1 We next tested 149 known cancer risk loci for eQTL effects, a
2 We next tested a separate cohort of cases from University of
3 We next tested candidate-specific immune responses.
4 We next tested five hypotheses to explain sex biases in domin
5 We next tested for ethanol-induced changes in striatal-relate
6 We next tested how presynaptic Ca(2+) transients affect the s
7 We next tested if food anticipation was directly encoded on a
8 We next tested interventions to reduce pragmatic bias.
9 We next tested Plek2 inhibitors alone or in combination with
10 We next tested refinements to our hypothesis, focusing on how
11 We next tested several isolated cardenolides in vitro on the
12 We next tested signaling through chimeras between the Galpha(
13 We next tested the abilities of both Dr(+) E. coli and a Dr(-
14 We next tested the ability of transduced DCs to raise class I
15 We next tested the capacity for hair cell regeneration follow
16 We next tested the causal link between the correlative human
17 GF can be cleaved to tcHGF by plasminogen activators (PAs),
we next tested the cultures for active PA.
18 We next tested the current linear model for polarity establis
19 tion normalizes PPI deficits evoked from nucleus accumbens,
we next tested the degree to which VP inhibition would normal
20 We next tested the effects of primaquine and dynasore on SK2
21 We next tested the effects of SMIT1 co-expression, in the abs
22 We next tested the effects of the influenza virus inhibitors
23 We next tested the functional role of TGFbeta/activin/nodal s
24 We next tested the growth of three tumor models from differen
25 We next tested the hypothesis that caspase activation led to
26 Based on this evidence,
we next tested the hypothesis that COX expression might promo
27 Because both features were also found in intron 13,
we next tested the hypothesis that intron 13 would be involve
28 On the basis of previous studies,
we next tested the hypothesis that NO decreases RhoA activity
29 e sensitivity of oxyR oprL bacteria to planktonic H(2)O(2),
we next tested the hypothesis that the biofilm mode of growth
30 We next tested the hypothesis that the SNAT4 isoform is funct
31 We next tested the in vitro and in vivo efficacy of vemurafen
32 We next tested the in vivo relevance of these findings using
33 We next tested the requirement for five EMT transcription fac
34 We next tested the requirement for proliferation and migratio
35 We next tested the response of primary and established cancer
36 learn the mechanism of thrombin-stimulated iPLA2 activity,
we next tested the role of p38 MAPK.
37 We next tested the selectivity of this system for BCC.
38 We next tested the soluble factors identified through in sili
39 matrix protein, is highly expressed in both types of cells,
we next tested,
using a transgenic approach, whether a 9.6-kb
40 We next tested various mutant forms of the FAC polypeptide fo
41 We next tested whether CD40, whose engagement delivers a pote
42 We next tested whether CD40-induced signaling pathways were d
43 We next tested whether Follistatin suppresses Myostatin activ
44 d Ras-Raf-induced NF-kappa B activation in monocytic cells,
we next tested whether HIV-induced NF-kappa B activation was
45 We next tested whether P. carinii genomic cdc2 (with six intr
46 We next tested whether the alterations in perinucleolar heter
47 We next tested whether the DDD or DDE mutants cleave single-s
48 orine to tacrolimus is an effective treatment for early CR,
we next tested whether these two immunosuppressive drugs dire
49 We next tested whether thirst and sodium appetite require ang