1 We sought to identify a circulating transcriptomic profile pr
2 We sought to identify a novel host target as a potential anti
3 or the treatment of central nervous system (CNS) disorders,
we sought to identify a positron emission tomography (PET) li
4 We sought to identify all sudden cardiac arrests that occurre
5 In this study,
we sought to identify and characterize the cardiac lincRNA tr
6 We sought to identify autoallergic targets of IgE in patients
7 We sought to identify biological pathways in ARDS that differ
8 We sought to identify characteristics that predicted higher r
9 In this study,
we sought to identify common and rare psoriasis-associated ge
10 We sought to identify cytokines that can promote proliferatio
11 t are potentially involved in T1D with cardiac dysfunction,
we sought to identify differentially expressed genes in the T
12 We sought to identify DNA methylation and gene expression cha
13 We sought to identify ERV progenitors, recombinational hot sp
14 We sought to identify gene profiles associated with adult-ons
15 We sought to identify genetic causes of early onset epileptic
16 We sought to identify genetic determinants of early postnatal
17 We sought to identify genetic markers of ICS response by cond
18 We sought to identify global proteomic changes downstream of
19 We sought to identify horizontally transferred genes within a
20 Here,
we sought to identify IFN-lambda-responsive cells by generati
21 our efforts to generate a new medicine for liver fibrosis,
we sought to identify improved small molecule functional mime
22 n the rising prevalence of NAFLD in the general population,
we sought to identify its prevalence and potential impact in
23 We sought to identify key transcriptional regulators of corti
24 Here,
we sought to identify lipid antigens in cow's milk, a promine
25 We sought to identify low-frequency genetic variants that str
26 Here,
we sought to identify neurophysiological signatures of such p
27 ase 1 (PP1c) is dictated by PP1c-interacting proteins, here
we sought to identify new PP1c interactors.
28 Here,
we sought to identify novel mechanisms involved in SCLC chemo
29 We sought to identify p53-independent CCAR2 functions in SCC
30 Here,
we sought to identify possible feedback regulation of VEGFR2
31 To more fully explore the biology of PDE2A inhibition,
we sought to identify potent PDE2A inhibitors with improved b
32 Using meta-analysis,
we sought to identify predictors of pharmacotherapy to furthe
33 We sought to identify pregnancy-related risk factors for acut
34 We sought to identify reliable systematic reviews to assist t
35 We sought to identify risk factors for pediatric extubation f
36 We sought to identify risk factors for suicide completions am
37 We sought to identify risk factors that might explain the inc
38 identify the mechanism by which ChAT activity is increased,
we sought to identify the biological target of stolonidiol.
39 Finally,
we sought to identify the brain regions through which ADX7144
40 We sought to identify the cause of disease in 3 unrelated Fin
41 Here
we sought to identify the galectin-3-binding molecule(s) on t
42 We sought to identify the genetic defect in these patients an
43 We sought to identify the genetic pathogenesis in this family
44 We sought to identify the MB signaling pathway of formoterol
45 We sought to identify the mechanisms involved in activation o
46 We sought to identify the optimal dosing of VOR for effective
47 We sought to identify the prevalence of tibial artery and ped
48 Here,
we sought to identify the RhoGEFs involved in monocyte chemot
49 We sought to identify the underlying mechanisms by which neon
50 idual variability in the MFO requires further clarification.
We sought to identify whether recent, self-selected dietary i