1 We propose that (
1) like its veterinary relatives, the o
2 We propose that a cocaine-induced shift from MRN-driven
3 We propose that a combination of failed clearance and ex
4 We propose that a dense liquid phase (containing 4-7 H2O
5 We propose that a hairpin stem length of 36 +/- 3 nt is
6 We propose that a pharmacological compound that abates T
7 We propose that a risk factor for seizures in patients w
8 We propose that a SMYD2-H4K20me1-L3MBTL1 axis contribute
9 We propose that a specific conformation of the Smc3p hin
10 Thus,
we propose that a threshold for the conformational entro
11 We propose that a value of kappas=1.1 (at RH=90%) is use
12 We propose that A. oligospora likely evolved the means t
13 Hence
we propose that absence of MAP1LC3A disrupts the autopha
14 We proposed that ACP in California was likely not introd
15 We propose that active cellular processes that can creat
16 We propose that ancient ancestral origins for ciHHV-6A a
17 We propose that antagonist binding prevents structural r
18 We propose that at RdDM target regions, PKL may be requi
19 We propose that AtLAZY proteins control plant architectu
20 We propose that autoreactive TH1/TH17CM cells expand in
21 We propose that bicarbonate enhances the rate of CO prod
22 We propose that bivalent interactions with the double st
23 We propose that blockade of IL-23 should have a therapeu
24 h and without oxygen and radical scavengers,
we propose that boron-imidates form under the basic reac
25 Based on these results,
we propose that brain activity is naturally structured i
26 Based on these data,
we propose that BRC1 regulates bud activation potential
27 We propose that BRCA2 is dispensable for RAD51-mediated
28 We propose that cell division arrest is programmed in co
29 Accordingly,
we propose that cell-cell interaction may be a factor re
30 We propose that chaperones such as trigger factor can wo
31 We propose that chronic suppression of calbindin by Delt
32 Finally,
we propose that cilia decapitation induces mitogenic sig
33 We propose that CML41 enables Ca(2+) -signalling specifi
34 We propose that co-option of single-copy genes may be a
35 We propose that,
conceptually, the principle of cognitio
36 We propose that,
contrary to the established paradigm, T
37 We propose that coupled use of Sr/Y and La/Yb is a feasi
38 We propose that CRP K100 acetylation could be a mechanis
39 We propose that CrPV-1A is a multifunctional, versatile
40 Although mechanistic studies are ongoing,
we propose that CuH-catalyzed silylation of unsaturated
41 We propose that Cx43 channels are important conduits for
42 We propose that DCAF15 expression may be a useful biomar
43 We propose that defective intercellular adhesion contrib
44 We propose that defects in intron removal in SMA promote
45 We propose that deficits in IP3-mediated Ca(2+) signalin
46 We propose that demographic range expansion of a microbi
47 Building on evidence from animal models,
we propose that deprivation accelerates the neurodevelop
48 We propose that despite the predominance of less defende
49 We propose that development of CARM1-specific inhibitors
50 nd induce virulence gene expression in EHEC,
we propose that DHMA acts as a molecular beacon to targe
51 We propose that differences in glycosylation between H3N
52 Thus,
we propose that discrete gene mutations in intellectual
53 We propose that disparate proinflammatory host signature
54 We propose that DNA melting is an active process initiat
55 With this evidence,
we propose that DNAJB9 is a strong biomarker for rapid d
56 We propose that double locking of the vieSAB promoter by
57 We propose that DR serotonergic neurons preempt reward d
58 We propose that early after trauma, complete dendritic s
59 We propose that emergent biophysical properties associat
60 We propose that epigenetic drift is a determinant of lif
61 We propose that EVs provide a physiologic link between m
62 We propose that exophers are components of a conserved m
63 We propose that exosomal muscle-specific miRNAs may be u
64 Thus,
we propose that FACT acts as a sensor of ADS formation i
65 We propose that FANCD2 and FANCI contribute to the organ
66 ity of molecular mechanisms involved [5, 6],
we propose that fibers generically arise from the aggreg
67 We propose that focal breast cancer susceptibility, at l
68 We propose that force-enhanced stator adhesion allows th
69 We propose that frequency-dependent behavioural and spat
70 Furthermore,
we propose that frequent opsin gene loss had a large inf
71 Based on these results,
we propose that FtsA antagonizes lateral interactions be
72 We propose that future research on alpha neurofeedback s
73 Based on our findings,
we propose that glia clear neurotoxic Abeta peptides in
74 We propose that glucosylsphingosine, a sphingolipid accu
75 Based on these findings,
we propose that GPCR signaling from endosomes functions
76 We propose that GPR88 expression within the striatum is
77 We propose that gradients of extracellular metabolites a
78 We propose that HDT1/2 function as part of a mechanism t
79 We propose that head-to-head motors may generate energy
80 We propose that HID-1 influences early steps in LDCV for
81 Overall,
we propose that HIV infection increases Cx43 expression
82 Thus,
we propose that HNF-1beta links extracellular inflammato
83 We propose that host interactions with microbes are crit
84 Therefore,
we propose that hydroxyproline modulates the rate of Zip
85 We propose that identification of PRC1-Br140 "bivalent c
86 Based on our findings,
we propose that IFSA is a potential threat to genomic st
87 We propose that IHC functional differentiation into matu
88 ere growth phenotype than cells lacking zwf1
We propose that in S. pombe Gcd1 and Idn1 act together t
89 We propose that in the absence of plasmon waves in biolo
90 We propose that in the course of CRISPR interference Cas
91 We propose that,
in concert with other drivers, an overp
92 We propose that,
in cyanobacteria, the CTDHs are caroten
93 We propose that,
in full-length TDP-43, association betw
94 We propose that infections of rhesus macaques with SIVma
95 Taken together,
we propose that intercellular connectivity supported by
96 We propose that Internet of Things technology can be use
97 in region is often spared in aphasic stroke,
we propose that it is a sensible target for future resea
98 We propose that LFY and AP1/CAL act as part of an incohe
99 We propose that,
like the human Y chromosome, the chicke
100 We propose that LKB1 acts as a safeguard against HPV-sti
101 We propose that measuring the levels of cyclin D3-CDK6 i
102 Here,
we propose that meiosis I-specific modulators of reducti
103 We propose that microRNA-200a functions as a "cell cycle
104 We propose that miR-26a delivery might not be a viable t
105 We propose that miRNAs compensate for lineage-specific d
106 lls and the known mutagenic profile of DnaE,
we propose that misincorporated ribonucleotides are remo
107 Thus,
we propose that mitochondrial calcium deregulation is a
108 We propose that mitotic cell rounding in columnar epithe
109 We propose that modifications in the catalytic head, tog
110 We propose that momentum-space mapping of the second-ord
111 Therefore,
we propose that more multifactorial experiments should b
112 We propose that mTOR folate sensing in trophoblast cells
113 We propose that multiple factors, including the SdhE ass
114 We propose that MUS81 provides a mechanism of replicatio
115 We propose that NDK5 dimer is an RS structural subunit w
116 We propose that NE enhances odor responses not through d
117 We propose that neuroscience research suggests that a si
118 We propose that NHE9 regulates TfR-dependent, recycling-
119 Thus,
we propose that NMB and GRP may transmit discrete itch i
120 ated analysis of HPC polarity and mechanics,
we propose that normal heart development requires bilate
121 We propose that novobiocin will be a useful tool for und
122 Based on these results,
we propose that NTRC plays a pivotal role in chloroplast
123 We propose that nuclear sub-domains, such as the nucleol
124 We propose that OM increases the myosin duty ratio, whic
125 We propose that opioid genes are regulated as interconne
126 We propose that ORCA coordinates with the histone and DN
127 Thus,
we propose that OsbHLH068 and AtbHLH112 share partially
128 We propose that p-S10H3 is a novel marker for nociceptiv
129 We propose that P2 characterizes familiarity with sound
130 We propose that paused Pol II helps prevent new initiati
131 We propose that PCNA and ATP-dependency serve as a multi
132 We propose that peropsin affects light-dependent regulat
133 Collectively,
we propose that PHF5A-SF3B1 forms a central node for bin
134 We propose that phosphorylation of 53BP1 at S380 acceler
135 We propose that physiological hypoxia coordinates homeos
136 We propose that PICK1 is a cargo-specific endocytic acce
137 moid time course of CFTR current activation,
we propose that PKA phosphorylation of the R domain is e
138 We propose that plants sense multiple abiotic stresses t
139 We propose that poising the fast-activating enhancers of
140 We propose that polarity-induced spatiotemporal control
141 Finally,
we propose that precise fine-tuning of conduction along
142 We propose that predation by bacteriophages that use T4P
143 ly shown to be required for CDK4 activation,
we proposed that proline-directed kinases might specific
144 We propose that PRR2 contributes to salicylic acid (SA)-
145 We propose that radiation-induced STING activation is im
146 We propose that rapid alteration of PTEN activity throug
147 Based on these results,
we propose that recycled jute should be considered for f
148 In light of these findings,
we propose that repurposing statin drugs for topical tre
149 We propose that residual T cell populations in resolved
150 We propose that Rev multimerization and NES masking regu
151 Therefore,
we propose that RNase H-deficient mutants convert some R
152 Thus,
we propose that RPA functions as a platform for targetin
153 Taken together,
we propose that RR and its signaling pathway may serve a
154 We propose that self-assembly of centromeric chromatin i
155 We propose that semaphorin-plexin signalling is an essen
156 We propose that SF3b1 functions to stabilize weak U2/BS
157 We propose that shielding of the cavity ensures that the
158 Based on these results,
we propose that signaling pathways involved in structura
159 We propose that similar DNA codes might be used to confe
160 We propose that small changes in the separation of the i
161 We propose that SOCE controls a critical "metabolic chec
162 We propose that some spectroscopic methods are influence
163 We propose that specific toxic RNAi-active sequences pre
164 Thus,
we propose that specifically because tDCS is diffuse, we
165 Hence,
we propose that spike timing-dependent plasticity enable
166 We propose that spinal cord regeneration in geckos repre
167 We propose that Spt4/5 may be important to coordinate th
168 We propose that sterile inflammation should be considere
169 In sum,
we propose that structural priming provides a new basis
170 We propose that structured RNAs prevent p53C aggregation
171 ugh we have focussed on the Trinity package,
we propose that such clustering is a useful initial step
172 We propose that such performance is likely due to the co
173 We propose that sulfatide is recognized only by human iN
174 We propose that sustained high catecholamine concentrati
175 We propose that switching between these DNA polymerases
176 urate and robust quantitation than XCMS, and
we propose that tailored peak detection should be the pr
177 We propose that targeted delivery of GD1a to MS lesions
178 We propose that Tau's GDP preference allows the cell to
179 We propose that tau-tubulin can be described as a "fuzzy
180 We propose that TDW identifies a discrete global brain a
181 We propose that temperate phages do not need to carry an
182 We propose that the ABA and DOG1 dormancy pathways conve
183 We propose that the activation of ELK channels involves
184 We propose that the affective reversal of sadness in mus
185 e DELLA repressors, substrate of SCF(SLY)(1)
We propose that the alf4 phenotype is partly due to incr
186 We propose that the ALT pathway preferentially occurs at
187 We propose that the antagonistic effects of H2A.Z and HS
188 We propose that the appearance of larger ice sheets over
189 We propose that the assembled tripartite pump acts as a
190 Here,
we propose that the bandgap in CH3NH3PbI3 has a direct-i
191 Based on these results,
we propose that the binding of each stator-unit is enhan
192 We propose that the blunted cholinergic excitability con
193 We propose that the bone marrow niche can be altered by
194 of capsomers in the cryo-EM reconstruction,
we propose that the capsids of CroV and related giant vi
195 We propose that the cell's transcription pattern is larg
196 We propose that the charged region and chain directional
197 We propose that the cis interaction with P2Y2R provides
198 We propose that the combinatorial use of transcription f
199 Taken together,
we propose that the concerted action of DDK, Polo-like k
200 We propose that the configuration of DNA polymerases at
201 We propose that the coupling of ocean chemistry and Earl
202 approaches have not yielded effective drugs,
we propose that the development of new antibiotics aroun
203 We propose that the differential expression of inhibitor
204 We propose that the direct binding to IL-1beta to integr
205 Instead,
we propose that the distinction between language and ges
206 We propose that the distinctive responses to O2 and H2O
207 We propose that the elevational gradient in the Amazonia
208 We propose that the evolutionary preferred positioning o
209 Finally,
we propose that the fourth band corresponds to the accum
210 We propose that the hard pericarp with this biomechanica
211 We propose that the higher-conducting states arise from
212 We propose that the HIRA chaperone complex represses inc
213 We propose that the homeostasis of the non-AUG translato
214 We propose that the hominin lineage experienced a declin
215 We propose that the hot electron and hole carriers excit
216 We propose that the HSP90/R2TP chaperone system promotes
217 We propose that the hybrid phenotype contributes to meta
218 We propose that the increase in photocatalytic activity
219 We propose that the induction of the post-transcriptiona
220 Taken together,
we propose that the influence of O2 availability on the
221 We propose that the inhibition of chromatin assembly rep
222 We propose that the interaction between the lncRNA, its
223 We propose that the involvement of STN in reactive contr
224 Based on these results,
we propose that the light-dependent regulation of CKI1 p
225 We propose that the long noncoding RNA species in the D-
226 We propose that the lumen-exposed residues, threonine 16
227 the regulation of gene expression by miRNAs,
we propose that the main function of m(6)A is post-trans
228 We propose that the maintenance of an existing IS is a d
229 We propose that the mechanochemical feedbacks underlying
230 We propose that the modulation of cortical myosin dynami
231 We propose that the modulation reflects a decision-relat
232 We propose that the observed continuum represents incohe
233 We propose that the opposing activities of ULK1-mediated
234 We propose that the orthogonal framework used in this wo
235 We propose that the overall lack of changes in synaptic
236 We propose that the polymerization-condensation dynamics
237 ogression among HIV-1 subtypes; furthermore,
we propose that the poorer viral replicative capacity of
238 Instead
we propose that the presence of ceramide in one of the m
239 We propose that the presence of rhythmically-modulated n
240 We propose that the primary mechanism of action of acyla
241 imal models and observational human studies,
we propose that the psychoneuroimmunology of early-life
242 e behavioural constellation of deprivation."
We propose that the relatively limited control associate
243 Thus,
we propose that the Sec61-IRE1alpha complex defines the
244 We propose that the shared evolutionary homology of teet
245 We propose that the siRNA pathway promotes X recognition
246 We propose that the software's reliability, flexibility,
247 We propose that the spatiotemporal dynamics of mRNAs can
248 We propose that the STRIPAK complex modulates dynein act
249 We propose that the tension generated by the E-cadherin/
250 We propose that the timing of Eco1 phosphorylation, and
251 Thus,
we propose that the uncontrolled expression of AtHB1 is
252 We propose that the uncoupling of neurogenesis and diffe
253 We propose that the upregulation of core PCP components,
254 We propose that the very high strength of SPB-microtubul
255 We propose that the Western lifestyle and diet promote i
256 We propose that the whole HoxD cluster is a dynamic TAD
257 We propose that these components may be associated with
258 We propose that these contigs correspond to linear singl
259 We propose that these differences are mediated by shifts
260 We propose that these insights will aid in designing int
261 We propose that these neurophysiological findings may re
262 ed with the trend of Pu concentration; thus,
we propose that these nitrogen and carboxyl functionalit
263 Here,
we propose that these phenomena are due, in part, to the
264 We propose that these topological idiosyncrasies are est
265 optosis are dependent on caspase activation,
we propose that they are forms of programmed necrosis.
266 tion band of TiO2 on an ms-s time scale, and
we propose that they lead to further reduction of the Re
267 We propose that this "fundamental" limit can be overcome
268 We propose that this architecture may allow for precise
269 mpartmental model of spine calcium dynamics,
we propose that this biased distribution in calcium sign
270 We propose that this bidirectional communication promote
271 We propose that this complex interplay amplifies signali
272 gical evidence and recent experimental data,
we propose that this concept should also include metabol
273 We propose that this cross-frequency interaction mechani
274 We propose that this differential success of cell type i
275 We propose that this distinction may be useful if situat
276 We propose that this effect is responsible for the hyper
277 We propose that this effort is updated every few years t
278 We propose that this field reflects the mechanical cross
279 On the basis of ITC results,
we propose that this initial prerequisite binding of MVA
280 -mediated signaling at the EC interface, and
we propose that this is a mechanism for limiting the phl
281 We propose that this is an entatic state imposed by the
282 We propose that this is at least in part due to large ga
283 We propose that this local processing enables the dense
284 We propose that this mechanism integrating BCR, TLR9, an
285 We propose that this process of information transfer is
286 We propose that this regulation constitutes a global 'si
287 We propose that this selectivity is the result of a key
288 is by standing on the shoulders of giants."
We propose that this sentiment is a powerful motivation
289 can act as a redox switch for activity, and
we propose that this switch can provide a rapid and reve
290 We propose that this trade-off, consistent with cost-ben
291 We propose that three landmarks of pre-existing tissue a
292 al and mutagenesis studies of MhsT and LeuT,
we propose that TM5i plays a key role in Na2 binding and
293 On the basis of these findings,
we propose that TRIP8b competes with a portion of the cA
294 We propose that trust in one's community-which, unlike g
295 We propose that ubiquinitation of RNAPII is induced by R
296 We propose that under normal conditions, insulin decreas
297 We propose that vimentin's role in cell motility is to g
298 We propose that when CFA arrives at the airway, it rapid
299 We propose that women with this BVAB1-dominated subtype
300 We propose that ZapA and ZauP promote efficient cytokine