ライフサイエンスコーパス: we crossed

1 vestigate the role of Crim1 during eye development in mice, we crossed a Crim1(flox) mouse line with the Ap2alpha-cre mou

2 To understand this mechanism of resistance, we crossed a p110alpha conditional (p110alpha(flx/flx)) mouse

3 We crossed a Wnt1-Cre transgenic mouse with a double-tandem T

4 We crossed APPswePS1DeltaE9 (APP.PS) mice with Cd38(-) (/) (-

5 igate interactions between heterozygosity and recombination we crossed Arabidopsis lines carrying fluorescent crossover r

6 First, we crossed ataxin-2 knockout mice with TDP-43 (also known as

7 h SRC-1 and SRC-2 die at birth due to respiratory distress, we crossed double-heterozygous males and females.

8 We crossed Drp1+/- mice with APP transgenic mice (Tg2576 line

9 We crossed Drp1+/- mice with Tau transgenic mice (P301L line)

10 To test this hypothesis, we crossed DYT1 knock-in with p58(IPK)-null mice.

11 has a functional role in Alzheimer's disease pathogenesis, we crossed endophilin-B1(-/-) mice with APPswe/PSEN1dE9 mice.

12 To test this, we crossed Ets1(-/-) mice to two different transgenic models

13 We crossed Fah(-/-), Rag2(-/-), Il2r(-/-) mice with nonobese

14 re associated with the enamel defects and hypophosphatemia, we crossed Fam20C(fl/fl) mice with Wnt1-Cre and Osr2-Cre tran

15 To test this hypothesis, we crossed gamma-sarcoglycan-null mice, a model of limb-girdl

16 We crossed Gsk3a/GSK3alpha-knockout mice with low-density lip

17 We crossed Hdac3 conditional knockout mice with Mb1-Cre knock

18 We used a factorial experiment in mesocosms wherein we crossed high- and low-predation guppy phenotypes with popu

19 he role of fractalkine signaling in macrophage recruitment, we crossed huDTR mice to a mouse line that lacks expression o

20 Idh1 mutation as an oncogenic driver in the T-cell lineage, we crossed Idh1-KI mice with conditional Trp53 null mice, a w

21 We crossed Irf6 heterozygous ( Irf6(+/-)) mice with transgeni

22 plore the function of KAT8 during mouse oocyte development, we crossed Kat8(flox/flox) mice with Gdf9-Cre mice to specifi

23 To test this further, we crossed Kit(W-sh/W-sh) mice with signal transducer and act

24 To further examine the relationship between LRRK2 and tau, we crossed LRRK2 R1441G BAC transgenic mice (Mus musculus) wi

25 ute 2 (MDM2)-p53 pathway to APC loss-induced tumorigenesis, we crossed mice bearing MDM2(C305F) mutation, which disrupts

26 that cooperate with STAT5 activation to initiate leukemia, we crossed mice expressing a constitutively active form of ST

27 To address this, we crossed mice harboring floxed Glp1r alleles to mice expres

28 whether the upstream kinase JAK2 exerts similar functions, we crossed mice harbouring a hepatocyte-specific deletion of

29 To address this, we crossed mice humanized for CYP2D6 or CYP3A4 with mice carr

30 To disambiguate these the two mechanisms, we crossed mice in which channelrhodopsin is endogenously exp

31 We crossed mice overexpressing human retinol-binding protein

32 onmuscle myosin II-A (NM II-A) to early cardiac development we crossed Myh9 floxed mice and Nkx2.5 cre-recombinase mice.

33 To test this, we crossed NCoRDeltaID and Src-1(-/-) mice to create mice def

34 In our current study, we crossed our Lpd conditional knockout mice with a mouse lin

35 We crossed our transgenic SclCre;V617F mice with a conditiona

36 We crossed PLP-EGFP reporter mice into a Plp1-null background

37 To analyze the fate of Prom1+ DP cells, we crossed Prom1C-L mice with Rosa26-CAG flox/stop/flox tdTom

38 To address this question, we crossed SOD1 (wild-type SOD1(WT) and mutant SOD1(G93A)) or

39 We crossed Sort1(-/-) mice onto a humanized Apobec1(-/-); hAP

40 We crossed the APP/PS1 mouse model of cerebral amyloidosis wi

41 To examine ApoE role, we crossed the human ApoE-targeted replacement mice with muta

42 urther define the roles of EAF2 in prostate carcinogenesis, we crossed the Pten+/- and EAF2+/- mice in the C57/BL6 backgr

43 We crossed the self-infertile ATP synthase beta subunit knock

44 We crossed these CD79-GFP lines to a recombination activating

45 We crossed these drivers to conditional DREADD mouse lines to

46 We crossed these mice with LSL-KrasG12D mice, which express a

47 In the present study, we crossed Thra1 (PV/+) mice with mice expressing a mutant Nc

48 In this study, we crossed Trem2(+/+) (T2(+/+)) and Trem2(-/-) (T2(-/-)) mice

49 To globally quantify these effects, we crossed two divergent mouse strains and asked how genotype

50 We crossed Vgat- and Vglut2-ires-Cre mice with an Esr1(lox/lo