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1  phenotypes for assessing the defensive role of terpenoids, we overexpressed a bifunctional spruce IDS, a geranyl diphosp
2                                                             We overexpressed a decahistidine-tagged version of SCO5745 an
3 n fluxes along the photosynthetic electron transport chain, we overexpressed a minor pea (Pisum sativum) Fd isoform (PsFd
4                                                             We overexpressed a TNC fragment containing the assembly and E
5                                                       Here, we overexpressed an active form of PKMzeta in rat hippocampus
6                                                             We overexpressed an Arabidopsis PME inhibitor to investigate
7                                                             We overexpressed an inactive mutant of Atg5 to create an auto
8 gE to five recombinant beta-conglutin isoforms (rbeta) that we overexpressed and purified and to their natural counterpar
9 xtensions on the enzymatic behavior of individual isoforms, we overexpressed and purified the three full-length human iso
10       To investigate how these mutations confer resistance, we overexpressed and purified wild-type (WT) HCMV Pol and thr
11                                                             We overexpressed Arabidopsis WRI1 in seeds of a transgenic li
12 r ASCL1 could restore neurogenic potential to mammalian MG, we overexpressed ASCL1 in dissociated mouse MG cultures and i
13  To further confirm the key role of beta-arrestin proteins, we overexpressed beta-arrestin2-(1-320), a dominant negative
14 tion sites directly caused medulloblastomas to disseminate, we overexpressed candidate genes in Nestin(+) neural progenit
15                                    To test this hypothesis, we overexpressed CXCR4 in HaCaT KC cells and treated them wit
16 2 and ALS4 mutations differentially affect gene expression, we overexpressed disease-specific SETX mutations in senataxin
17                                            In HEK293 cells, we overexpressed E2F1, which significantly reduced OGT and MG
18                                                  Therefore, we overexpressed each of the 6 rat 12/15-LOX enzymes in HEK-2
19 arget sequences to leverage endogenous EC-specific miR-126, we overexpressed exogenous p27 in VSMCs, while selectively in
20  Further, CXCR3-B-induced apoptosis was down-regulated when we overexpressed HO-1.
21                                    To accomplish this goal, we overexpressed human A53T-alpha- synuclein (hA53T-alpha-syn
22                                                             We overexpressed human eNOS W447A and W447F mutants in novel
23 rocal dosage change of Rbm8a alters embryonic neurogenesis, we overexpressed human RBM8A in two animal models.
24          To gain mechanistic insight into HvCBF4/CBFIV CBFs we overexpressed Hv-CBF2A in spring barley (Hordeum vulgare)
25                                 To determine the mechanism, we overexpressed hyaluronidase 1 (Hyal1) as a fluorescent fus
26    To identify yet unknown oncogenic functions of miR-106b, we overexpressed it in LNCaP human prostate cancer cells to e
27 significance of JAZ in the regulation of neuronal survival, we overexpressed it in neurons.
28                                                             We overexpressed lin-28 homolog A (Lin28a) to inhibit let-7 m
29                                                             We overexpressed microRNA160 to silence a set of repressor au
30                                                             We overexpressed microRNA393 to silence the auxin receptor ge
31       In order to characterize the functional significance, we overexpressed miR-335 in human cancer cells and found that
32                                                       Here, we overexpressed mouse DGKeta in human embryonic kidney 293 c
33                               This effect is abolished when we overexpressed mutants lacking part of N-terminal domains,
34 Tmc interaction is relevant to mechanotransduction in vivo, we overexpressed N-terminal fragments of Tmc2a in zebrafish h
35                                                To this end, we overexpressed normal and mutant COL4A3 chains (G1334E muta
36          To investigate the function of NR4A1 in lymphomas, we overexpressed NR4A1 in several lymphoma cell lines.
37                                                             We overexpressed PDR1 (PDR1 OE) to investigate whether increa
38                To study whether miR-181c regulates mt-COX1, we overexpressed precursor miR-181c (or a scrambled sequence)
39               To address this conundrum in skeletal muscle, we overexpressed Rad and Rem in flexor digitorum brevis (FDB)
40                                                             We overexpressed Secondary Wall-Associated NAC Domain 1s (Ptr
41 onship between p-FAK-Tyr(397) and localization of nectin-3, we overexpressed sFRP1 using lentiviral vectors in the Sertol
42                                                        Here we overexpressed SGLT1 in MDCK cell monolayers and reconstitu
43                                                             We overexpressed SHB1 in canola to explore the possibility of
44                                                             We overexpressed stromal interaction molecule 1 (STIM1) and O
45                                                             We overexpressed the mutant constructs in HEK 293T cells and
46                                                       Here, we overexpressed the PtdSer receptor BAI1 in mice lacking Mer
47                                    To test this hypothesis, we overexpressed Trx-1 (cytosolic form of Trx) in the VMH of
48                                                    Finally, we overexpressed ts-46 and ts-47 in two lung cancer cell line
49                                                             We overexpressed various forms of CREB1, CREB2, or cJun in th
50                                                             We overexpressed Wnt5a, Wnt8a, Wnt16, and WISP1 in the synovi

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