1 We searched for 5'-UTRs that are folded at low temperatu
2 lor exhibited in our CHO-derived antibodies,
we searched for 8 different oxidation and 28 different g
3 l significance when considered in isolation,
we search for a close approximation to the quantitative
4 In this graph,
we search for a motif as the maximum density subgraph, w
5 Next,
we search for a set of SGA events that carries strong in
6 Then, for each transcriptomic module,
we search for a set of SGA genes (driver modules) such t
7 More specifically, we demonstrate that, when
we search for a single solution in a database having 409
8 When
we search for a target in a crowded visual scene, we oft
9 ries of attention rely on the idea that when
we search for a target in a visual display the brain boo
10 We searched for a candidate segment that supported the c
11 We searched for a convergent evolutionary signal in the
12 When
we searched for a family molecule(s) for TMEPAI, we foun
13 We searched for a gene on chromosome 17p13 that contribu
14 Here
we searched for a role of this protein in spine morphoge
15 esin-1 tail are required for autoinhibition,
we searched for a second molecule that contributes to ac
16 in-2 lacks an intracellular signaling motif,
we searched for a signal adaptor that permits it to tran
17 Initially,
we searched for a ZEBRA mutant that supports viral repli
18 We searched for Abeta-hydrolyzing human IgV domains (IgV
19 We searched for abnormalities of the proapoptotic Bcl-x(
20 In this paper
we search for additional targets of Smicl.
21 Stimulated by these observations,
we searched for additional anatomical structures that ex
22 In this study,
we searched for additional factors that contribute to sp
23 hogenic mechanism of RAGE in these diseases,
we searched for additional ligands.
24 the function of myospryn in striated muscle,
we searched for additional myospryn paralogs.
25 We searched for additional recurrent focal CNAs using th
26 In this study,
we searched for additional sites in 2C(ATPase), near N25
27 We searched for all clinical trials undertaken in Blanty
28 We searched for all controlled trials of mobile technolo
29 We searched for all controlled trials of mobile technolo
30 using a coarse-grained computational model,
we searched for all possible simple networks that can ac
31 better insight into TBK1 survival signaling,
we searched for altered phosphoproteins using mass spect
32 We searched for alternative signal 3 receptor pathways a
33 We search for and evaluate associations between all pres
34 to explain the strong binding of RLF to LGR8
we searched for and found an extended region where littl
35 In this study,
we searched for and identified two putative excitatory p
36 We searched for any clinical study assessing cardiopulmo
37 We searched for any cohort study, case-control study, or
38 died the nucleolytic properties of PALF, and
we searched for any modulation of PALF by NHEJ component
39 We searched for articles from PubMed, Embase, Cochrane,
40 We searched for articles published between 1980 and Apri
41 We searched for articles published from Jan 1, 1998, to
42 To further corroborate these findings,
we searched for association of single nucleotide polymor
43 We searched for associations between eight growth and wo
44 In this study,
we searched for associations between polymorphic TE (pol
45 et insights about ATRA regulation of miRNAs,
we searched for ATRA-modulated transcription factors bin
46 In this study,
we searched for autoantibodies related to narcolepsy usi
47 Therefore, in the present study,
we searched for AZD8055-based combination therapies.
48 ineate the cause of the impaired locomotion,
we searched for binding partners to the N-terminal uniqu
49 r dendritic spikes in CA1 pyramidal neurons,
we searched for biologically-plausible long-term potenti
50 of flight mass spectrometry (SELDI-TOF MS),
we searched for biomarkers of PAH in plasma specimens fr
51 We searched for BMP6 mutations in relatives of 5 proband
52 Using fMRI and multivoxel pattern analysis,
we searched for brain regions that encode temporally int
53 We searched for candidate genes using three different ap
54 For this systematic review,
we searched for case-control and cohort studies publishe
55 We searched for cell proteins that interact with ORF29p
56 transcriptional regulator in keratinocytes,
we searched for cell-cycle-related genes that could illu
57 We searched for cell-surface-associated proteins overexp
58 We searched for cells in the neonatal rat testis with th
59 In our attempt to study macaque AMCase,
we searched for CHIA-like genes in human and macaque gen
60 hese genes are direct targets of ChREBP.Mlx,
we searched for ChoRE-like sequences in the 5'-flanking
61 We searched for clinical studies in MEDLINE, EMBASE, and
62 Here,
we search for CNEs among the ancestral repeat classes in
63 We searched for CNVs affecting exons of PARK2 in a sampl
64 he lymphangiogenic growth factor Vegfc gene,
we searched for collagen- and calcium-binding epidermal
65 alence of peripheral artery disease in which
we searched for community-based studies since 1997 that
66 We searched for comorbid phenotypes with motion sickness
67 To address these challenges
we searched for compounds that affect feeding behavior i
68 Using the GDSC databases,
we searched for compounds with high selectivity for ccRC
69 We searched for conserved features of the polymerase dom
70 the genetic alterations in this tumor type,
we searched for copy number alterations using high-densi
71 We searched for copy number variants that were increased
72 We searched for correlates of protection using a viral s
73 ochastic, age-stratified transmission model,
we searched for cost-effective booster vaccination strat
74 We searched for CPGs and CS on the use of infliximab, et
75 We searched for data on IDU prevalence, characteristics
76 We searched for de novo mutations in a family quartet wi
77 Here
we search for defective gene family interaction networks
78 onal overexpression of MDM2 in cancer cells,
we searched for deletion or substitution mutation in the
79 We searched for deletions in the germ-line genome among
80 We searched for determinants of Na(+) self-inhibition by
81 Here, in an attempt to crystallize the HBD,
we search for DGCR8 homologues and show that DGCR8 from
82 s as follows: using closed-loop experiments,
we searched for different stimulus patterns in the surro
83 We searched for DISC1 transcripts in adult and fetal hum
84 We searched for disruptive, genic rare copy-number varia
85 We searched for double-blind, placebo-controlled randomi
86 against other steps in the viral life cycle,
we searched for drugs active against the EBV SM protein,
87 Dyrk1A binds tightly with cell constituents,
we searched for Dyrk1A binding proteins in the Triton X-
88 We searched for Earth-size planets that cross in front o
89 Using a proteomics approach,
we searched for endogenous alphaT3-1 proteins that parti
90 antigen for eliciting 2G12-like antibodies,
we searched for endogenous yeast proteins that could bin
91 We searched for English-language and non-English-languag
92 To gain insight into GDU1's role,
we searched for ethyl-methanesulfonate suppressor mutant
93 We search for evidence of such effective forces by study
94 Next,
we search for evidence of toxicological relationships be
95 We searched for evidence of functional elements in the c
96 In our study,
we searched for evidence of selection that might have fo
97 ad been documented only at transcript level,
we searched for evidence that would also support the tra
98 We searched for factors on participants from survey year
99 We searched for factors that could regulate neural tempo
100 We searched for FCTA publications and reviewed them for
101 a series of co-expression networks, in which
we search for frequently occurring network patterns.
102 In this study,
we searched for FRGY2a-interacting proteins and investig
103 Using protein affinity purification, here
we search for functional partners of TET proteins, and f
104 Using computational methods,
we searched for G-Quadruplex Sequence (GQS) patterns in
105 efit of Gecko 3 and to exemplify our method,
we search for gene clusters in a dataset of 678 bacteria
106 To improve upon these analyses,
we searched for gene colinearity (microsynteny) between
107 We searched for gene dosage suppressors of top3 and iden
108 We searched for gene expression patterns that correlate
109 We search for genes that are recurrently affected by rar
110 embedded within the Escherichia coli genome,
we searched for genes capable of restoring growth of a T
111 regulates upper lip and palate morphogenesis
we searched for genes downregulated in response to RARga
112 ncing in a cohort of 30 individuals with KS,
we searched for genes newly associated with KS.
113 We searched for genes that are required for the survival
114 We searched for genes that fit several criteria establis
115 We searched for genes that have the potential to be ther
116 upon cell cycle regulation becomes clearer,
we searched for genes that might specify such control in
117 We searched for genes that regulate mitochondrial Ca2+ a
118 In this study,
we searched for genes that were distinctly expressed at
119 We searched for genes whose expression levels correlate
120 We searched for genetic factors that might cause this di
121 We searched for genetic predictors of treatment outcome
122 oma clustering is observed in some families,
we searched for genetic predisposing factors.
123 We searched for genetic variants associated with CIPO to
124 ere positive for microsatellite instability,
we searched for germ-line mutations in the MLH1, MSH2, M
125 signaling, regulates Hh pathway activation,
we searched for GLI1-interacting proteins.
126 expression profiling and the Dam-ID method,
we searched for Hb9-regulated genes, uncovering transcri
127 the metabolism of this heme-free eukaryote,
we searched for heme-containing proteins in its de novo
128 We searched for heterogeneity and interactions using a c
129 c pathways were encompassed by the data set,
we searched for homologs of the enzymes leading from gly
130 Then,
we searched for homozygous deletions and amplifications
131 and a genome-wide association study design,
we searched for human genetic determinants of plasma lev
132 To test this hypothesis,
we searched for IgG anti-FIXa Abs in APS patients.
133 derstand miRNA function in Treg development,
we searched for important miRNAs and their relevant targ
134 We search for influential spreaders by following the rea
135 tand the functional process involving EndoQ,
we searched for interacting partners of EndoQ and identi
136 To understand the function of NPX1,
we searched for interacting proteins and found that an A
137 of protein synthesis with cycloheximide, so
we searched for interacting proteins that might affect p
138 a better understanding of ALDP dysfunction,
we searched for interaction partners of ALDP and identif
139 Here,
we searched for intracellular Ca-carbonate inclusions in
140 the molecular mechanisms of PLA2R function,
we searched for its endogenous binding partners.
141 herefore, to better elucidate PATZ function,
we searched for its molecular partners.
142 -CcP to use alternative reducing substrates,
we searched for its subcellular localization in the infe
143 We searched for listeriosis case series and outbreak inv
144 Finally,
we search for local adaptation between geographically cl
145 To this extent,
we searched for markers that are elevated in melanoma an
146 y and history of this essential gene family,
we searched for maturase homologs in recently sequenced
147 We searched for members of the elav/hu family in the cut
148 We searched for microRNAs (miRs) that are affected by hi
149 e the molecular mechanisms mediated by Mili,
we searched for Mili-interacting proteins.
150 t transcriptionally silence gene expression,
we searched for miRNA target sites proximal to known gen
151 iRNAs) in modulation of cellular phenotypes,
we searched for miRNAs regulated during the degenerative
152 iRNAs) in modulation of cellular phenotypes,
we searched for miRNAs that were regulated during cardia
153 omplex developmental and disease phenotypes,
we searched for miRNAs that were regulated during the po
154 ate how cancer drivers genetically interact,
we searched for modifiers of epidermal growth factor rec
155 gnificance of this proteolytic modification,
we searched for molecular interacting partners of proteo
156 Using in vivo phage display,
we searched for molecular markers of the neurovascular u
157 We searched for morphofunctional abnormalities of the mi
158 sms underlying this differentiation process,
we searched for muscle specific-binding partners of paxi
159 We searched for mutation(s) associated with S-HSCR by co
160 We searched for mutations in BAG3 by direct sequencing.
161 through coupling splicing to EJC deposition,
we searched for mutations in hCWC22 that affect eIF4AIII
162 Here
we search for naturally occurring genetic interactions i
163 Here,
we search for neural activity compatible with the violat
164 Here,
we search for neural correlates of this property in the
165 To demonstrate the utility of this approach,
we search for neurocognitive and neuroanatomic endopheno
166 How do
we search for new instances of a motif in this sea of DN
167 Using this temporal transformation
we search for new interacting pairs.
168 e EFA6/Arf6 function in vesicular transport,
we searched for new EFA6 partners.
169 We searched for new evidence using PubMed from 2005 to S
170 We searched for new markers of neovasculature in PDAC an
171 We searched for new mutations and sought to define the f
172 We searched for new prions of yeast by fusing random seg
173 insight into IRF1's role in these processes,
we searched for new target genes by performing chromatin
174 ractive events must be taken into account as
we search for newer, more effective therapeutic interven
175 As a strategy to improve clinical efficacy,
we searched for novel agents capable of potentiating the
176 Using a yeast two-hybrid screen,
we searched for novel AIRE-interacting proteins and iden
177 We searched for novel associations between height and co
178 Therefore, in the present study,
we searched for novel binding receptors for the soluble
179 in a deeper understanding of the role of L2,
we searched for novel cellular L2-interacting proteins.
180 bation of cells with Listeria monocytogenes,
we searched for novel host proteins undergoing tyrosine
181 approach of 447 fasting plasma metabolites,
we searched for novel molecular markers that arise befor
182 We searched for Nras, Kras, and Ptpn11 point mutations i
183 To identify novel NTS2 selective analgesics,
we searched for NTS2 selective nonpeptide compounds usin
184 In addition to URE deletion
we searched for other mechanisms that in the absence of
185 We searched for overlapping BH and aPKC phosphorylation
186 sphorylated tau (P-tau) in cultured neurons,
we searched for P-tau by immunohistochemistry.
187 Using an integrated whole-genome approach,
we searched for P. aeruginosa virulence genes with multi
188 neurochemical and behavioral phenotype, here
we searched for pathways that may mediate lithium's/the
189 rt study in 173 German intensive care units,
we searched for patients with and without asplenia and c
190 address possible mechanisms of PDI effects,
we searched for PDI interactome by systems biology analy
191 In this systematic review
we searched for peer-reviewed and grey literature public
192 approaches to study plant drought tolerance,
we searched for phenotypes conferred by drought stress a
193 both positive and negative control studies,
we searched for pleiotropic influences on comorbid subst
194 We searched for policies electronically with the search
195 We searched for polymorphism at NPY1R by systematic rese
196 We searched for polymorphisms in or near COMMD1 that wer
197 Subsequently,
we searched for possible underlying mechanisms using RV
198 and performed two independent analyses: (i)
We searched for potential compensatory mutations spatial
199 l renal tissue might protect against damage,
we searched for potential influences of the female hormo
200 Using subtractive hybridization,
we searched for previously undetected genes in diverse c
201 Using an oligonucleotide microarray,
we searched for previously unrecognized transcription un
202 onal knowledge about the LecRLK gene family,
we searched for previously unreported motifs and checked
203 We searched for prospective cohort studies reporting ass
204 onal response of stem cells to HH signaling,
we searched for proteins binding to GLI proteins, the tr
205 sing transcriptomic and proteomic profiling,
we searched for proteins in mouse brain endothelial cell
206 rsal-ventral and nuclear morphology defects,
we searched for proteins that interact with Glorund.
207 Here,
we searched for proteins that interact with MCOLN1 in a
208 ns form the backbone of signal transduction,
we searched for proteins that interact with the catalyti
209 Using this methodology,
we searched for proteins which are specifically deregula
210 We searched for psychiatric surveys that were based on i
211 In this systematic review,
we searched for published and unpublished double-blind r
212 We searched for published systematic reviews and new ref
213 We searched for randomised controlled trials (RCT) asses
214 systematic review and network meta-analysis,
we searched for randomised controlled trials of interven
215 We searched for randomised trials of interventions to pr
216 We searched for randomised trials of intravenous rt-PA v
217 We searched for randomized control trials (RCTs) and obs
218 We searched for randomized controlled trials (RCTs) inve
219 We searched for randomized controlled trials and observa
220 METHODS AND
We searched for randomized trials that compared longer t
221 ome sequence data and an imputation approach
we searched for rare sequence variants in CHRNA4 and tes
222 In the present study,
we searched for rare variants within or near the DIRAS2
223 Plasmodium RBPs during parasite replication,
we searched for RBPs that might play a role during liver
224 riation in congenital heart diseases (CHDs),
we searched for regulatory mutations impacting the activ
225 We searched for related studies published in PubMed, Web
226 Moreover
we searched for relationships between serum FGF19 and la
227 We searched for relationships in the expression of Wnt p
228 In this review,
we searched for relevant studies published in MEDLINE (P
229 We search for replacements by applying screening criteri
230 We searched for reports of trial results in Ovid Medline
231 We searched for reviews of physical activity interventio
232 For each transcript,
we searched for secondary loci interacting with primary
233 proven difficult to target therapeutically,
we searched for secreted, druggable proteins induced by
234 N/dS-based test in a phylogenetic framework,
we searched for selection footprints on LSE and single-c
235 co-regulation within co-expression clusters,
we searched for shared cis-regulatory motifs in putative
236 Here,
we searched for signatures of ionizing radiation in 12 r
237 Finally,
we searched for signatures of post-admixture selection a
238 Here,
we searched for similar genes in a second major clade of
239 Here,
we searched for similar genes in species that retained c
240 Here,
we searched for similar genes in the Laurasiatheria clad
241 We searched for similar genes within the suborder Rumina
242 th little prion protein (PrP), and therefore
we searched for similar virus-like particles in situ in
243 We searched for single nucleotide polymorphisms (SNPs) r
244 We searched for single-nucleotide polymorphisms (SNPs) t
245 teria, including Mycobacterium tuberculosis,
we searched for small molecules to augment the IFNgamma
246 We searched for splicing regulators interacting with thi
247 In this study,
we searched for ssDNA in mutating V regions in which DNA
248 We searched for studies in any language in which adult i
249 We searched for studies in MEDLINE and EMBASE and classi
250 We searched for studies in Medline, Embase, and CINAHL i
251 We searched for studies in PubMed and Embase and confere
252 In this systematic review and meta-analysis,
we searched for studies in which SD-OCT was used to look
253 In this systematic review and meta-analysis,
we searched for studies investigating the risk of venous
254 We searched for studies of ICU patients with recorded se
255 We searched for studies published between Jan 1, 1955, a
256 We searched for studies published since 2000 through ele
257 We searched for studies published up to Jan 6, 2015, tha
258 We searched for studies that compared LV volumes and EF
259 We searched for studies using positron emission tomograp
260 We searched for studies, reporting patient and family da
261 We searched for substrates encoded in the ameba genome a
262 Here,
we search for such developmental signals by combining bi
263 ht induce c-myc to Ig V-J(H) translocations,
we searched for such events in both interleukin (IL) 6 t
264 We searched for such factors using newly developed cultu
265 We searched for such proteins by yeast two-hybrid assay,
266 to specific assaults in a selective manner,
we searched for such vulnerabilities in CML LSCs.
267 We searched for superconductivity in weakly interacting,
268 uired for this crucial step in pathogenesis,
we searched for surface-exposed, cell wall-anchored prot
269 uire dorso-medial prefrontal cortex (dmPFC),
we searched for synaptic adaptations in dmPFC resulting
270 We searched for systematic reviews on the effectiveness
271 We searched for T-UCRs that regulate growth of the intes
272 ssays to define Spt- and Ntl-binding motifs,
we searched for target regulatory sequence via a combina
273 Therefore,
we searched for targets of the AR that may contribute to
274 We searched for tau in the conditioned medium of N2a cel
275 We searched for terms related to severe sepsis and septi
276 in exploiting this process in photovoltaics,
we search for the direct signature of singlet fission, p
277 each high-frequency allele by homozygosity,
we search for the expected decay of adjacent SNP linkage
278 Moreover,
we searched for the CacyBP/SIP domain important for dime
279 Here,
we searched for the functional domains within Ac45 by an
280 We searched for the genetic defect in PRRT2-negative, un
281 Since its location is not known,
we searched for the Hv permeation pathway.
282 r gene, GT198 (PSMC3IP), as a unique marker,
we searched for the identity of GT198 mutant cells in ov
283 We searched for the model that included the items and th
284 Using a simulated annealing (SA) procedure,
we searched for the most informative combination and the
285 We searched for the primary defect in 3 children from 2
286 We search for "
themes"-segments of at least 35 residues
287 We searched for these by tandem mass spectrometry in pla
288 We search for third-order interactions, discovering an i
289 We searched for this particular state at later sites of
290 We searched for time representations in cortico-basal ga
291 ng an assumption about size or conformation,
we searched for toxic forms of recombinant PrP after dil
292 Here,
we search for trans-factors and identify Yy1 as a requir
293 d in the S-box regulatory mechanism in vivo,
we searched for trans-acting mutations in Bacillus subti
294 We search for transcription factors (TFs) that control P
295 expression compared with CD4- NKT cells, and
we searched for transcription factors that are up-regula
296 First,
we search for transcriptomic modules with genes that are
297 Here
we search for unique features in the synaptic outputs ma
298 In this study,
we searched for unique functional properties of IFN-lamb
299 s in 40 additional dermatomyositis patients,
we searched for vascular deposits of IgG, IgM, and the C
300 Here
we search for violation of Lorentz symmetry for electron