ライフサイエンスコーパス: we first tested

1 We first tested 808 patients who were homozygous for the Delt

2 We first tested a lineage rank approach, but this was unsucce

3 We first tested cytotoxicity of cesium-137 irradiation plus v

4 tion factor IX (hFIX) expression cassette in an adenovirus, we first tested different hFIX expression sequences with or w

5 We first tested each gene for increased or decreased burden o

6 begin to understand the evolutionary forces that shape gbM, we first tested for correlations in methylation levels across

7 We first tested GluA1(-/-) mice on a standard food-carrying h

8 We first tested how RAGE impacts tumor cell-intrinsic mechani

9 We first tested myocardium from 11 subjects with ARVC; of the

10 We first tested our algorithm on simulated data to elucidate

11 We first tested our approach in peduncular hallucinosis, a sy

12 We first tested our method on Homo sapiens genome; using a ve

13 We first tested syngeneic transplantation by implanting islet

14 We first tested SYSMOLE with synthetic data and evaluated its

15 We first tested the association between a prolonged PR interv

16 We first tested the canonical aminoacylation role of LdTyrRS.

17 beta (IL-1beta) plays a key role in AML cell proliferation, we first tested the effect of PAO on OCIM2 and OCI/AML3 AML c

18 a(IL-1beta) plays a key role in proliferation of AML cells, we first tested the effect of resveratrol on the AML cell lin

19 We first tested the effects of miR inhibitors (antagomirs), w

20 In order to understand the role of AHLs in vivo, we first tested the effects that deletions of the synthase ge

21 of this Ca(2+) release could play a role in Wnt signaling, we first tested the hypothesis that Ca(2+)/calmodulin-depende

22 We first tested the hypothesis that motion signals are integr

23 We first tested the hypothesis that PM blocks Amadori-to-CML

24 To gain insight into the source of these mutations, we first tested the hypothesis that the mutagenic effect of 5

25 isiae mRNA-binding protein Npl3 (ScNpl3) as a model system, we first tested the importance of the RG domain for protein f

26 Using transcranial magnetic stimulation (TMS), we first tested the involvement of the right STG in a task co

27 th the human leukocyte antigen (HLA) HLA-DQB1*06:02 allele, we first tested the pathogenic contribution of CD4 Th1 cells.

28 We first tested the performance of this approach using one-di

29 study the similarity of JDV and HIV tat in transactivation, we first tested the responses of a series of HIV LTR mutants

30 We first tested the role of cysteine residues in the formatio

31 n between LGN and V1 was essential for cortical plasticity, we first tested the role of layer 6 corticothalamic (CT) V1 n

32 To elucidate the protein network and their functions, we first tested their protein-protein interaction profiles.

33 We first tested these LPA analogues on insect Sf9 cells induc

34 To examine this system, we first tested thiram, a DTC pesticide known to display neur

35 Here we first tested this hypothesis with preclinical experiments

36 We first tested this method on heterozygous flies carrying ei

37 We first tested this method with sections of adult mouse inne

38 We first tested whether an acute decline in SRIF, independent

39 We first tested whether effects of rosette vernalization pers

40 We first tested whether hosts and parasites constrained each

41 Using adult wild-type (WT) and GPR54 knock-out (KO) mice, we first tested whether kisspeptin-GPR54 signaling is necessa

42 from 75,725 participants in two general-population studies, we first tested whether low levels of nonfasting triglyceride

43 We first tested whether rats exposed to SD may exhibit brain-

44 Here, we first tested whether similar color-selective or disparity-

45 We first tested whether the deletion mutants could replace th

46 We first tested whether the LDLR drives apoB degradation in a

47 We first tested whether the senescence-related p21(WAF1/Cip1)

48 We first tested whether there are correlated fluctuations in