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1 We provide evidence that 6mA occurs mostly in the AT motif of

2 We provide evidence that differences in the function of CpG-A

3 We provide evidence that Dnd1 protein, expression of which is

4 We provide evidence that hydrogen(s) present at C10 of AA pla

5 We provide evidence that increased AD risk may be at least pa

6 We provide evidence that maternal immune activation hits a ke

7 We provide evidence that miR-29a and miR-29c are increased in

8 We provide evidence that once technological macaques reach a

9 We provide evidence that our method can identify conformation

10 We provide evidence that PrMPK9-1 has a cell type-specific fu

11 We provide evidence that RIM1alpha and RIM1beta are highly li

12 We provide evidence that salivary exosomes from mice with PDA

13 We provide evidence that the HAstV capsid spike is a receptor

14 We provide evidence that the risk-associated SNPs are associa

15 We provide evidence that these anomalies are primarily due to

16 We provide evidence that this is associated with the suppress

17 We provide evidence that TMEM18 has four, not three, transmem

18 irement maps to IL-4Ralpha expression by stromal cells, and we provide evidence that it regulates thymic exit via a proce

19 Here, using luciferase-based reporter assays, we provide evidence that NRP1 regulates HH signaling specific

20 cylglycerol acyltransferase (DGAT)-dependent LD biogenesis, we provide evidence that LDs are dispensable for ERAD in mamm

21 g our results with expression quantitative trait loci data, we provide evidence that variation at these regulatory region

22 Finally, we provide evidence that Cos2 is also sumoylated, which count

23 Finally, we provide evidence that group 2 innate lymphoid cells are a

24 Finally, we provide evidence that overexpression of miR-183, the human

25 Finally, we provide evidence that TET2 colocalizes with EBNA2-EBF1-RBP

26 Finally, we provide evidence that this can be explained by differences

27 Further, we provide evidence that genes facilitating fetal development

28 Furthermore, we provide evidence that the disordered interdomain linker mo

29 Furthermore, we provide evidence that the L1 protein interacts directly wi

30 Here we provide evidence that cells from hepatocellular carcinoma

31 Here we provide evidence that tenascin-C (TNC), an extracellular m

32 Here we provide evidence that VE-cadherin is cleaved by calpain up

33 Here, we provide evidence that glycosphingolipids play an important

34 Here, we provide evidence that hypoxia causes economic impacts on a

35 Here, we provide evidence that intraflagellar transport protein 20

36 Here, we provide evidence that Mlh1-Mlh3 does not behave like a str

37 Here, we provide evidence that not all evolutionary lineages of ECM

38 Here, we provide evidence that proteolytic cleavage of the macropha

39 Here, we provide evidence that rod length is limited by the width o

40 Here, we provide evidence that the right temporoparietal junction (

41 Here, we provide evidence that, contrary to the time-dependent pore

42 and mouse transcriptome analyses in syngeneic mouse models, we provide evidence that tumour-infiltrated mMDSCs facilitate

43 Moreover, we provide evidence that a nuclear pore complex associates wi

44 In this paper we provide evidence that a general model in the network scien

45 In the late study phase, we provide evidence that MARV can be horizontally transmitted

46 In this report, we provide evidence that antibody binding to a PorB epitope c

47 In the current study, we provide evidence that infection of mice with either lympho

48 In this study, we provide evidence that efficacious responses to this drug c

49 In support of this, we provide evidence that the treatment is safe and effective

50 Together, we provide evidence that senescence and a stem cell-associate

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