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1                                                             We present evidence for a novel role of astrocytes in PA flow
2                                                             We present evidence for an A-type K(+) current in spinal CPG
3                                                             We present evidence for cytoplasmic translocation of RNase II
4                                                             We present evidence for functional A2A receptors, which have
5                                                             We present evidence for intercellular communication in the br
6                                                             We present evidence for lake redox stratification, establishe
7                                                             We present evidence for photoreception via the light-sensitiv
8                                                             We present evidence for the co-variation of the Type III comp
9                                                             We present evidence for the existence of two different reacti
10                                                             We present evidence for the imbalance between inflammatory an
11                                                             We present evidence for the presence of two distinct mechanis
12                                                             We present evidence for the role of alpha2 in facilitating PT
13                                                             We present evidence for two inactivation pathways that are op
14                                                             We present evidence for vessel-to-neuron communication in the
15 zing a variety of computational and biochemical approaches, we present evidence for a class of nuclear introns that relie
16 tion, molecular dynamics simulations, and electrochemistry, we present evidence for two populations of PmB molecules: pep
17                                                    Finally, we present evidence for an electrostatic interaction between
18                                                        Here we present evidence for a different mechanism in which two se
19                                                        Here we present evidence for a neural mechanism for feature bindin
20                                                        Here we present evidence for a neuronal computation that maximizes
21                                                        Here we present evidence for a palaeo-subglacial lake on the Antar
22                                                        Here we present evidence for a preponderance of exocellular As in
23                                                        Here we present evidence for a separate generator in local circuit
24                                                        Here we present evidence for an alternate view of core formation b
25                                                        Here we present evidence for hyperphosphorylation as a mechanism a
26                                                        Here we present evidence for microtubule (MT)-based targeting of a
27                                                        Here we present evidence for persistent active drainage networks-i
28                                                        Here we present evidence for phosphorylation-driven regulation of
29                                                        Here we present evidence for previously unappreciated B-cell immun
30                                                        Here we present evidence for strong diastereomeric interaction eff
31                                                        Here we present evidence for successful use of morpholino oligomer
32                                                        Here we present evidence for the formation of aerosol particles fr
33                                                       Here, we present evidence for a direct recruitment of UHRF1 by the
34                                                       Here, we present evidence for a third strategy: directionally selec
35                                                       Here, we present evidence for a unique, tonically active, excitator
36                                                       Here, we present evidence for DNA damage as an unexpected means to
37                                                       Here, we present evidence for glucagon-like peptide 1 (GLP-1)-media
38                                                       Here, we present evidence for multiplexed encoding of tactile skin
39                                                       Here, we present evidence for oncogene-directed cancer metabolic re
40                                                       Here, we present evidence for the influence of phospholipid bilayer
41                                    Using this novel method, we present evidence for direct proportionality of volumetric
42                      Using Arabidopsis thaliana as a model, we present evidence for the involvement of a plastidial cis-p
43 first report of N-N coupling between NO molecules in a MOF, we present evidence for a species that is consistent with a f
44                                               In this paper we present evidence for this mechanism in nanograined Yttria
45                                       In the present paper, we present evidence for the idea that speech motor learning i
46                                             In this report, we present evidence for an alternative mechanism of deglutath
47 rges after the first retinal synapse.SIGNIFICANCE STATEMENT We present evidence for spatially antagonistic interactions b
48                                              In this study, we present evidence for the intracellular delivery and ultras
49                                              In this study, we present evidence for two spontaneous mutations as the mole
50                                                       Thus, we present evidence for a new mechanism of Zap1-mediated gene

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