ライフサイエンスコーパス: we infected

1 To establish a model for study, we infected 39 macaques by feeding them single high doses of

2 Here, we infected 8 beagles through multiple experimental vector tr

3 e are naturally resistant to Brucella infection; therefore, we infected anti-interferon gamma (IFN-gamma)-treated, or IFN

4 We infected boa constrictors and ball pythons by cardiac inje

5 We infected boa constrictors and ball pythons with purified r

6 o examine the humoral immune response against B. miyamotoi, we infected C3H/HeN mice with B. miyamotoi strain LB-2001 exp

7 We infected C57BL/6 (wild-type [WT]) and mast cell-deficient

8 Thus, we infected C57BL/6 mice with two populations of Chlamydia mu

9 e if the destabilizing mutation affects viral pathogenesis, we infected C57BL/6 mice with WT or AM2 virus and found that

10 We infected cardiomyocytes using a short hairpin RNA lentivir

11 We infected caspase-1-deficient mice with murine gammaherpesv

12 le of gp130 signaling in vivo during acute viral infection, we infected Cd4-cre Il6st(fl/fl) mice, in which gp130 is cond

13 ysiological function for CXCL14 in innate immunity in vivo, we infected CXCL14-deficient mice with lung pathogens and we

14 We infected different primary cell types from mid- and late-g

15 e was a reduction in replication in cell cultures, and when we infected domestic swine, the natural host of CSFV host, we

16 To simulate ascending infection, we infected EEC aggregates with commensal and pathogenic bact

17 mortality; whereas to include the role of barrier defences we infected flies by dusting the cuticle with fungal spores.

18 To drive type 1 immunity in CNS tissues, we infected GL261 tumor-bearing mice with attenuated rabies v

19 of human corneal epithelial (HCE) cells to HSV-1 infection, we infected HCE cells at three different dosages of HSV-1 and

20 We infected Huh7 and Huh7.5.1 cells and primary human hepatoc

21 We infected human bronchial epithelial (NHBE) cells and murin

22 e the role of the OM response to B. pseudomallei infection, we infected human olfactory ensheathing cells (OECs) in vitro

23 in the transcriptional response to L. pneumophila METHODS: We infected human-blood-derived macrophages (BDMs) with L. pn

24 To mimic intracellular disease, we infected human-derived THP-1 macrophages with Mtb and inoc

25 y of IFIT1 to have an impact on negative-sense RNA viruses, we infected Ifit1(-/-) and wild-type control mice and primary

26 Here, we infected Irf1 (-/-) mice with two distantly related arthri

27 ls (PMNs) are required for diarrhea for Salmonella colitis, we infected kanamycin-pretreated interleukin 8R (IL-8R) mutan

28 f abortive infection in driving CD4(+) T cell loss in vivo, we infected macaques with simian-human immunodeficiency virus

29 To identify immune responses that are specific to MTB, we infected macrophages with eight different bacteria, includ

30 To investigate these differences, we infected male and female mice of different age groups with

31 We infected mice carrying different glycosylated forms of PrP

32 To investigate this, we infected mice depleted of platelet GPVI or CLEC2 by antibo

33 idate the role of Notch signaling during fungal infections, we infected mice expressing the pan-Notch inhibitor dominant

34 We infected mice that express different forms of glycosylated

35 To address this, we infected mice with a point mutation in Roquin1/Rc3h1 (sanr

36 To address this issue, we infected mice with Leishmania major and 2 wk later with ly

37 We infected mice with spores, that is, the infectious particl

38 To evaluate the role of sfRNA in flavivirus transmission, we infected mosquitoes with the flavivirus West Nile virus an

39 We infected neonatal C57BL/6 or C3H mice with H. pylori or tr

40 We infected nonhuman primate cell cultures and then crab-eati

41 We infected nontransformed human intestinal enteroid cultures

42 f Salmonella Typhimurium to replicate in human macrophages, we infected primary monocyte-derived macrophages (MDM) that h

43 Here, we infected rhesus macaques with simian-human immunodeficienc

44 To increase our understanding of the immune response, we infected the retinal pigment epithelial (RPE) cell line, A

45 f virulence determinants or the evaluation of therapeutics, we infected them with a green fluorescent protein-expressing

46 CD73 and extracellular adenosine in T. gondii pathogenesis, we infected wild-type (WT) and CD73(-/-) mice with T. gondii

47 We infected wild-type (WT) mice and AhR knockout (AhR KO) mic

48 To elucidate the mechanisms of this susceptibility, we infected wild-type and hepcidin-deficient mice with the si

49 We infected wild-type and IFN-alphabeta receptor knockout mic

50 To explore approaches to classify susceptibility to TB, we infected with MTB dendritic cells (DCs) from putatively re