1 Here
we establish a "two-hit" stress model in mice wherein stress
2 Thereby
we establish a connection between the normal state responses
3 Instead,
we establish a critical role for phosphatidylinositol 3-kinas
4 ting the light-sheet and sectioning the heart mechanically,
we establish a holistic strategy for 3-dimentional reconstruc
5 Thus,
we establish a new paradigm for mosquito surveillance that ta
6 Here
we establish a novel system for mechanistic analysis of the p
7 Here
we establish a physiological context for this regulation and
8 In this article,
we establish a proof-of-principle for methods to initiate and
9 To investigate such protein interactions,
we establish a ribosome affinity purification method that une
10 og 1 (AKT1), and the protease cathepsin H (CTSH), for which
we establish a role in filaggrin expression and processing.
11 Using this mouse line,
we establish a specific requirement for the P1-RUNX1C isoform
12 Here,
we establish a versatile blastocyst complementation platform
13 straints from ssNMR and cryo-electron microscopy (cryo-EM),
we establish an atomic resolution model of the RSV CA tubular
14 ges of computational studies in other life science domains,
we establish an authentic data-driven modelling framework to
15 Here
we establish design rules for implementing this sequence-cont
16 We establish detailed maps for subtelomere gap regions in the
17 Through de novo transcriptome reconstruction,
we establish dynamic expression profiles of lncRNAs at differ
18 of K11/K48-linked chains in cell-cycle and quality control,
we establish heterotypic ubiquitin conjugates as important ca
19 Here
we establish PPM1A as a checkpoint target used by Mtb to supp
20 We establish small error rates in the identification of evoke
21 Here,
we establish that although ICP34.5 binds TBK1 under certain c
22 rizone (CPZ) model of demyelination and mice of either sex,
we establish that Cav1.2 deletion in OPCs leads to less effic
23 MENT By comparing brain tumor patients to healthy children,
we establish that changes in the microstructure of the optic
24 Here
we establish that DOCK5 has a non-redundant role in regulatin
25 Using real-time single-molecule analysis,
we establish that leading- and lagging-strand DNA polymerases
26 Using time-resolved spectroscopy,
we establish that luminescence via triplets occurs within 350
27 We establish that monomeric BslA is necessary and sufficient
28 In this study,
we establish that RAGE-PR3 interaction mediates homing of pro
29 We establish that SNX5 and SNX6 associate with the CI-MPR thr
30 We establish that substantial changes in vegetation and fire
31 lotting, immunolocalization and in vitro functional assays,
we establish that the "neuron-specific" K(+)Cl(-) co-transpor
32 Here,
we establish that the HSV-1 Us3 protein kinase subverts the n
33 Here,
we establish that the ISG15 gene product itself directly regu
34 We establish that the presence of either D2L or D2S can suppo
35 We establish that these microbial consortia act as cartels, w
36 Moreover,
we establish that this decision is made deterministically, do
37 Here
we establish that Toxoplasma gondii aspartyl protease 3 (ASP3
38 Validating its utility as a discovery pipeline,
we establish that two of these proteins promote ubiquitin-lin
39 stic puff model and a single-channel data-based IP3R model,
we establish the dependencies of lambda and xi on two importa
40 Here,
we establish the Drosophila system to study the m(6)A pathway
41 Here,
we establish the dynamics of the molecular cations in FAPbI3
42 First,
we establish the existence of exclusively surface-dominated t
43 Here
we establish the feasibility of statistically proving the con
44 Here,
we establish the landscape of human cell-specific microRNA ex
45 Here,
we establish the mechanism of synergistic cooperation between
46 Further,
we establish the sensitivity of our pipeline by detecting sub
47 We establish the state diagram and find that short active rod
48 We establish these effects are mediated by both postsynaptic
49 Here
we establish,
through fine mapping, genome sequencing, geneti
50 We establish ZFP91 as a bona fide IMiD-dependent CRL4(CRBN) s