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2 c signals and metabolic pathways can be readily elucidated, we induced a glycolytic tumor in the Drosophila wing imaginal
10 al agency over the VB movements, we conducted a study where we induced body ownership using visuotactile (VT) synchrony r
13 een the N terminus and loop2 in full-length Orai1 channels, we induced close proximity of the N terminus and loop2 via cy
15 role for PTEN in pre-B acute lymphoblastic leukemia (ALL), we induced Cre-mediated deletion of Pten in mouse models of p
18 By genetic sex reversal of a specific gut region, we induced female-like aging pathologies in males, associated
19 and the molecular mechanisms underlying these observations, we induced glomerular proteinuria in two animal models.
20 ere not the consequence of the actions of gonadal hormones, we induced gonadal sex reversal to alter the hormonal environ
22 on rather than one involving a specific GPP130 determinant, we induced homo-oligomerization of two unrelated Golgi-target
23 To identify the receptors involved, we induced latent sensitization in mice and rats by injecting
24 To study the role of Stat1 in a lupus model, we induced lupus-like chronic graft-versus-host disease (cGVH
25 e the metabolic requirements for productive KSHV infection, we induced lytic replication in the presence of inhibitors of
28 he role of CCR2 activation on Nf1(+/-) neointima formation, we induced neointima formation by carotid artery ligation in
31 tS in C57BL/6 mice by feeding them high-fat diet (HFD), and we induced periodontitis by periodontal injection of Aggregat
33 toxin-initiated OM inflammation on the olfactory bulb (OB), we induced persistent rhinitis in mice and analyzed the spati
34 ity lipoprotein receptor-deficient mouse (LDLR(-/-)) model, we induced plasma APN levels using a recombinant adenovirus e
37 the droplet monolayer with nanolitre injections of buffer, we induced quantifiable DHB tension, which could be related t
39 To decipher ribosome kinetics at stall sites, we induced ribosome stalling at specific codons by starving t
42 UBJECTS:: Male wild type and transgenic miceINTERVENTIONS:: We induced sepsis in mice using the colon ascendens stent per
44 2 (hAgo2) and Cryptosporidium single-stranded RNA (ssRNA), we induced specific slicing in Cryptosporidium RNA targets.
47 CMV Armstrong strain, but the more rapidly replicating LCMV-WE induced T cell exhaustion and viral persistence.
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