1 We next examined a potential dopaminergic mechanism for this
2 We next examined a potential role of PTP1B in VEGF-induced an
3 We next examined a virion-enriched band purified by density g
4 We next examined changes in ApRas and ApRap activity that acc
5 We next examined cocaine-elicited effects on wild-type human
6 We next examined embryos with a tissue-specific deletion of t
7 ve mechanisms to compensate for fluctuations in TBP levels,
we next examined expression of the two known vertebrate TBP h
8 We next examined HGF responsiveness in pancreatic cancer line
9 We next examined how vHipp CB1R signaling may control the sal
10 tionality during the later phases of chronic toxoplasmosis,
we next examined if adoptive transfer of functional CD8(+) T
11 We next examined if Pol iota affected tandem mutations genera
12 We next examined KP1019 sensitivity of strains defective in D
13 thout exerting a specific functional effect on these cells,
we next examined NKT cell biology on a per-cell basis.
14 We next examined potential roles of SAP97 and 4.1N in cocaine
15 We next examined responses to individual alarm pheromone comp
16 We next examined the action of SST and subtype-selective SST
17 We next examined the activity of a tetrameric Cin8 lacking on
18 We next examined the activity of prominent interneurons withi
19 We next examined the alterations that occur in the R6/2 mouse
20 We next examined the biochemical basis for the loss of activi
21 We next examined the contribution of NEIL3 to the maintenance
22 We next examined the effect of Tia1 deletion in novel C (+/+)
23 We next examined the effects of these lesions on action seque
24 etermine whether these observations were promoter specific,
we next examined the effects of using the CaMKIIalpha promote
25 We next examined the efficiency of initiation at AUG, CUG, an
26 We next examined the F2 generation and F3 generation of mice
27 We next examined the function of different domains within the
28 We next examined the functional consequences of the observed
29 We next examined the impact of hDna2 on OF maturation and rep
30 We next examined the influence of amino acids on these free f
31 We next examined the interactions of soluble GP64 proteins an
32 We next examined the potential of both BTN1A1 and BTN2A2 to i
33 We next examined the regression analyses that purportedly yie
34 We next examined the response of 48 h pretreated cultures to
35 We next examined the responses of the healthy muVM to a vasot
36 ecause the endocannabinoid system regulates aBNST activity,
we next examined the role of cannabinoid type-1 receptors (CB
37 We next examined the role of latency-associated transcript (L
38 We next examined the role of the system Xc(-) transporter in
39 Since Rab8a is implicated in vesicular trafficking,
we next examined this process in Ahi1-knockdown cells.
40 We next examined whether AVP signaling in kisspeptin neurons
41 To address the role of NE in mammary tumorigenesis,
we next examined whether deregulated NE activity enhances mam
42 We next examined whether DH infusion of a behaviorally subeff
43 We next examined whether direct or indirect projections of VH
44 We next examined whether miRNA-206 and -9* modulate the aberr
45 We next examined whether other members of the Drosophila Syt
46 nduces matrix calcification of valvular and vascular cells,
we next examined whether RANKL mediates forskolin-induced mat
47 We next examined whether selectively elevating MBV without in
48 We next examined whether the high plasma free fatty acid (FFA
49 We next examined whether the spatial subcellular localization
50 We next examined whether these effects were due to disrupted