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3 To confirm the importance of pS269 in AQP2 re-distribution, we expressed an AQP2 S269A mutant in LLC-PK1 cells, and found
5 Taking advantage of FLiCRE's modular design, we expressed an optogenetic channel selectively in this cell
8 ling is also involved in the behavioral effects of ethanol, we expressed astrocyte-specific excitatory DREADDs in the pre
9 ation Efficiency for primary emission-to-particle pathways, we expressed changes in particle-related mortality in terms o
14 onstrate the utility of this new viral expression strategy, we expressed GCaMP6f in Lm128C cells in the superficial layer
18 aluate the effect of ALS-associated mutant Sod1 expression, we expressed human Sod1 WT and mutants in human cells knocked
20 ing of the function of this fusion protein in cancer cells, we expressed in E. coli, purified and characterized human NPM
25 of visual arrestin-1 oligomerization in rod photoreceptors, we expressed mutant arrestin-1 with severely impaired self-as
27 To test the role of arrestin-1 self-association, we expressed oligomerization-deficient mutant in arrestin-1 k
31 also used a transcellular Tat transactivation assay, where we expressed Tat proteins of HIV-1 clade B (Tat-B) or C (Tat-
35 In a rat model of C2 hemisection SCI, we expressed the axon guidance molecule, brain-derived neurot
38 n order to identify host lncRNAs affected by HPV infection, we expressed the high-risk HPV-16 E6 oncoprotein in primary h
39 e rejection and further suppress adaptive immune responses, we expressed the immunomodulatory factors PD-L1, HLA-G, and t
41 To study the proBDNF structure and function, here we expressed the protein and its nonglycosylated N127Q mutant
42 In trying to elucidate whether THIK-1 is regulated via PKA, we expressed THIK-1 channels in a mammalian cell line (CHO ce
44 To demonstrate the efficacy of CRAGE, we expressed three known and six previously identified but ex