1 We next determined downstream targets of YAP/TAZ in the poste
2 We next determined FAT1 expression levels in 64 muscles isola
3 We next determined if activation by phosphorylation caused th
4 EF-2 kinase is chaperoned by heat shock protein 90 (Hsp90),
we next determined if disruption of the Hsp90-eEF-2 kinase co
5 We next determined if higher expression of an X gene in XY co
6 alterations in the expression of bcl-2, or metal chelation,
we next determined if the poorly metabolized ketohexoses, tag
7 We next determined if these cytokines directly promote CD8(+)
8 We next determined that (1) GTN treatment of primary aortic s
9 We next determined that APC directly increases Rac1 activity,
10 We next determined that bridge axons could regenerate caudal
11 We next determined that E2F activity directly regulates and i
12 We next determined that intravascular administration of donor
13 We next determined that the same amount of cAMP produced from
14 We next determined that TNF-alpha secretion from both donor a
15 We next determined that within this region is also a potentia
16 We next determined the ability of orally vaccinated animals t
17 loss of apicobasal polarity are early hallmarks of cancer,
we next determined the cellular mechanism of oncogenic strati
18 We next determined the effect of 5-HT on mouse and human MC d
19 We next determined the effect of LH reversible inactivation b
20 inositol 3-kinase (PI3K)/Akt signaling in tumor promotion,
we next determined the effect of topical application of Lupeo
21 We next determined the effect on smooth muscle contractility
22 We next determined the exact location of the saccade-related
23 We next determined the interactome of three SMALED2-linked mu
24 We next determined the partial human SULT1C1 gene sequence fo
25 To examine the kinetics of rapamycin action,
we next determined the rapamycin sensitivity of rhabdomyosarc
26 tissue that revealed a prominent induction of murine CD73,
we next determined the role of CD73 in protection against int
27 We next determined,
using a series of luciferase reporters, t
28 tin receptors (MCR) are involved in some actions of agouti,
we next determined whether agouti's antilipolytic effect is e
29 We next determined whether Ascl1 prevented RGC development.
30 ltiple myeloma cell adhesion to both fibronectin and BMSCs,
we next determined whether CHIR-12.12 inhibits this process.
31 We next determined whether cholesterol release leads to chang
32 We next determined whether GM1 induces the release of neurotr
33 We next determined whether ipsilateral responses evoked in on
34 We next determined whether matrix metalloproteinase-9 (MMP-9)
35 We next determined whether MbetaCD treatment induced tyrosine
36 ieties can be attached to glycolipids (i.e., gangliosides),
we next determined whether MNV-1 uses gangliosides during inf
37 We next determined whether PKC activation abbreviates the tim
38 We next determined whether prospectively computed expression
39 We next determined whether SCD1 deficiency could prevent diab
40 We next determined whether the effect of systemic PPF on rein
41 We next determined whether the results obtained using culture
42 We next determined whether VacA physically associates with li
43 FKHR/FOXO1a, BAD, and AFX/FOXO4) to regulate cell survival,
we next determined which downstream effector(s) is activated
44 We next determined which phase of PKD activation is responsib