ライフサイエンスコーパス: we do not know

1 -speed relationship might be optimum for the latter purpose we do not know.

2 Here, we describe and discuss what we know and what we do not know about various PCD pathways and how the host in

3 Although hosts are discriminating, we do not know exactly what cues they use, and to answer this

4 In contrast, we do not know how brain-derived serotonin favors bone mass a

5 dorant concentration is a key feature of olfactory stimuli, we do not know how concentration is translated into perceived

6 Yet, we do not know how distinct endothelial and haematopoietic fa

7 However, we do not know how functional connectivity of the default-mod

8 alysis have identified the AS as a force-generating tissue, we do not know how individual cell behaviours are transformed

9 olecular components that mediate kinetochore binding [5-7], we do not know how kinetochores physically interact with poly

10 ssed predominantly by the left hemisphere of the brain, but we do not know how or why.

11 However, we do not know how quercetin and trans-RSV individually affec

12 However, we do not know how Rad1/Rad10 recognizes these structurally d

13 However, we do not know how reliably these measures predict the streng

14 ilitate cell shape change and buffer mechanical stress, but we do not know how reservoir dynamics are regulated.

15 However, we do not know how symbionts affect host fitness when the lat

16 However, we do not know how the asymmetric effects of foundation speci

17 In particular, we do not know how the temporal organization of cell division

18 However, when quantum effects are involved, we do not know how to compute channel capacities.

19 Cell fate decisions are fundamental for development, but we do not know how transcriptional networks reorganize during

20 Despite intense study of AMPs on model membranes, we do not know how well the mechanism of attack translates to

21 However, when walking at faster speeds, we do not know if and how these reflexes are changed.

22 However, we do not know if differences in the spatial environment diff

23 rter expressed in the liver, is one ribose transporter, but we do not know if others exist.

24 Currently, we do not know if the efficiency of transmission is very low

25 What we do not know is which populations, sectors, or countries wi

26 lowly proliferating cancer cells that resist treatment, but we do not know precisely how these cells arise.

27 owever, important unanswered questions remain--for example, we do not know the cause of AQP4-IgG-negative disease, how as

28 he tyrosine kinase c-Met contributes to cyst formation, but we do not know the downstream mediators.

29 loops has been shown to contribute to the above phenomenon, we do not know the exact the role of these individual loops.

30 Currently, we do not know the importance of global protein localization

31 However, we do not know the neural activity patterns in C. elegans tha

32 in patients undergoing assessment for epilepsy surgery, but we do not know their potential for providing helpful clinical

33 ter speech-in-noise perception compared with non-musicians, we do not know to what extent binaural processing contributes

34 ing of the forces dynamically maintaining poles is limited: we do not know where and how quickly they act or their streng

35 e bears the load of chromosome movement far from poles, but we do not know where and how-physically and molecularly-this

36 H during major extinctions in the geological past, and thus we do not know whether human impacts are pruning the tree of

37 Moreover, we do not know whether improvements in door-to-balloon times

38 e the rate of rebleeding compared to non-high-dose PPIs but we do not know whether intravenous non-high-dose PPIs reduce

39 However, we do not know whether maternal experience persistently alter

40 Additionally, we do not know whether previously observed correlations betwe

41 The study was limited as we do not know whether the additional patients presenting to

42 However, we do not know whether the distributions of symbionts across

43 Surprisingly, we do not know whether the firing of cortical neurons is affe

44 supports efficient viral transmission in mammals; however, we do not know whether the four mutations in the H5 HA identi

45 irths within the UN Millennium Development Goals framework, we do not know whether women are deciding against home birth

46 ut of which many are useless and only a few are useful, but we do not know which ones they are.

47 As we do not know which specific proteins, if any, regulate BRM,

48 However, we do not know why some patients develop more QT prolongation

49 In particular, we do not know why these infections respond poorly to antibio

50 It has been suggested that we do not know within an order of magnitude the number of all