1 We determined the ability of plasma GPBB to discriminate betw
2 First,
we determined the ability of platelets to kill S. aureus dire
3 We determined the abundance of more than 7,900 proteins and s
4 We determined the accuracy of an extubation readiness test (R
5 Here
we determined the activity of the individual olefin isomers.
6 We determined the affinity of hnRNP A1 for all possible seque
7 We determined the association of cerebral perfusion (mL/100mL
8 Using FISH,
we determined the distribution of cccDNA under conditions mim
9 In addition,
we determined the distribution of T-2 toxin and DON in Spragu
10 We determined the effect of 9 days of isocaloric fructose res
11 Based on the correlational Fos results,
we determined the effect of bed nucleus of the stria terminal
12 We determined the effect of fibrin on the diffusion, intratum
13 and glycemic load (GL) value determinations remains unclear.
We determined the effect of meals that varied in macronutrien
14 We determined the effect of multi-antigenic construct express
15 We determined the effect of three enhanced strategies for ter
16 To evaluate SGE-516 in Scn1a (+/-) mice,
we determined the effect of treatment on hyperthermia-induced
17 We determined the effectiveness of common extraction conditio
18 We determined the effects of disruption Mir34a, Mir34b, and M
19 We determined the effects of the miR-29 family on glucose met
20 We determined the effects of the substitutions on peptide oli
21 We determined the function of Toll-like receptor (TLR) signal
22 We determined the impact of a modest increase in dietary zinc
23 sthma in a birth cohort using administrative databases, and
we determined the impact of adjusting for potential confounde
24 We determined the impact of HCA on the CD4(+) T lymphocyte ex
25 We determined the impact of residential traffic on mortality
26 le produces sustained MAPK/ERK activation in adult SCs, and
we determined the impact of such activation on nerve repair.
27 In this study,
we determined the impact of the R753Q TLR2 polymorphism on ma
28 ired Fe-S cluster biogenesis associates with human disease,
we determined the importance of FBXL5 for regulating IRP1 whe
29 We determined the influence of LAC on post-ischemic inflammat
30 We determined the kinetics of growth and turnover of nitric o
31 Finally,
we determined the levels of IgE specific for staple foods and
32 We determined the number of CFHR3 and CFHR1 gene copies by qu
33 We determined the number of OHCAs that occurred within 100 m
34 We determined the prevalence of early and late AMD in Europe
35 large health care organization's EHR between 2000 and 2013,
we determined the prevalence of food allergy and intolerance
36 Using paired recordings and two-photon imaging,
we determined the properties of the primary input to granule
37 To provide clarification,
we determined the proportion of access-related deaths in a re
38 We determined the proportion of patients with newly diagnosed
39 We determined the risk of an alcohol abuse diagnosis on incid
40 We determined the risk of clinically significant (stage 4 and
41 OS status is pivotal to inflammation and bacterial killing,
we determined the role of DJ-1 in bacterial sepsis.
42 Here
we determined the role of IFN-I-driven, ISGF3-independent sig
43 In this study,
we determined the role of IL-21R signaling in Mycobacterium t
44 In the present study,
we determined the role of KCC2-protein interactions in regula
45 Here,
we determined the role of mitochondrial antiviral-signaling p
46 We determined the spectrum of SNVs in a single human cell aft
47 We determined the structure of an anti-termination complex in
48 Using cryo-electron tomography and subtomogram averaging,
we determined the structure of the COPI coat assembled on mem
49 We determined the structures of the CL40 and CL59 complexes w
50 Here, using maize (Zea mays) as a model plant system,
we determined the timing of zygote development and generated