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1 We have shown that amplification of sex-determining region Y-

2 We have shown that C. albicans co-opts amino acid catabolism

3 We have shown that CTLA-4(+)PD-1(-) memory CD4(+) T cells are

4 We have shown that fusion-inhibitory peptides delivered intra

5 We have shown that nanoparticles (NPs) can be used as ligand-

6 We have shown that PAR2 activation in the airways is involved

7 We have shown that peach PpeS6PDH gene is down-regulated in f

8 We have shown that precipitation, degradation and loss of bio

9 We have shown that PS1 regulates Abeta levels by a unique bif

10 We have shown that systemic beta-adrenoceptor blockade with p

11 We have shown that the amino-acid order is meaningful in mimo

12 We have shown that the intensity of the carotenoid Raman sign

13 We have shown that the NAD(+) precursor, nicotinamide mononuc

14 We have shown that the plasmonic response of the nanoarray ca

15 We have shown that the proliferation of late gestation (embry

16 We have shown that the reactivity of the donor-acceptor cyclo

17 We have shown that the transcription factor Atonal homologue

18 We have shown that treatment with the KATP channel agonist pi

19 We have shown that we can improve the stability of SAT2 virus

20 We have shown that with the appropriate weighting scheme, we

21 In conclusion, we have shown that binding of UL20 to GODZ in the Golgi appar

22 Finally, we have shown that sphingosine 1-phosphate, an endogenous mic

23 Here we have shown that 3 nonsynonymous ITGAM variants that produc

24 Here we have shown that a new CXCR4 receptor antagonist IgG1 antib

25 Here we have shown that CD47-signal regulatory protein alpha (SIRP

26 Here we have shown that VHL-/- RCC cells rely on RC-derived aspart

27 Here, we have shown that at steady state, white adipose tissue cont

28 Here, we have shown that blocking the activation of PERK using GSK2

29 Here, we have shown that cancer cells respond to clinically relevan

30 Here, we have shown that chemogenetic stimulation of GCG-producing

31 Here, we have shown that intrinsic and immune cell gene signatures

32 Here, we have shown that MLL-ENL and MLL-AF10 constitutively activa

33 Here, we have shown that transplantation of adipose-derived stem ce

34 Here, we have shown that URMC-099 facilitates ARV sequestration and

35 In addition, with this method, we have shown that the high-resolution raw data can be fully

36 y quantitative fluorescence lattice light-sheet microscopy, we have shown that ESCRT-III subunits polymerize rapidly on y

37 Moreover, we have shown that zinc coordination is also important for th

38 ain-of-function alleles to dial E2F transcriptional output, we have shown that copy number gains in E2f1 or E2f3b resulte

39 Previously we have shown that CRM1 binds NESs in both polypeptide orient

40 Previously, we have shown that intracellular substrates are recruited to

41 Previously, we have shown that mature cytoplasmic tRNAs are cleaved durin

42 Recently we have shown that BCD has an anti-inflammatory effect on CC

43 Recently, we have shown that coexpression of hMet and mutant-beta-caten

44 Separately, we have shown that a direct intratumoral injection of immunoc

45 In previous studies, we have shown that stimulation of cultured neurons with surro

46 In a previous study we have shown that the amplification of the 11q22.1-q22.2 reg

47 In this previous study, we have shown that while Seqkernel performs better than stand

48 However, using CRISPR/Cas9 technology, we have shown that ACLY is not essential for HCMV growth and

49 Furthermore, for the first time, we have shown that SR Ca content is increased in old atrial m

50 In our own prior work, we have shown that six codons in VH4-containing genes in B ce

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