1 ing the ferric heme to undergo doming, which
we identify.
2 We identified 1,224 consecutive patients evaluated at In
3 We identified 10 antimicrobials that would have qualifie
4 In the cohort
we identified 101 incident cases of BCC.
5 We identified 11 cellular pathways required for HIV-1 re
6 We identified 11,842 cases and 59,210 controls.
7 We identified 110 kidney, 67 liver, 85 pancreas, 68 hear
8 We identified 123 patients for the CPI group and 147 pat
9 ribing records of the remaining 109 clinics,
we identified 13 657 eligible patients who were sent an
10 We identified 13 shared causal genes, 16 shared causal p
11 We identified 14 potent Nape-pld inhibitors in the Spect
12 We identify 1434 protein-coding genes and 39,806 noncodi
13 From 87,941,718 analyzed lives,
we identified 16,292,018 opioid prescriptions filled by
14 In total,
we identified 1699 cases of cardiomyopathy (mean age at
15 By comparing human and mouse data,
we identified 17 molecular cell types that have been gai
16 mechanisms associated with this phenomenon,
we identified 18 novel RALF-likes from multiple species
17 We identified 2 high-confidence risk genes, each contain
18 In this large and multiethnic study,
we identified 2 loci, TMEM40 and BAG3, associated with E
19 We identified 2056 women with CHD (2334 pregnancies) and
20 We identified 21 novel compounds that selectively inhibi
21 We identified 220 OR open reading frames, 20 of which ar
22 We identified 27 hits that were ranked using a using an
23 hrough sequence similarity network analyses,
we identified 29 Opp and 19 Dpp sequences that shared ve
24 From 832 high-LD variants,
we identify 39 candidate functional variants from 14 loc
25 for RV bronchiolitis (median age, 4 months),
we identified 4 distinct endotypes-mainly characterized
26 E and EAC tissues, along with clinical data,
we identified 4 subtypes that were associated with patie
27 We identified 45 candidate genes with ancient polymorphi
28 We identified 451 causal variants, which underlie geneti
29 We identified 454 differentially expressed genes in hiPS
30 We identify 48 genes (25 newly reported) showing signifi
31 h PH registry.Measurements and Main Results:
We identified 49 PH-NF1 cases, characterized by a female
32 We identified 49 single-base-substitution, 11 doublet-ba
33 A and perceived stress states in these data,
we identified 5 miRNA (including let-7f-5p and miR-181a-
34 ants, including 184,535 non-EUR individuals,
we identified 5,552 trait-variant associations at p < 5
35 We identified 526,808 people with atopic eczema and 2,56
36 We identified 611 loci associated with time-to-event phe
37 We identified 7 eligible studies published between 2008
38 ession models.Measurements and Main Results:
We identified 7 miRNAs significantly associated with FEV
39 We identified 771 incident cases of breast cancer during
40 We identified 816 differentially methylated CpG position
41 We identified 9,054 patients with HF with 5,839 hospital
42 We identified 98 783 hospitalized patients with candidem
43 sing third generation, long-read sequencing,
we identified a 120 kb insertion in the wingless allele.
44 wever, in several soluble-globular proteins,
we identified a class of nonconserved positions for whic
45 We identified a compound, BCH070, that inhibits asexual
46 We identified a cullin-RING ubiquitin ligase (CRL), cont
47 In addition,
we identified a differential requirement for LAMTOR4 and
48 Using whole genome sequencing,
we identified a heterozygous frameshift variant (p.Ser11
49 F plus smaller hyperreflective specks (HRS);
we identified a histologic candidate for HRS.
50 stress condition known as "tassel blasting."
We identified a mutant, necrotic upper tips1 (nut1), tha
51 We identified a new Fur-controlled regulator that is upr
52 Notably,
we identified a non-canonical cytochrome P450 that catal
53 Here,
we identified a novel mTORC1-interacting protein called
54 In this study,
we identified a novel neutralizing epitope in the head r
55 Using multidisciplinary approaches,
we identified a novel quinoline core ligand, BMPQ-1, whi
56 We identified a novel, biological difference between RV
57 We identified a number of conceptual, practical, and ana
58 Through additional analysis,
we identified a pattern of epitope substitutions in the
59 In this study,
we identified a pharmacologically inducible vasculoprote
60 Thus,
we identified a potential role for hnRNP H in basal and
61 In conclusion,
we identified a previously uncharacterized population of
62 We identified a Raman biomarker from glycogen to disting
63 Here,
we identified a role for host atypical chemokine recepto
64 nd temporal resolution live-cell microscopy,
we identified a role for mitochondria-lysosome contacts
65 We identified a second variant in FAF1 in the validation
66 Overall,
we identified a significant excess of loss-of-function D
67 Importantly,
we identified a single mutation that augments spontaneou
68 Using the voxel-wise approach,
we identified a small cluster in the left lateral fronta
69 However,
we identified a small set of seed-related orthologs with
70 gene expression to control for tumor burden,
we identified a subgroup of patients with an adverse TME
71 We identified a subpopulation of olfactory sensory neuro
72 ting proteomics and RNA-sequencing datasets,
we identified a subset of genes with apparent post-trans
73 We identified a subset of melanopsin-expressing intrinsi
74 Cas9 approaches to target CTNND1 in Xenopus,
we identified a subset of phenotypes that can be linked
75 Here,
we identified a type of Ca(2+) "mini-sensor" in YfkE, a
76 Using bioinformatics, mutation and NMR,
we identify a 7-residue sequence, named P1 (residues 36-
77 Here,
we identify a central role for the human DEDD deadenylas
78 We identify a circZNF827-nucleated transcription-repress
79 Here,
we identify a family of over 30 enzymes, which are homol
80 We identify a historical substitution that has pleiotrop
81 seq profiling of the intima of lesions, here
we identify a macrophage-specific lncRNA MAARS (Macropha
82 We identify a multiprotein complex between AURKA and the
83 Here,
we identify a neuronal circuit in the nematode Caenorhab
84 Here, using scanning tunnelling microscopy,
we identify a new topological kagome magnet, TbMn(6)Sn(6
85 Here,
we identify a nonredundant role for IFN-lambda in immune
86 Here
we identify a nontransitive evolutionary sequence in a 1
87 Here
we identify a novel and critical function of IKK2 and it
88 Finally,
we identify a novel group of CA1 neurons that robustly e
89 We identify a pH-sensitive electrostatic interaction bet
90 ntravital imaging of NSCs and their progeny,
we identify a population of Gli1-targeted NSCs showing l
91 Here,
we identify a pre-emptive pathway that reduces synthesis
92 Herein
we identify a previously unrecognized connection between
93 We identify a quartet of transcriptional regulators prom
94 Here,
we identify a single amino acid substitution (M159I) tha
95 We identify a single analytical factor, the specificatio
96 We identify a total of 868,476 genome-wide SNPs, of whic
97 In the current study,
we identify a unique granulocyte subset, with characteri
98 ranscription of bacterial CRISPR arrays, and
we identify a widespread antitermination mechanism that
99 We identified adults hospitalized with laboratory-confir
100 tudy nested within the US Renal Data System,
we identified all hip fracture events recorded among pat
101 ate this hypothesis and unravel its details,
we identified all phosphate-binding protein lineages in
102 We identified all specimens that were grossly examined a
103 We identified all subjects <21 years old who received fi
104 We identified alleles in 4 loci that were associated wit
105 Here
we identify alphaV-integrin (CD51) as an essential regul
106 In line with this,
we identified an altered perinatal and/or postnatal expr
107 Using MS,
we identified an ester bond formed between a thermolysin
108 We identified an insertion of eight residues that is con
109 Here
we identify an adaptive circuit-based mechanism that ena
110 Furthermore,
we identify an allele of the GTPase obgE that is synthet
111 We identify an ATS pertaining to which drug add-ons to r
112 We identify an interaction between HELLS and CtIP and es
113 discriminative epigenome annotation system,
we identified and assigned epigenetic states simultaneou
114 We identified and profiled both neuronal (glutamatergic
115 We identified and replicated 10 cerebrospinal fluid prot
116 In doing so,
we identified and validated previously uncharacterized R
117 Here,
we identify and functionally validate a CRC 'trio' const
118 We identified anteiso-branched-C18 SO (meC18SO) as the p
119 Furthermore,
we identified AP2IX-1 as a transcription factor that con
120 We identified apolipoprotein E (ApoE) as one of the prot
121 In this study,
we identified bHLH121 as an ILR3-interacting transcripti
122 We identified bi-allelic pathogenic KDELR2 variants as a
123 We identify binding sites for substrate K(+) and Cl(-) i
124 We identified both gene and protein markers for three te
125 We identified both known and novel interactors of the en
126 Here,
we identify BRCA2 and MEILB2-associating protein 1 (BRME
127 Here,
we identified c-Maf as an essential regulator of ILC3 ho
128 Taken together,
we identified CAMSAP3 as being important for the formati
129 We identify candidate members of the gut microbiome that
130 We identified caspase-2 as the antagonistic caspase that
131 Beyond these known subsets,
we identify CD4(-)CD8(-)TCRalphabeta(+), double-negative
132 We identified cell signaling molecular targets by meta-a
133 Using a genome-wide screen,
we identify centriole distal appendages as critical for
134 imity labeling and super-resolution imaging,
we identify CEP112 as a basal foot protein and other can
135 Finally,
we identified classes of DMS-defined variants with signi
136 We identified classical signatures of stress response in
137 n JBTS-associated proteins CEP104 and CSPP1,
we identified coiled-coil domain containing 66 (CCDC66)
138 We identified coinciding activation of pattern recogniti
139 We identify convergence of antibody sequences across SAR
140 Here,
we identify corresponding transcriptional profiles in hu
141 astatic prostate adenocarcinoma and CRPC-NE,
we identified CRPC-NE features detectable in the circula
142 We identified Csf2/GM-CSF as a primary complement-depend
143 We identified DBeQ as a promising lead compound for stru
144 croscopy with glutamate immunogold labeling,
we identified decreased intracellular glutamate density
145 to particular cell size control mechanisms,
we identify dependencies that point to potentially new m
146 We identified developmental trajectories of early-life m
147 Using the P L(2,3)-edge,
we identified different organic P species, including tho
148 quid 4-methyl-2-pentanol used as an example,
we identify different pressure-volume-temperature ranges
149 ncing (scRNA-seq) and genetic reporter mice,
we identified discrete lineages of intestinal antigen-sp
150 Here,
we identify disordered micrometer-size organic phases ra
151 We identify distinct mechanisms of resistance, in which
152 We identified Drosophila Tao kinase, the ortholog of the
153 In this report,
we identified dysregulation of members of the Dlk1-Dio3
154 an in vitro mouse oocyte development system,
we identified eight transcription factors, each of which
155 Finally,
we identified eleven colocalized outlier SNPs associated
156 Using this system,
we identified enhanced-fidelity SaCas9 (efSaCas9) (varia
157 Using epigenetic signatures,
we identified enhancers for each developmental stage.
158 of proteomics and gene expression analysis,
we identify enzymes involved in carbohydrate metabolism
159 We identified evolutionary divergence in the DNA methyla
160 We identified exposure-specific mutation spectra of each
161 Using Bacillus subtilis,
we identified factors that revealed the link between chr
162 combination of metabolic profiling and GWAS,
we identified FADS3 to be essential for forming Delta14Z
163 Through an RNAi screen,
we identified FBXO44 as an essential repressor of REs in
164 In this study,
we identify Fgl2 as a soluble TFR cell effector molecule
165 We identified five patients with biallelic variants in E
166 We identify five new colonies, and 21 additional colonie
167 We identified focal kidney fibrin thrombi in 6 of 42 (14
168 Using our method,
we identified,
for the first time, a hyper-mutation (kat
169 In conclusion,
we identified FOSL1 as a novel regulator of sepsis-induc
170 We identified four LysM-type receptors controlling nodul
171 We identify future research areas to accelerate the sust
172 Using RNA-seq and ChIP-seq approaches
we identified genes regulated directly and indirectly by
173 Furthermore,
we identify genes involved in cellular transport, includ
174 We identified genetic variants for MR analysis to invest
175 Here
we identify globally distributed haplotypes from 15,789
176 Using bioinformatics,
we identified Glt(Ph) gain-of-function mutations in the
177 Here
we identified gp83 of the thermophilic bacteriophage P74
178 We identified &
gt;6,000 protein phosphorylation sites that
179 ed of novel fibril-like substructures, which
we identify here by three-dimensional single-molecule su
180 by fluorescence lifetime imaging microscopy
we identified higher order assemblies containing 40 mole
181 We identify host-derived peptides of 4-11 residues, vary
182 We identified hundreds of candidate domestication genes
183 contiguous, chromosome-length scaffolds, and
we identify hundreds of TE insertions that were missed b
184 We identified important protein interaction sites, in ad
185 esolution microscopy and electron microscopy
we identified,
in adult cardiac myocytes, a Na(V)1.5 sub
186 We identify indicator micropollutants for further studie
187 hylodynamic model of influenza transmission,
we identified indicators of future evolutionary success
188 otheses in between waves of data collection,
we identified individual, contextual, and temporal condi
189 Within the CNS synaptic transmission set,
we identify individual significant candidate genes to wh
190 We identified individuals ages 18-64 with International
191 Applying PhyteByte to the human PPARG gene,
we identified irigenin, sesamin, fargesin, and delta-san
192 entral to the adaptation-enhancing principle
we identify is the ability of noise to mitigate populati
193 lar communication networks within the heart,
we identified key intercellular trophic relationships an
194 We identify key methodological shortcomings of current e
195 Here,
we identified Leishmania donovani heat shock protein 78
196 in vitro site-directed mutagenesis approach,
we identify loss of STAT3 O-GlcNAc at Threonine 717 as a
197 Specifically,
we identify LRRC8A, an essential component of volume-reg
198 Using bulk and single-cell RNA sequencing,
we identify molecular changes in the epidermal, fibrobla
199 We identify molecular markers that are predictive of dif
200 We identified Neural Cell Adhesion Molecule (NCAM1) as a
201 ofiling of the H3K27ac histone modification,
we identify neuron-subtype-specific regulatory elements
202 We identified neuronal Prkag3 mRNA as a mechanistic subs
203 Hence,
we identify new therapeutic targets and putative therape
204 Finally,
we identified NF-kappaB activator 1 (Act1), p38 mitogen-
205 ed datasets from human cancer RNA-Seq, where
we identify novel putative biomarker genes in different
206 We identified one of the involved cell types as sustaine
207 We identify outstanding questions and methodological cha
208 Using this motif,
we identified over 70 putative SNX-BAR ligands, many of
209 From metagenome-assembled genomes
we identified over 90 putative bacterial and archaeal ge
210 tive genomics and surface plasmon resonance,
we identified parasite-derived peptides that have the se
211 We identified PARP1 as an interacting partner for IAV HA
212 In the highly interconnected network,
we identify pathway communities and hundreds of previous
213 rtment (ED) database, between 2013 and 2016,
we identified patients who had refractory cardiac arrest
214 Using the Nationwide Readmissions Database,
we identified patients with aortic valve disease admitte
215 MEI genotypes with gene expression profiles,
we identify pMEI-associated expression quantitative trai
216 We identified potential angiocrine interactions between
217 We identified potential targets for repurposing of licen
218 We identify PRKCI, SPZ1, MUTYH, MAP2K4, FETUB, and TGFBR
219 on that do not result in ring hydroxylation,
we identified products formed after the initial reaction
220 We identified prohibitin as a potential target in mediat
221 We identified proinflammatory TNFalpha/NFkappaB signalin
222 Moreover,
we identified regulated phosphorylation sites in numerou
223 We identified residues specifically important for beta(2
224 ones harboring individual guide RNAs (gRNA),
we identify RNA-binding proteins (RBPs) that influence t
225 Using S. cerevisiae proteins,
we identified sequence and structural features within AL
226 use of Chromosome Conformation Capture (3C),
we identified sequences at the 5' and 3' boundaries of t
227 We identified seven previously unknown inhibitors of GMI
228 We identified several consequences of EAE that may contr
229 We identified significant clinical differences but also
230 We identified significantly associated cytosine-guanine
231 with long-range ferromagnetic spin systems,
we identified simple topological counting rules to predi
232 By performing an optimization analysis,
we identified six fractions with high peptide number and
233 We identify six antimicrobial peptides, two plant immune
234 Using a CRISPR-mediated knockout screen,
we identify SLC35B2 and myosin-7B (MYO7B) as critical en
235 screening assay that we previously reported,
we identified small-molecule compounds that modulate the
236 We identified specific gene combinations that were assoc
237 We identified specific gut bacteria and their metabolic
238 We identified splice junctions that could be generated o
239 From genetic screens in C. elegans,
we identified splicing factor RNP-6/PUF60 whose activity
240 Furthermore,
we identified that aging alters Sirtuin-1-hepatic nuclea
241 RS-CoV from persistently infected bat cells,
we identified that bat cells repeatedly selected for vir
242 We identified that open and active chromatin at the majo
243 Herein,
we identified that vascular endothelial growth factor (V
244 We identify that a fusion of the catalytic domain of TET
245 We identify that ARID1A inactivating mutations are prese
246 Here,
we identify that coactivator-associated arginine methylt
247 the model nucleophile N-alpha-acetyl-lysine,
we identified the alpha,beta-unsaturated dialdehyde 2-bu
248 We identified the aryl hydrocarbon receptor (AHR) pathwa
249 Using recombinantly expressed CLIC1,
we identified the best conditions to maximise protein in
250 In this study,
we identified the C. jejuni butyrate-modulated regulon a
251 Using FACS-assisted shRNA screens,
we identified the cell-surface adhesion receptor CD44 as
252 Subsequently,
we identified the cellular ring nuclease Crn1, which slo
253 We identified the chemical properties of the aggregation
254 We identified the decreased intestinal VDR significantly
255 Using positron emission tomography (PET),
we identified the dorsal striatum as the brain area most
256 Here,
we identified the E3 ubiquitin ligase Peli1 as an import
257 We identified the gene responsible for triuret decomposi
258 We identified the molecules bufalin and lycorine as drug
259 molecular modeling and mutational analyses,
we identified the nucleotide bases in the occluding mRNA
260 Specifically,
we identified the role of an early-season national show
261 Because STING directly binds to TRIF,
we identified the STING-interacting domain of TRIF and g
262 Based on six alternative land-use plans,
we identified the synergies and trade-offs between the b
263 Furthermore,
we identified the transcript profile of two cell states
264 Moreover,
we identified the viral peptide, HGSQHSGMIVNDTGHETDENRAK
265 Herein,
we identify the CBP/p300-interacting transactivator with
266 Here,
we identify the conditions and substrates that decouple
267 By means of a linear stability analysis
we identify the critical conditions triggering channel f
268 biodegradation pathways and mechanisms, and
we identify the current major knowledge gaps for future
269 We identify the dimensions of neural activity associated
270 Herein
we identify the dual function cytokine IL-33 as an orche
271 We identify the features of TOP sequences that determine
272 sis data from different strains and mutants,
we identify the general underlying design principles for
273 Here
we identify the nuclear receptor peroxisome proliferator
274 tion of the knot region by NMR spectroscopy,
we identify the SAM-binding region and observe changes i
275 We identify the SOX4 transcription factor as an importan
276 In this study,
we identify the vesicular soluble N-ethylmaleimide-sensi
277 We identify these intertwined density waves as being Fer
278 ransporter, NorC, from Staphylococcus aureus
We identified this antibody in a yeast display screen bu
279 erived neurons that model developing brains,
we identified thousands of genetic variants exhibiting a
280 We identified three DMRs at HTR2A, SLC17A9 and HDAC4 tha
281 We identified three eligible trials and were able to obt
282 We identified three such epitopes derived from highly co
283 We identify three CVA16-specific neutralizing monoclonal
284 We identified TmAAE1 and TmAAE5 as the most efficient en
285 Here,
we identified TOLLIP as a stabilizer of STING through di
286 Within posts made by users in our sample,
we identify topics that appear more often within users'
287 Here
we identify TRADD(4-6), an adaptor protein, as a direct
288 Finally,
we identified transcription factors that regulate stimul
289 We identified transcriptional signatures in the lymph no
290 hrough international research collaboration,
we identified twelve individuals with de novo loss-of-fu
291 Importantly,
we identified two FDA-approved drugs able to ameliorate
292 By gene expression analysis,
we identified two molecules that could have a role in th
293 Here
we identify two consanguineous families, each with two a
294 We identify two major classes of PVT neurons-termed type
295 We identify two robust spatial gradients of intrinsic dy
296 etion and U(S)3 kinase-dead recombinant MDV,
we identified U(S)3-responsive MDV genes during infectio
297 We identified Ube2v1 (ubiquitin-conjugating enzyme E2 va
298 st all of the transcriptional silencers that
we identified were also active enhancers in other cellul
299 s developed by the ENIGMA-DTI working group,
we identified widespread reductions in mean, axial and r
300 We identified YAP as a critical mediator of ERBB2 signal