1 o determine how this signaling network is altered in maize,
we first examined effects of a knockout mutation in an essent
2 insight into this important biomarker of radiosensitivity,
we first examined genomic patterns reflective of defects in D
3 Here,
we first examined how 11 years of warming during different se
4 To fill the void,
we first examined hybridization isotherms generated on differ
5 To evaluate the role of chemotaxis in our model,
we first examined in vivo chemotaxis in the absence of wound
6 We first examined isolated compounds (monomers) and evaluated
7 We first examined its role in established melanoma cells.
8 We first examined long-term trends in childhood pneumonia mor
9 To evaluate whether PKCdelta regulates Rab7,
we first examined lysosomal morphology in cells with reduced
10 We first examined mitochondrial respiration before investigat
11 To validate our technique,
we first examined one modulating aromatic ligand, Congo Red,
12 We first examined p42 ERK (ERK2) activation in the NAcc after
13 In this study,
we first examined putative cargo complexes associated with sy
14 We first examined redox (oxidation/reduction) properties and
15 We first examined retinas from control, non-transplanted, age
16 In this active vaccination study,
we first examined safety and immunogenicity for a broad serie
17 To investigate the importance of SLURP2,
we first examined Slurp2 knockout mice in which exon 2-3 sequ
18 We first examined the ability of the model to predict the flo
19 We first examined the association of recipient HCV status wit
20 We first examined the association of recipient HCV status wit
21 We first examined the association of statin treatment with va
22 To explore these questions,
we first examined the cellular localization of endogenous myo
23 Here,
we first examined the distribution of aKIR genes and haplotyp
24 We first examined the effect of genetic deletion of MOR and D
25 We first examined the effect of the auxiliary subunits.
26 Here,
we first examined the effects of chronic EtOH on DLS neuronal
27 In this study,
we first examined the effects of MSH3 deficiency on cytotoxic
28 We first examined the familial coaggregation of clinically di
29 We first examined the fundamental steps by which protein comp
30 We first examined the integrity of reproductive isolation whi
31 investigate early events associated with antigen exposure,
we first examined the interaction of PF4/heparin complexes wi
32 We first examined the levels of Grx1 in postmortem midbrain s
33 We first examined the location of Thy-1(+) CAFs within human
34 To explore this question,
we first examined the morphological effects of expressing sho
35 Using a mouse infection model,
we first examined the mouse colonization ability of an H. pyl
36 Using a prospectively recruited cohort,
we first examined the relation between these geometric measur
37 We first examined the relationship between fasting plasma TMA
38 se IS is an agonist of the aryl hydrocarbon receptor (AHR),
we first examined the relationship between IS levels and AHR-
39 We first examined the relationship of KF11 responses in B cla
40 To test this hypothesis,
we first examined the response of peripheral blood mononuclea
41 We first examined the role of cognitive strategies by having
42 We first examined the role of residue 283 in macrophage tropi
43 We first examined the roles of Mg(2+) by kinetic analysis of
44 We first examined the temporal relationship between GC activi
45 We first examined this switching process in an auditory event
46 We first examined two nature reserves with contrasting free-r
47 We first examined whether mice and humans experience similar
48 Here
we first examined whether such deletions could occur during r
49 We first examined whether the acute-phase protein, alpha-2 ma
50 We first examined which KCNQ subunits are targeted by AKAP79