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1 We speculated that a carbohydrate-reduced, high-protein (CRHP
2 We speculated that altering the selective pressure on mutant
3 We speculated that binding of lymphoma BCRs to self-antigens
4 We speculated that CN(-) and the CO precursor (-):CO2H may be
5 We speculated that common antigens shared by these viruses ex
6 We speculated that detection of proteolytic fragments release
7 We speculated that ERK inhibition might thus enhance PDAC dep
8 We speculated that hyperglycemia inhibits skin DC migration t
9 We speculated that indels within 5' untranslated regions woul
10 We speculated that it could be an effect of their participati
11 We speculated that loss of GABA signalling from AgRP-expressi
12 We speculated that low-frequency oscillations observed in the
13 We speculated that replacing the native viral promoter that d
14 We speculated that selective attenuation of PEPD-enhanced res
15 We speculated that serum pantetheinase might contribute to er
16 We speculated that the complex functions of the abnormal LysR
17 We speculated that the contribution of skeletal muscle adapta
18 We speculated that the invasion of L. trifolii likely occurre
19 We speculated that the natural beta-glycoform of antithrombin
20 We speculated that the underlying cause of portal vein thromb
21 We speculated that this plays important roles in the structur
22 We speculated that: (i) young children could respond randomly
25 anges in coding genes during dedifferentiation of PHHs, and we speculated that these modulations could drive the hepatic
27 on of reactive oxygen species and DNA double-strand breaks, we speculated that DNA damage might also constrain leukaemic
28 is pathway and malaria patients have low blood cholesterol, we speculated that it scavenges cholesterol from them in some
29 Considering that metformin can reduce glucose consumption, we speculated that metformin would enhance the anti-neoplasia
30 Given their inherent gross structural chromosomal damage, we speculated that they may arise from chromothripsis, a rece
31 Since complex OM is often a polymicrobial disease, we speculated that reduction of complex OM by PCVs would be a
34 from inflammasome activation) is typically observed in IRI, we speculated that aPC ameliorates IRI by inhibiting inflamma
35 ance to Ags presented on the same surface as their ligands, we speculated that the B cell siglecs are key players in tole
38 oss-of-heterozygosity, promoter methylation, and mutations, we speculated that an inhibitory transcription factor is regu
39 ->3)-beta-d-GlcNAc-(1-->3)-alpha-d-GlcNAc-(1-->OPO3-->]n ), we speculated that the two predicted CPs (CslA and CslB) work
41 sistent synaptic plasticity in other hippocampal subfields, we speculated that the metabotropic glutamate receptor mGlu5
42 receptor is mediated by heparan sulfate (HS) and syndecans, we speculated that a deranged HS/syndecans regulation impairs
43 on the results of the allergen component specific IgE test, we speculated that the pathogens in this case was group5 alle
47 an independent risk factor for the development of BM, thus we speculated that higher circulating estrogens in young, pre
50 stimuli as well as reduced hippocampal gray matter volume, we speculated that childhood maltreatment results in similar