1 Here,
we provide evidence for a novel role of CTRP6 in modulating b
2 In this article,
we provide evidence for a specific and exclusive role of the
3 We provide evidence for a whole-genome triplication event spe
4 At molecular level
we provide evidence for non-canonical mode of action of Wnt5a
5 In summary,
we provide evidence for the existence of a sortable populatio
6 We provide evidence for the formation of uniquely stable tetr
7 In this article,
we provide evidence for the functional roles of Pn3P-specific
8 Here
we provide evidence for the usefulness of a Drosophila larva
9 In this study,
we provide evidence from cytometry by time-of-flight analysis
10 Here
we provide evidence from two different foraging tasks that ne
11 e, using a combination of techniques from cell to behavior,
we provide evidence of a new glutamatergic pathway activating
12 Here
we provide evidence of a polarization of both peak load and o
13 Furthermore,
we provide evidence of bilateral CSD recorded by fMRI during
14 ics, single-cell genomics, and metatranscriptomic analyses,
we provide evidence of metabolic diversification of enteric m
15 Here,
we provide evidence of tumor suppressive activity of PPARD in
16 We provide evidence showing asymmetric subgenome domesticatio
17 Here,
we provide evidence suggested that Netrin-1 has a critical ro
18 We provide evidence suggesting that LRRK2 G2019S and SYNJ1 lo
19 Here
we provide evidence supporting TDP-43 acetylation as a trigge
20 We provide evidence that 6mA occurs mostly in the AT motif of
21 In this paper
we provide evidence that a general model in the network scien
22 Here
we provide evidence that cells from hepatocellular carcinoma
23 We provide evidence that differences in the function of CpG-A
24 We provide evidence that Dnd1 protein, expression of which is
25 In this study,
we provide evidence that efficacious responses to this drug c
26 Here,
we provide evidence that glycosphingolipids play an important
27 Finally,
we provide evidence that group 2 innate lymphoid cells are a
28 Here,
we provide evidence that hypoxia causes economic impacts on a
29 We provide evidence that increased AD risk may be at least pa
30 In the current study,
we provide evidence that infection of mice with either lympho
31 irement maps to IL-4Ralpha expression by stromal cells, and
we provide evidence that it regulates thymic exit via a proce
32 In the late study phase,
we provide evidence that MARV can be horizontally transmitted
33 We provide evidence that maternal immune activation hits a ke
34 We provide evidence that miR-29a and miR-29c are increased in
35 Here,
we provide evidence that not all evolutionary lineages of ECM
36 We provide evidence that once technological macaques reach a
37 We provide evidence that our method can identify conformation
38 Finally,
we provide evidence that overexpression of miR-183, the human
39 We provide evidence that RIM1alpha and RIM1beta are highly li
40 Here,
we provide evidence that rod length is limited by the width o
41 We provide evidence that salivary exosomes from mice with PDA
42 Here
we provide evidence that tenascin-C (TNC), an extracellular m
43 Furthermore,
we provide evidence that the disordered interdomain linker mo
44 We provide evidence that the HAstV capsid spike is a receptor
45 Furthermore,
we provide evidence that the L1 protein interacts directly wi
46 We provide evidence that the risk-associated SNPs are associa
47 g our results with expression quantitative trait loci data,
we provide evidence that variation at these regulatory region
48 Here
we provide evidence that VE-cadherin is cleaved by calpain up
49 Here,
we provide evidence that, contrary to the time-dependent pore
50 We provide evidence to firmly associate KMT2C, ASH1L, and KMT