1 We then analyze multiple markers of neurodegeneration and neu
2 We then analyzed gene expression profiles of human breast can
3 We then apply our graph structure beyond clustering, using it
4 We then apply the analytical methods to the anatomical brain
5 We then apply the XP-PCM method to a selection of other peric
6 We then asked subjects to estimate virtual-room size with eit
7 We then characterize Muller glia spatial patterning, revealin
8 We then co-injected (mice, intraocular) unimNPs with the glut
9 To examine the neural locus of this effect,
we then combined the adaptation paradigm with TMS.
10 We then combined these analytical tools with in vivo models o
11 We then compared these culture-independent genomes to existin
12 We then compared trajectories through maps both with and with
13 We then constructed sequential deletions in regions that span
14 We then constructed two RBDs mutated in multiple key residues
15 We then coupled that data with conventional DCB extraction.
16 We then decompose the uncertainty into that associated with f
17 Using a translational approach,
we then demonstrate that human induced Treg cells suppress sy
18 Using these experiments,
we then demonstrate that, in the pentamer, three anaesthetic-
19 We then designed provisional HRCT diagnostic criteria based o
20 We then develop a population-dynamic model quantifying killin
21 We then elaborate the two main future directions that are cen
22 We then evaluate the performance of the best method for the i
23 We then evaluated polymorphisms that showed the strongest ass
24 We then explore approaches to limit immunotoxicity, and discu
25 We then interpret our dissolution data in a framework that in
26 We then investigated the association of DNA methylation with
27 We then investigated the effect of overexpression of human al
28 s residue is just before the gamma-secretase cleavage site,
we then investigated whether the p75(alphagamma) peptide, whi
29 We then made substitution mutants in DnaK residues suggested
30 We then optimized a Gag immunoprecipitation procedure that pe
31 We then performed a pilot clinical study using BiliSpec to me
32 We then performed RNA sequencing to investigate the effect of
33 We then propose a neural model of the self as an emerging pro
34 We then purposely altered water distribution and myofibrils s
35 We then quantify contemporary patterns of shrub, shrub seedli
36 We then realize epitaxial (SrFeO2.5)1/(CaFeO2.5)1 thin film s
37 We then retrospectively compared the patients in this study w
38 We then review evidence that self-related and social consider
39 We then review the few recent studies that document contempor
40 We then screened it for pyrazinamide resistance both in vitro
41 We then simulated the initiation of an entirely LTACH-focused
42 rom C57BL6 mice, with or without the S1P antagonist FTY720;
we then studied HSC mobilization and localization.
43 We then subdivided AAC eyes into four subgroups: crowded-angl
44 We then test the ability of the VPM to respond to spatial pat
45 We then tested 39 patients with severe growth restriction of
46 We then tested our STR capture strategy on P. diadema fecal D
47 We then tested the role of JAK2/STAT3 in ketamine-induced pla
48 We then used cluster analysis to identify patterns of individ
49 We then used statistical simulations, informed by the adminis
50 We then used the signaling diversity results obtained from Al