1 We then ask whether variation in movement based on (i) age, (
2 We then asked whether 17B1.3 prevents signaling by binding to
3 Since glycosylation can enhance the function of a compound,
we then asked whether adding a glucose makes aesculin activat
4 We then asked whether miR-3189 was expressed in clinical samp
5 We then asked whether prolonged PSAP/IntA experience would ne
6 We then asked whether the aberrant tangential migration of GA
7 We then asked whether the replication and the mitotic phenoty
8 We then asked whether the responses in these regions reflecte
9 Using a series of recombinant X genotypes,
we then asked whether X overexpression in hybrids is controll
10 We then assessed whether baseline characteristics moderated t
11 We then assessed whether particularly high levels of signal m
12 We then assessed whether the stability of all neural activity
13 We then determined whether bull trout genetic diversity was r
14 We then determined whether JIP3 activates tetrameric kinesin-
15 We then determined whether machine learning could further imp
16 We then determined whether oocytes synthesized betaine and wh
17 We then determined whether perinatal factors modify these lef
18 We then determined whether pneumonia hospitalization increase
19 ial structure of the Autism Genetic Resource Exchange data,
we then determined whether the mtDNA haplogroups correlate wi
20 We then evaluated whether a treatment could increase the secr
21 We then evaluated whether changes in BVOC emissions can affec
22 We then evaluated whether,
despite the barriers, it is possib
23 We then evaluated whether iron-associated variants were assoc
24 om cardiovascular causes, myocardial infarction, or stroke;
we then evaluated whether random assignment to prompt revascu
25 We then evaluated whether treatment with trastuzumab or chemo
26 We then evaluated whether ultrasound is effective in deliveri
27 We then examined whether the CB2R agonists could attenuate in
28 We then examined whether the properties of the starch synthes
29 We then examined whether the temperature-mortality associatio
30 We then investigated whether endothelial cells differentiated
31 We then investigated whether or not chondroadherin fragmentat
32 We then investigated whether pharmacological or genetic appro
33 We then investigated whether the benefits that facultative ba
34 We then investigated whether the changes observed during veno
35 s residue is just before the gamma-secretase cleavage site,
we then investigated whether the p75(alphagamma) peptide, whi
36 We then studied whether glucocorticoid treatment can be impro
37 We then test whether administering adenosine significantly im
38 We then tested whether centrality in the network of host spec
39 We then tested whether CNO administration would produce behav
40 We then tested whether factors that contribute to this signal
41 We then tested whether heritability was fixed and independent
42 We then tested whether microhabitat use was associated with s
43 We then tested whether our newly generated, stage-specific tr
44 We then tested whether scheduled wheel access could improve d
45 We then tested whether SmbFT can confer protection against ha
46 We then tested whether the combination of the involuntary mov
47 We then tested whether the corticospinal excitability of the
48 We then tested whether the lack of endogenous tau protects ag
49 We then tested whether these predictors were also associated
50 We then tested whether variants showing significant associati