1 We first tested 808 patients who were homozygous for the Delt
2 We first tested a lineage rank approach, but this was unsucce
3 We first tested cytotoxicity of cesium-137 irradiation plus v
4 tion factor IX (hFIX) expression cassette in an adenovirus,
we first tested different hFIX expression sequences with or w
5 We first tested each gene for increased or decreased burden o
6 begin to understand the evolutionary forces that shape gbM,
we first tested for correlations in methylation levels across
7 We first tested GluA1(-/-) mice on a standard food-carrying h
8 We first tested how RAGE impacts tumor cell-intrinsic mechani
9 We first tested innate responses in lung.
10 We first tested myocardium from 11 subjects with ARVC; of the
11 We first tested our algorithm on simulated data to elucidate
12 We first tested our approach in peduncular hallucinosis, a sy
13 We first tested our method on Homo sapiens genome; using a ve
14 We first tested six different energy ramps and analyzed the f
15 We first tested syngeneic transplantation by implanting islet
16 We first tested SYSMOLE with synthetic data and evaluated its
17 We first tested the association between a prolonged PR interv
18 We first tested the canonical aminoacylation role of LdTyrRS.
19 We first tested the capacity of the recombinant HAstV capsid
20 a(IL-1beta) plays a key role in proliferation of AML cells,
we first tested the effect of resveratrol on the AML cell lin
21 We first tested the effects of miR inhibitors (antagomirs), w
22 We first tested the hypothesis that genetic variation in inbr
23 We first tested the hypothesis that motion signals are integr
24 We first tested the hypothesis that PM blocks Amadori-to-CML
25 To gain insight into the source of these mutations,
we first tested the hypothesis that the mutagenic effect of 5
26 isiae mRNA-binding protein Npl3 (ScNpl3) as a model system,
we first tested the importance of the RG domain for protein f
27 Using transcranial magnetic stimulation (TMS),
we first tested the involvement of the right STG in a task co
28 th the human leukocyte antigen (HLA) HLA-DQB1*06:02 allele,
we first tested the pathogenic contribution of CD4 Th1 cells.
29 We first tested the performance of this approach using one-di
30 n between LGN and V1 was essential for cortical plasticity,
we first tested the role of layer 6 corticothalamic (CT) V1 n
31 We first tested the sensitivity to d-Amphetamine's stimulator
32 We first tested the system ability to detect changes in these
33 To elucidate the protein network and their functions,
we first tested their protein-protein interaction profiles.
34 We first tested these LPA analogues on insect Sf9 cells induc
35 To examine this system,
we first tested thiram, a DTC pesticide known to display neur
36 Here
we first tested this hypothesis with preclinical experiments
37 We first tested this method on heterozygous flies carrying ei
38 We first tested this method with sections of adult mouse inne
39 We first tested this on the two most prevalent FLG variants.
40 To accomplish this,
we first tested three inducible mural cell-specific mouse lin
41 We first tested whether effects of rosette vernalization pers
42 We first tested whether hosts and parasites constrained each
43 Using adult wild-type (WT) and GPR54 knock-out (KO) mice,
we first tested whether kisspeptin-GPR54 signaling is necessa
44 from 75,725 participants in two general-population studies,
we first tested whether low levels of nonfasting triglyceride
45 We first tested whether rats exposed to SD may exhibit brain-
46 Here,
we first tested whether similar color-selective or disparity-
47 We first tested whether the deletion mutants could replace th
48 We first tested whether the LDLR drives apoB degradation in a
49 We first tested whether there are correlated fluctuations in
50 We first tested xCas9-3.7, the most effective xCas9 variant i