteristics associated w
34                                      Due to limited access, we could not measure wells in most major active oil and gas f
35 udy is constrained by gaps in the published data, such that we could not model the impact of antiviral therapy based on s
36                                 Although at this resolution we could not observe individual chemical groups, we could una
37 ants at 1.8-2.0- angstrom resolution revealed features that we could not predict by molecular dynamics simulations, inclu
38 -induced Akt signaling dynamics and cell cycle progression, we could not predict the fate of individual cells.
39 as high in many studies and event rates were variable, thus we could not present pooled results.
40 icroparticles, pH sensors, and 3D microfluidic devices that we could not produce using traditional 3D printing.
41                                                    Although we could not provide a conclusive explanation for this excess
42           Hydro-EVE was present in a subset of samples, but we could not quantify it.
43                                                    However, we could not rescue an SA11 reassortant with a human RV strai
44 male mosquitoes following oviposition, with the caveat that we could not rule out batch effects due to the unanticipated
45 ere largely due to unmeasured confounding factors, although we could not rule out small independent associations, particu
46                                                          As we could not rule out the elimination of past transposition i
47                  Some estimates were imprecise, which meant we could not rule out the presence of smaller clinically impo
48         There was no evidence of publication bias; however, we could not separate the contributions of obesity-related ca
49 selection, or are under mechanisms of sexual selection that we could not test (e.g. balancing selection).
50 c constraints and the slow developmental rate of icefishes, we could not test for long-term hybrid viability, fertility,