1 We next addressed one-bond NMR coupling constants in ethers a
2 We next adopted a chemogenetic approach using a 5-HT2CR(CRE)
3 rscore a potentially significant role of 5RK in exocytosis,
we next amperometrically analyzed catecholamine release from
4 We next analyzed human transcriptomes and found expression co
5 We next applied these rules to the creation of a synthetic di
6 We next apply this strategy to two brownmillerite-structured
7 We next assessed the association of MCID estimates for improv
8 We next characterized muscle-specific isoforms of BIN1 and DN
9 We next compared differences in Nav subtype expression betwee
10 We next conducted a clinical study with more than 100 blond-
11 We next conducted an investigator-blinded, randomized, bilate
12 ng can be motivated either by selfish or otherish concerns,
we next consider the costs and benefits of the motivation und
13 We next describe critical EBP competencies and the challenges
14 We next developed this technique for quantitative biodistribu
15 We next discuss current data on the mechanisms of sexual diff
16 We next discuss the most popular theories regarding general m
17 We next employed inducible lineage tracing to fate map, throu
18 To transition seamlessly between environments,
we next established a freely controllable virtual reality sys
19 We next examined a potential role of PTP1B in VEGF-induced an
20 We next examined the effect of Tia1 deletion in novel C (+/+)
21 We next explored the effect of arginine on pH dynamics by pre
22 We next exposed females to blood meals supplemented with allo
23 We next found that S1P effectively could reverse alcohol-indu
24 We next generated PH and PA strain-specific Th17 clones and s
25 We next identified residues important for the covalent attach
26 We next identified that the phosphorylation of Myristoylated
27 We next identify signatures that are differentially active un
28 of resting UCB monocytes from both groups were comparable,
we next investigated the role of epigenetic differences.
29 We next investigated the role of Ucp2 in cytokine-induced apo
30 We next isolated two anti-muPA nanobodies; an active-site bin
31 To identify proteins mediating apigenin's effect,
we next overlapped a 122-gene signature unique to HSCs with a
32 We next perform over 80,000 in silico experiments to infer ho
33 We next profiled the efficacy of compounds targeting CHK1 and
34 We next recorded from and controlled homologous neuromodulato
35 We next review evidence that the effects of social influence
36 We next show that BCL11A(2-16) pulls down RBBP4, RBBP7, and o
37 We next show that optogenetic activation of GHT neurons selec
38 Critically,
we next show that parietal tSMS enhances the detection of nea
39 We next show that, when co-applied with visual stimuli, the m
40 We next showed a link between NANOG and autophagy activation
41 We next showed that coordination of calcium by an EF-hand in
42 We next summarize designs of reporters and biosensors and des
43 We next tested the effects of primaquine and dynasore on SK2
44 We next tested the effects of SMIT1 co-expression, in the abs
45 We next tested the in vitro and in vivo efficacy of vemurafen
46 We next tested the requirement for proliferation and migratio
47 We next uncovered that HDAC2 is a direct target of cAMP respo
48 ervate overlapping populations of ventral pallidal neurons,
we next used optogenetics to examine whether changes in synap
49 We next used pre-pubertal hormone treatment to model early pu
50 We next used viral adaptation and a set of Vif mutants to ide