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4 Using bioinformatics, mutation and NMR, we identify a 7-residue sequence, named P1 (residues 36-42),
6 Using RNA-seq profiling of the intima of lesions, here we identify a macrophage-specific lncRNA MAARS (Macrophage-As
11 Through intravital imaging of NSCs and their progeny, we identify a population of Gli1-targeted NSCs showing long-t
13 ely terminate transcription of bacterial CRISPR arrays, and we identify a widespread antitermination mechanism that antag
23 ions attributed to particular cell size control mechanisms, we identify dependencies that point to potentially new mechan
25 h a combination of proteomics and gene expression analysis, we identify enzymes involved in carbohydrate metabolism as tr
30 partment composed of novel fibril-like substructures, which we identify here by three-dimensional single-molecule superre
31 roduces highly contiguous, chromosome-length scaffolds, and we identify hundreds of TE insertions that were missed by Ill
33 Central to the adaptation-enhancing principle we identify is the ability of noise to mitigate population bo
34 gether with an in vitro site-directed mutagenesis approach, we identify loss of STAT3 O-GlcNAc at Threonine 717 as a driv
35 genome-wide profiling of the H3K27ac histone modification, we identify neuron-subtype-specific regulatory elements that
37 of large curated datasets from human cancer RNA-Seq, where we identify novel putative biomarker genes in different tumor
39 In the highly interconnected network, we identify pathway communities and hundreds of previously un
40 Using a CRISPR-mediated knockout screen, we identify SLC35B2 and myosin-7B (MYO7B) as critical endocyt
47 al characterization of the knot region by NMR spectroscopy, we identify the SAM-binding region and observe changes in the
50 Within posts made by users in our sample, we identify topics that appear more often within users' socia