1 We set out to address this issue by studying how various mRNA
2 We set out to address this problem by generating multiple gen
3 We set out to address whether neglect presentation could be m
4 tial to directly recognize microbial or endogenous signals,
we set out to assess whether their functions are locally infl
5 We set out to bridge this gap.
6 Herein
we set out to characterize the carbohydrate minimal binding e
7 To understand its apparent biological activity,
we set out to chemically synthesize d-allo-ShK and determine
8 We set out to compare the incidence of bowel repair and/or re
9 We set out to define the role of 1alpha,25-dihydroxy vitamin
10 We set out to define the role of UL31 in CMV replication.
11 Here,
we set out to delineate the protein degradation mechanism of
12 In this study,
we set out to determine if the same is true for B cells using
13 on genomics of var genes in cases of uncomplicated malaria,
we set out to determine if there was any evidence of a select
14 Here,
we set out to determine the extent to which dynamic changes t
15 Here
we set out to determine the mechanisms underpinning variant a
16 We set out to determine the molecular mechanisms underlying t
17 We set out to determine the specific AMPAR subunit required f
18 Here,
we set out to determine whether changes in rate constants res
19 Here
we set out to determine whether oscillatory activity in the b
20 prebiotics increases iron absorption in infants is unclear.
We set out to determine whether prebiotic consumption affects
21 We set out to determine whether this important mechanical com
22 sphorylated by cellular protein kinases, and in this study,
we set out to determine whether this modification is required
23 We set out to develop a method to overcome this obstacle by u
24 As both are heritable,
we set out to examine whether there is a genetic correlation
25 Here,
we set out to explore dynamic aspects of twister RNA folding
26 In this study,
we set out to further understand CD27 as an immunomodulatory
27 Here,
we set out to gain further insights into the function of SMAR
28 We set out to identify and phenotype allergen-responsive cell
29 We set out to identify geographical characteristics that cons
30 Here
we set out to identify mechanisms of specificity in protein p
31 We set out to identify novel and functionally important endot
32 ancer drugs or compounds currently in clinical development,
we set out to identify novel effective treatments for T-PLL p
33 rther insights into the regulation of Glut4 palmitoylation,
we set out to identify the palmitoyl acyltransferase (PAT) in
34 Here,
we set out to investigate effects of UV-A and solar-simulated
35 We set out to investigate how PGE2 regulates human ILC2 funct
36 We set out to investigate if the failure of cultured mesenchy
37 To this end,
we set out to investigate the metabolic effects of NO in cult
38 Here,
we set out to investigate whether Galphai-GIV is a druggable
39 Here
we set out to learn more about the underlying mechanism by wh
40 Therefore,
we set out to map H2-IA(b)-restricted epitopes along the PVM
41 ince receptors are important determinants of viral tropism,
we set out to map the CV-A24 receptor repertoire and establis
42 Here,
we set out to quantify Mcp5 organization and identify the bin
43 We set out to study the role of MOF in general hematopoiesis
44 As a basis for studies of antimalarial efficacy,
we set out to survey parasite carriage in 3 communities in No
45 We set out to systematically identify these subsets in human
46 We set out to take a different approach and mimic the synthes
47 ct is often a prerequisite for ExPEC-mediated pathogenesis,
we set out to understand how ExPEC colonizes this niche.
48 Here,
we set out to understand the extent to which shifts in genome
49 We set out to understand the functions of the ATXN1-CIC compl
50 on data from patients diagnosed between 1985 and 2005, and
we set out to update it by incorporating more recently report