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4 ermine if and how the cost could be reduced, we subjected plasmid-containing bacteria to 1100 generat
5 usion model and an intraventricular balloon, we subjected hearts to 20 minutes of ischemia followed b
6 andomized, counterbalanced crossover design, we subjected participants to 3 weeks of chronic sleep re
9 ted Langendorff perfused rabbit heart model, we subjected hearts to 8 hours of hypothermic ischemia w
12 yze the behavioral effects of CN deficiency, we subjected CN mutant mice to a comprehensive behaviora
13 is causally related to neointimal formation, we subjected mice lacking Mac-1 to a novel form of mecha
15 onal residues involved in 3-PGA interaction, we subjected P52L LS DNA to a second round of mutagenesi
16 origin of these inter-individual variations, we subjected rats to a differential auditory fear condit
17 ics at a scale relevant for these organisms, we subjected soil microcosms to a heat disturbance and f
20 he development of chronic tissue remodeling, we subjected these graft recipients to a battery of hist
22 expand our understanding of Inc function(s), we subjected putative C. trachomatis Incs to affinity pu
24 nse to decreased parathyroid hormone levels, we subjected osteocytes to an in vitro unloading environ
26 Th17 phenotype in acute glomerulonephritis, we subjected nephritic mice to CD3-specific Ab treatment
28 produce a rapid and extensive demethylation, we subjected mouse ES cells to chemically defined hypome
29 ure overload-induced myocardial hypertrophy, we subjected transgenic mice lacking gp91phox to chronic
32 system approximating the in vivo situation, we subjected mouse hippocampal slice preparations to con
33 We subject the alga Chlamydomonas reinhardtii to conditi
38 sis for the RNA-binding specificity of RRMs, we subjected 330 aligned RRM sequences to covariance ana
39 macologically reduce FoxM1 activity in HCCs, we subjected these HCC-bearing mice to daily injections
41 A homeostasis remains to be defined in vivo We subjected Stra6-knockout mice to diets sufficient and
42 Here we subject this collective guidance hypothesis to direct
50 Finally, we subjected animals with preexisting hypertrophy to HDA
51 O metabolites could modulate cytoprotection, we subjected CS-eNOS-Tg mice to hepatic ischemia-reperfu
53 ntilator-induced lung injury (VILI) in vivo, we subjected 12 anesthetized, paralyzed rabbits to high
54 nt in protection against reperfusion injury, we subjected the flies to hypoxia, followed by recovery
55 was inactivated in these murine MTC samples, we subjected tumor tissue to immunohistochemical stainin
56 in both neural and glioblastoma stem cells, we subjected both cell types to in-vitro differentiation
57 fications relevant in myocardial infarction, we subjected isolated myocardial complex II to in vitro
62 To investigate the role of antigen, we subjected mice with disrupted microbiota to intranasa
65 he potential role of MPO in atherosclerosis, we subjected LDL receptor-deficient mice to lethal irrad
66 ilepsy result in similar structural changes, we subjected rat pups to lithium-pilocarpine-induced sta
68 d resolve individual molecular mass species, we subjected purified calsequestrin to mass spectrometry
69 To define the nature of this response, we subjected Dictyostelium discoideum cells to measurabl
70 We subjected neonatal rat ventricular myocytes to mechan
72 thods and Results-- To test this hypothesis, we subjected isolated, perfused rat hearts to mechanical
73 volatile organic compounds (VOCs) analysis, we subjected the plant to mechanical damage and monitore
76 y of antimycobacterials and drug candidates, we subjected Nos2 (-/-) mice with TB to monotherapy befo
77 carries out its role in coat morphogenesis, we subjected its gene to mutagenesis and studied the eff
80 o understand drivers of plant stoichiometry, we subjected four herbaceous legume species to nine leve
82 distinguish between these two possibilities, we subjected owls to optical conditions that differed in
86 ess temporal variation of olfactory stimuli, we subjected flies to precisely defined odor concentrati
87 ve regions of functional importance in CheZ, we subjected cheZ to random mutagenesis and isolated 107
89 n various steps of CPE action, in this study we subjected the cloned cpe gene to random mutagenesis i
90 ious Cys-scanning analysis of permease XanQ, we subjected YgfU to rationally designed site-directed m
92 the kidney from ischemia-reperfusion injury, we subjected hif1a(+/-) and hif2a(+/-) mice to renal isc
93 ischemic changes that tumor tissues undergo, we subjected harvested xenograft tumors to room temperat
95 50-180 microm) at speeds approaching 10 m/s, we subject the protein to shear rates dv(z)/dr as large
99 the molecular basis of this delayed injury, we subjected rats to spinal cord injury and identified g
101 irectly demonstrate this function for miR-7, we subjected the networks to temperature fluctuation and
102 Here, we subject As(V)-coprecipitated schwertmannite to therma
105 erstanding the genetic determinants of Ref1, we subjected Ref1-positive TE671 cells to three sequenti
106 d in the adult ventricle during hypertrophy, we subjected animals to transverse aortic constriction (
109 assess the generalizability of our findings, we subjected the hypomorphs to unilateral ureteral obstr
110 amin D receptor (VDR) in renal fibrogenesis, we subjected VDR-null mice to unilateral ureteral obstru
112 To address this question, we subjected neurons genetically lacking NR3A to various
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