1 We identified 1 392 289, 530 771, and 1 125 231 hospital
2 We identified 100% of the germline NF1 mutations and fou
3 We identified 1044 patients, among whom 460 (44.1%) rece
4 Additionally,
we identified 12 potential new species among the 76 isol
5 We identified 13 randomised controlled trials done in 73
6 We identified 13 TAS2R16 residues that contribute to lig
7 Through RNA-Seq analyses,
we identified 137 genes that are missing in chicken, inc
8 In the PA mouse liver
we identified 14 significantly dysregulated miRNAs.
9 We identified 14 studies on the topic (distinct by regio
10 Additionally,
we identify 15 tales that are more likely to be predomin
11 Using mass spectrometry approaches,
we identified 16 VTPs of the CyHV-3 FL strain.
12 Recipients data from June 2013 to June 2015,
we identified 1768 DD livers exported to regional candid
13 We identified 19 rotor domains in 10 patients (1.8+/-1.1
14 We identify 19 new TGCT risk loci, roughly doubling the
15 However,
we identified 2 or more missense variants predicted to b
16 Using hierarchical cluster analysis,
we identify 21 epidemic clusters, of which 12 were cross
17 In the combined meta-analysis,
we identified 22 loci associated at genome-wide signific
18 Using a circRNA microarray,
we identified 226 differentially expressed circRNAs, of
19 We identified 235 children with CMA; 66 of these childre
20 We identified 238 (XP-EHH) and 213 (XP-CLR) positively s
21 We identified 25 new SNP-CAD associations (P < 5 x 10(-8
22 We identified 25 new susceptibility loci, 3 of which con
23 We identified 26 ancient and more recent polyploidy even
24 We identified 265 specimens to species or genus using DN
25 We identified 3 mechanisms underlying local progression
26 We identified 3 such proteins and show that they interac
27 We identified 31 cases of PTLD during the study period.
28 We identified 348,462 patients with one of the eligible
29 We identified 35 articles, including 7112 respondents.
30 We identified 4 host gene expression profiles, among whi
31 We identified 402 patients from 233 articles published i
32 We identified 411 (198 annotated) and 27 (15 annotated)
33 During follow-up,
we identified 5,462 incident cases of rosacea.
34 We identified 50 genes essential for the survival of A.
35 Results
We identified 51 trials assessing 14 drugs across 15 con
36 In ocular tissues of healthy house finches,
we identified 526 total bacterial operational taxonomic
37 For 2010-2016,
we identified 53 national care continua with viral suppr
38 We identified 57 articles from 27 countries.
39 We identified 57 longitudinal studies exploring the asso
40 mbinant inbred Arabidopsis thaliana genomes,
we identified 6502 segregating structural variants.
41 We identified 764 genes with at least a 2-fold differenc
42 We identified 79 associations influencing 13 primary and
43 We identified 89,790 incident PD cases and 118,095 compa
44 METHODS AND
We identified 905 patients that were married at the time
45 We identified 968 patients with brain metastases at the
46 Using these switching rules,
we identified a 3-month and a 6-month cohort of "treatme
47 We identified a causal mutation in 1 of 14 genes in 50%
48 We identified a centrin-binding site within H. sapiens P
49 urther Palestinian-Jordanian SPG23 pedigree,
we identified a complex homozygous 4-kb deletion/20-bp i
50 In summary,
we identified a conopeptide that targets the nociceptor-
51 f genetics, proteomics and RNA biochemistry,
we identified a core set of mRNA m(6) A writer proteins
52 ognize known major antigenic sites in MeV-H,
we identified a D4 genotype variant that escapes neutral
53 We identified a feedback loop within the NANCI (Nkx2.1-a
54 Through this analysis,
we identified a ferrireductase: six-transmembrane epithe
55 By whole-exome sequencing,
we identified a heterozygous truncating mutation (c.279d
56 e antibodies spanning the dysferlin protein,
we identified a highly reproducible jigsaw map of dysfer
57 First,
we identified a light-sensing B12-binding transcriptiona
58 We identified a new set of genes encoding subunits of th
59 eover, by using mutated promoter constructs,
we identified a NF-kappaB site as critical in this activ
60 We identified a novel 1 bp deletion in YAP1 in a boy wit
61 zation, expression and phylogenetic analyses
we identified a novel class of Arabidopsis thaliana poll
62 In this study,
we identified a novel direct interaction between APP and
63 TLR3 and TLR4 ligands as previous reported,
we identified a novel group of TRIM genes (TRIM14, 15, 3
64 By using whole-exome sequencing,
we identified a novel missense mutation in the binding d
65 In summary,
we identified a novel pathway for regulation of beta-cat
66 As a validation of this approach,
we identified a novel Sertoli cell enhancer upstream of
67 We identified a novel splice mutation in IKBKG (c.518+2T
68 Furthermore,
we identified a novel therapeutic approach in crescentic
69 Furthermore,
we identified a nuclear export signal (NES) at the N ter
70 ecifically, based on the relative abundance,
we identified a panel of 11 proteins to distinguish CRC
71 From this,
we identified a panel of 6 genes, ALDH1A1, HSP90AB1, KIT
72 Moreover,
we identified a pathogenic CD8(+) T cell MPO epitope (MP
73 Previously,
we identified a potent neutralizing antitoxin against Bo
74 Here
we identified a range of small organic molecules, drugs,
75 ry of 46 656 heparan sulfate hexasaccharides
we identified a rare sequence consisting of consecutive
76 First,
we identified a self-feeding mechanism by which CCL1 pro
77 Here,
we identified a series of peptides that interact specifi
78 Moreover,
we identified a significant global decrease in effective
79 We identified a small conopeptide of only four amino aci
80 We identified a small molecule, AC1903, that specificall
81 Overall,
we identified a small number of immunodominant regions,
82 In this study,
we identified a special case of cross-order recombinatio
83 Instead,
we identified a specific set of markers associated with
84 Here
we identified a two-amino-acid substitution in RORgammat
85 Here
we identified a Y/F-x-x-Y/L/F-x-Y/F motif, evolutionaril
86 Here
we identify a brain-region-specific neural progenitor-ba
87 Here
we identify a complex, nutrient-rich organic coating on
88 We identify a conserved region in the Ulp2 C terminus th
89 Here
we identify a critical role for ETAA1 in this process by
90 We identify a distinct C-terminal autoinhibitory four-re
91 We identify a functional EptA ortholog responsible for t
92 Here
we identify a lysosomal switch that enhances germline pr
93 Here,
we identify a new pathway that acts through NIMA-related
94 pitation with sequencing and RNA sequencing,
we identify a novel B-lymphoid program for transcription
95 Here,
we identify a novel boundary subdividing the mdFP into t
96 We identify a novel Myst2-associated protein, the tumour
97 We identify a novel susceptibility signal in the immunog
98 Here, using genome-resolved metagenomics,
we identify a number of CRISPR-Cas systems, including th
99 Here,
we identify a presynaptic effector molecule of the Wingl
100 By contrast,
we identify a pull-push inhibitory circuit in frontal co
101 Cumulatively,
we identify a signaling cascade that provokes structural
102 Unexpectedly,
we identify a subset of genes that adopted increased or
103 cal translation in the cortex of mice, where
we identify a subset of mRNAs that are translated in den
104 using gene expression and mutation profiles,
we identify a unique subpopulation based on addiction to
105 We identified alterations in signaling pathways and prot
106 model and human biopsies of prostate cancer,
we identify alterations in tumours affecting the product
107 d healthy controls (both males and females),
we identified an abnormally widespread hub formation in
108 nalysis including >300,000 European samples,
we identified an additional nine novel loci.
109 We identified an atherosclerosis-associated single-nucle
110 To prevent the IPDs caused by ST2,
we identified an effective ST2 neoglycoconjugate vaccine
111 We identified an FXR-responsive element on the Tgr5 gene
112 By cDNA library screening,
we identified an immune cell-specific, co-stimulatory re
113 Mechanistically,
we identified an interaction between moesin and TGF-beta
114 We identified an N-TIMP2 mutant, with five mutations in
115 Here,
we identified an orphan protein (Plu2236) from Photorhab
116 Here
we identify an additional motif that drives VE-cadherin
117 Finally,
we identify an association between high MCAM levels in p
118 In this study,
we identified and characterized IgE-binding proteins fro
119 We identified and cloned four AQPs from C. lectularius,
120 We identified and confirmed the highly potent antiandrog
121 Here,
we identified and deleted a polyketide synthase (PKS) ge
122 ogy called CANE developed in our laboratory,
we identified and selectively labeled noxious-stimulus-a
123 Last,
we identified and studied three novel patient XIAP mutat
124 Finally,
we identify and analyze the conserved syntenic blocks am
125 Here
we identify and characterise an epigenetic predictor of
126 Finally,
we identify and compare appropriate tunable-by-doping ma
127 Here,
we identify and determine that the previously uncharacte
128 n to protect against coronary heart disease;
we identified APOC3 homozygous pLoF carriers in our coho
129 We identified Arabidopsis pex6 and pex26 mutants by scre
130 These data suggest that the dynamic CGs
we identify are not specific to the Col-Cvi recombinant
131 effector spleen tyrosine kinase (SYK), which
we identified as an HSP90 client protein.
132 We identified associations between the FEV1/FVC ratio an
133 Using genome-wide association analysis,
we identify associations between several candidate genes
134 Here
we identify AUTEN-99 (autophagy enhancer-99), which acti
135 We identified autophagy as a pivotal cell response deter
136 We identified bacteria from the regurgitant of field-col
137 We identified basement membrane (BM) and collagen IV in
138 We identified both inflammatory monocytes and tissue-res
139 We identified BRPF1 deletions or point mutations in six
140 with strain maps of the developing skeleton,
we identify canonical Wnt signalling as a candidate for
141 Thus,
we identify CATH functional families that are currently
142 Importantly,
we identified chaperonin containing TCP1 subunit 6A (CCT
143 noparticles to eight tissues simultaneously,
we identified chemical properties promoting delivery to
144 We identify clusters corresponding to known tetraloop fo
145 We identified compounds that match the pharmacophore of
146 as chromatography mass spectrometry (GC-MS),
we identified compounds typically associated with plant
147 Furthermore,
we identified conserved miRNA-targets regulations in the
148 Here,
we identified core FtsH target proteins in S. aureus.
149 Here,
we identify dense innervation in the microenvironment of
150 We identify discriminatory features, which may be useful
151 Within this interval,
we identified disruptive mutations in two genes.
152 Furthermore,
we identified distinct epigenetic methylation patterns t
153 rewards and punishments of different nature,
we identify distributed neural representation of value,
154 Using Caenorhabditis elegans mutants,
we identify DNA repair factors that protect against the
155 o-hybrid protein-protein interaction method,
we identified eight novel interaction partners of EGFR,
156 In this study,
we identified elevated levels of matrix metalloproteinas
157 To elucidate Grh functions
we identified embryonic Grh targets by ChIP-seq and gene
158 ding motifs in a machine learning framework,
we identify EOR-1 as a unique transcription factor that
159 We identify features associated with chiral edge plasmon
160 We identified few horizontally transferred genes, but so
161 We identify filamentation self-compression scaling strat
162 On the basis of these dimensions,
we identified five distinct subtypes of perinatal depres
163 We identified five loci associated with emphysema distri
164 We identify five factors, including the HIV co-receptors
165 By pharmacologic and genetic studies,
we identify FKBP12 as a novel hepcidin regulator.
166 We identified Flunarizine - a well-known anti-migraine c
167 ng locally, nationally, and internationally,
we identified four communities that each shared a type o
168 SYNTHESIS:
We identified four issues highlighting the key areas of
169 Here,
we identified four unique mbf1 genes in the lichenized f
170 We identified gene expression signatures and clinical da
171 Here
we identify gene expression outliers, or individuals sho
172 it neuroblastoma metastasis in vivo Overall,
we identify gene expression signatures and candidate the
173 nterneurons to those of more mature neurons,
we identified genes important for human interneuron diff
174 e change within a species geographic range),
we identified global hotspots of species at risk from cl
175 We identified heavy smokers that were resistant (n = 65)
176 We identified heterozygous nonsense mutations in four an
177 We identified Hhex as a direct target of RUNX1 and FLT3-
178 We identified IL-18 as a cytokine that cooperates with a
179 investigate the role of two gtr operons that
we identified in the S Typhi genome.
180 Previously,
we identified increased translocation of the fatty acid
181 We identified individual and shared defects in PLP1 mRNA
182 Here,
we identified inositol-requiring enzyme 1alpha (IRE1alph
183 Instead,
we identify Insensible (Insb), another neural nuclear No
184 function of these and other p190A segments,
we identified interacting proteins by tandem mass spectr
185 Here
we identified IRAP(+) endosomes as major cellular compar
186 We identified less common conservation strategies that h
187 DLK inhibition is only partially protective,
we identify leucine zipper kinase (LZK) as cooperating w
188 In this study,
we identified LGR4 as a master controller of Wnt/beta-ca
189 ise many of the elements heavier than iron.)
We identify line features in the spectra that are consis
190 on and repair by next-generation sequencing,
we identified locations of elongation complexes and tran
191 We identified low but escalating risk of severe M. chima
192 Furthermore,
we identify Maf as a downstream target of the CIC-ETV5 a
193 nsequences of aneuploidy on cell physiology,
we identified mechanisms that eliminate aneuploid cells.
194 Overall,
we identify miR-500a-5p as an oxidative stress response
195 We identify molecular pathways and segmentation phenotyp
196 Overall,
we identified more infections (n = 146) in the denosumab
197 In mouse,
we identified more than 800 LTRs from ORR1, MT, MT2, and
198 In this study
we identify MRAP2 as a partner of ghrelin-GHSR1a signali
199 Moreover,
we identify multiple neurite-targeted non-coding RNAs an
200 In conclusion,
we identified mutations in MAPKBP1 as a genetic cause of
201 In this study,
we identify new functional roles of RASSF4.
202 We identified nineteen differentially methylated gene re
203 with comprehensive untargeted metabolomics,
we identified novel alterations in purine metabolism, gl
204 We identified novel and previously reported BMI-related
205 context of a quantum description of solids,
we identify novel capabilities for polarization- and pha
206 the fields of genomics and development, and
we identify organizational similarities between networks
207 Using bioinformatic analysis,
we identified over a thousand of human L1 loci containin
208 Unexpectedly,
we identified PABPN1-dependent ALYREF binding near the 3
209 We identified pathogenic mutations in 36 of 204 (17.6%)
210 We identified PKC delta and varepsilon as required and s
211 Through forward genetic screens
we identified PKL, a gene required for developmental reg
212 In this study,
we identified pleckstrin homology domain and leucine-ric
213 We identified predictors of final macular status, and de
214 Utilizing high-throughput screens,
we identify prion-curing mutants and engineer "anti-prio
215 We identify profound rearrangement in cellular proteosta
216 Further,
we identify promoters able to drive strong expression in
217 Using a connectivity map,
we identified prostaglandin E1 (PGE1) as a small molecul
218 KEY MESSAGE:
We identify proteins that associate with the THO core co
219 nse to PD-1 blockade in a mesenchymal tumor,
we identified PTEN mutations and reduced expression of g
220 Using the North American power grid,
we identified,
quantified, and analyzed the set of netwo
221 In addition,
we identified Rab27 as an important regulator of the int
222 downstream targets and effectors of miR-32,
we identified RAC2 as a potential, and clinically releva
223 We identify RIPK1 and caspase-8 as linearly ubiquitinate
224 Here,
we identified seven unrelated individuals affected with
225 Using fMRI,
we identified several areas of parietal, occipitotempora
226 In particular,
we identified several arginine residues that interact wi
227 We identified several different aggregating sites from l
228 We identified several genes involved in disease resistan
229 tional modeling and the BiLC reporter assay,
we identified several novel small-molecule compounds tha
230 Here
we identify several acetylation sites of the influenza A
231 Here,
we identify several novel CO2-dependent changes in the N
232 We identify SIAH2, a RING finger E3 ubiquitin ligase ass
233 Using multiple logistic regression,
we identified significant associations between whether a
234 We identified significant changepoints in all sites and
235 We identified significant group differences in striatal
236 We identified single-point mutations in the Fc domain (e
237 Among others,
we identified sites catalyzed at faster rates with poten
238 -susceptible gene locus, PSS1 In this study,
we identified six candidate PSS1 genes by comparing sing
239 We identify six mutational signatures (E1-E6), and Signa
240 Here
we identify soluble ephrin-B2 (sEphrin-B2) as a new prof
241 Here
we identify soluble epoxide hydrolase (sEH) as a key enz
242 Furthermore,
we identified splice variants encoding distinct nuclear
243 chromatography coupled to mass spectrometry,
we identified statistically significant differences in t
244 We identified substantial differences among CA, AA, and
245 In this work,
we identified suppressor mutations in the M1 protein whi
246 We identified surface markers associated with each chrom
247 Mechanistically,
we identified TBK1 as a key regulator of mTORC1 activity
248 We identified that HBx K91 and K95 as the key HBx NEDDyl
249 We identified that miR-195 was packaged in the extracell
250 Additionally,
we identified that NF-kappaB and MAPK signaling pathways
251 Here
we identified that the lysine-specific demethylase KDM3A
252 Here,
we identify that infection of host cells with reovirus c
253 biophysical and crystallographic approaches,
we identify that PrimPol possesses two RPA-binding motif
254 Here
we identify that tumour necrosis factor-alpha (TNFalpha)
255 We identified the band gap Eg and phonon cut-off frequen
256 We identified the BSs and assessed the effects on transc
257 Here,
we identified the conserved NK-2 homeobox gene ceh-24 to
258 t interact with ASK1 in the context of NASH,
we identified the deubiquitinase tumor necrosis factor a
259 From these empirical studies,
we identified the features of interaction patterns assoc
260 We identified the following variables as unfavourable pr
261 apillae deposition on the cell wall surface,
we identified the GLASSY HAIR 1 (GLH1) gene, which is ne
262 ntegrated analysis of the HIF-alpha network,
we identified the major protein kinase C substrate MARCK
263 We identified the minimal threshold that the participant
264 urthermore, using a yeast two-hybrid screen,
we identified the motor protein Kif15 as a potential int
265 First,
we identified the optimal NP formulation through compreh
266 olate host receptors for bacterial cdNs, and
we identified the oxidoreductase RECON.
267 In this study,
we identified the proliferating cell nuclear antigen (PC
268 Here,
we identified the spatial, temporal, and amplitude chara
269 Thus,
we identified the sublateral SIA neurons to control the
270 (ii)
We identified the variables that impact Env's glycosylat
271 Finally,
we identify the challenges that the field currently face
272 Here,
we identify the conserved RNA helicase Aquarius/EMB-4 as
273 Moreover,
we identify the ditiocarb-copper complex as the metaboli
274 novel ex vivo spinal cord preparation, here
we identify the functional organization of muscle and cu
275 and a custom CRISPR/Cas9 screening platform,
we identify the H3K9 methyltransferase SETDB1 as a novel
276 Using ex vivo and in vivo models,
we identify the Hedgehog (HH) paracrine system as a cand
277 Here
we identify the Iw1 gene from durum wheat and demonstrat
278 In this study,
we identify the metabolic sensor AMP-activated protein k
279 We identify the MuvB protein Lin37 as an essential facto
280 PA has a six-bladed beta-propeller fold, and
we identify the region that interacts with PfRh5.
281 Using an unbiased approach,
we identified thousands of genes whose expression was en
282 We identified three key components of the Notch pathway,
283 Here,
we identify TIAM1 as a critical antagonist of CRC progre
284 Using genome-wide association,
we identified Tnni3k as one gene that influences variati
285 Besides a predominant Neolithic background,
we identify traces of Post-Neolithic Levantine- and Cauc
286 rveillance in reprogramming gene expression,
we identified transcriptome-wide binding sites for RNA p
287 Here
we identify twenty-six DHC-derived features that provide
288 Here
we identified two classes of unmyelinated sensory neuron
289 Here,
we identified two conserved residues (R151, I155) in the
290 Through mutational analysis,
we identified two glutamine residues and a beta-hairpin
291 In a forward genetic approach,
we identified two Lotus japonicus mutants defective in A
292 We identify two bulge-depleted regions on the miRNA stem
293 We identify two distinct modes of kinetic stabilization
294 We identify two modes of transformation between stereoch
295 In addition,
we identified up to 161 genes that encode transcriptiona
296 We identified viral antigen in multiple organ tissues wh
297 Among the genes
we identified were some with obvious colonization-relate
298 We identified women diagnosed with unilateral stage I to
299 We identified ZEB1 binding sites within the LIF (stemnes
300 We identify zyxin as a regulator of stress fibre mechani