1 We therefore asked whether cells in the tumour stroma might e
2 We therefore asked whether LPL functions downstream of Mst1.
3 We therefore asked whether neonate-specific mechanisms preven
4 We therefore asked whether OSNs in different locations evolve
5 We therefore asked whether the DNA-binding signature of ERalp
6 We therefore asked whether the functionality of Nodal can be
7 ntal angle at which a vibrissa first touches an object, and
we therefore asked whether this parameter could provide the r
8 We therefore assessed whether pharmacokinetic/pharmacodynamic
9 We therefore assessed whether PSS could predict future MACE i
10 We therefore evaluated whether EGFR, ASAP1, ERBB2 and ERBB4,
11 We therefore examined whether a similar treatment regimen wou
12 We therefore examined whether breastfeeding influences emotio
13 We therefore examined whether EIPC improved patients' coping
14 We therefore examined whether in the murine model of tubercul
15 We therefore examined whether interaction of thrombospondin w
16 We therefore examined whether liverwort and moss species have
17 We therefore examined whether mucosal B cells express GrB and
18 We therefore examined whether pretreatment expression levels
19 We therefore examined whether the selective serotonin reuptak
20 We therefore examined whether transgenic expression of CXCR3
21 We therefore explored whether cone function-structure could b
22 We therefore explored whether DC-derived exosomes can present
23 We therefore explored whether substrates themselves could be
24 We therefore investigated whether IAV infection alters Na,K-A
25 We therefore investigated whether MVs affect SynI distributio
26 We therefore investigated whether MyD88-dependent signaling m
27 We therefore investigated whether myeloid progenitor cells tr
28 We therefore investigated whether nAChR signaling in the amyg
29 We therefore investigated whether NB disruption might be muta
30 We therefore investigated whether or not differences in expre
31 We therefore investigated whether phosphorylation can allow K
32 We therefore investigated whether RyR1 gating behaviour is mo
33 We therefore investigated whether there are corresponding dif
34 We therefore investigated whether Tric-a gene knockout (KO) a
35 We therefore investigated whether vitamin D3 (colecalciferol)
36 We therefore speculated whether increased expression of EBI2
37 hypertrophy and increase strength in our prior murine work;
we therefore tested whether ActRIIB-mFc could improve weaknes
38 We therefore tested whether altered NAc dopamine release acco
39 We therefore tested whether genetic variants in the IL1RL1 di
40 We therefore tested whether induction of islet neogenesis in
41 We therefore tested whether LIGHT might similarly promote col
42 We therefore tested whether other genes/pathways targeted by
43 We therefore tested whether PPARgamma and ERK associated in p
44 We therefore tested whether Retnlbeta (mouse ortholog of huma
45 We therefore tested whether the association of triglyceride-a
46 We therefore tested whether the CKiD Study participants with
47 e at onset and genetic variants in DNA repair pathways, and
we therefore tested whether the modifying effects of variants
48 We therefore tested whether the suppressive effects of HCD on
49 We therefore tested whether vasopressin 1a receptor (V1aR) in
50 In this study
we, therefore, investigated whether metal sulfides that are m