1 We therefore conclude that 604del is blocked in its abil
2 We therefore conclude that a histone modifying enzyme is
3 We therefore conclude that a monomeric bFGF.oligosacchar
4 We therefore conclude that a single event, such as vicar
5 We therefore conclude that a tyrosine kinase-dependent a
6 We therefore conclude that A22 is a competitive inhibito
7 We, therefore, conclude that activation of NFAT in CD4+
8 We therefore conclude that adenosine deficiency may be r
9 We therefore conclude that adenosine, acting via A(1) re
10 We therefore conclude that age-related DNAm changes in p
11 We therefore conclude that AHPC confers acute neuroprote
12 We therefore conclude that AIB1 can exert its oncogenici
13 We therefore conclude that allospecific human CD8+ T cel
14 We therefore conclude that alpha(2)-adrenergic stimulati
15 We therefore conclude that although Hif-1alpha and Hif-2
16 found from this broad white population, and
we therefore conclude that although of great interest, t
17 We therefore conclude that although telomeres are elonga
18 We therefore conclude that,
although induction of Ikappa
19 We, therefore, conclude that an increase in MAP1S levels
20 We therefore conclude that antennal grooming removes exc
21 We therefore conclude that anthropogenic warming is incr
22 We therefore conclude that AP-1 factor is a pivotal regu
23 We therefore conclude that APC-derived TSP-1 suppresses
24 We therefore conclude that ASPM underwent strong adaptiv
25 We therefore conclude that axon regeneration promoted by
26 We therefore conclude that BAG1 is not essential for cys
27 We therefore conclude that benign enhancement of cardiac
28 We therefore conclude that bone marrow toxicity may be m
29 We therefore conclude that both a direct and indirect ro
30 We therefore conclude that both BAF protein structure an
31 We therefore conclude that both low and high affinity in
32 We, therefore, conclude that both CB2-selective activati
33 evoked by pancreatitis-promoting agents and
we therefore conclude that bradykinin plays a role in ac
34 We therefore conclude that Ca2+ mediates cyclin B1 degra
35 We therefore conclude that capsid, not integrase, is the
36 We therefore conclude that CAV-1 functions as a scaffold
37 We therefore conclude that CAV-1 may function as a scaff
38 We, therefore, conclude that CD31-/Sca1+ cardiac SP cell
39 We therefore conclude that CH4 from the Norwegian Sea ve
40 We therefore conclude that clonal expansion rather than
41 We therefore conclude that CO2 alone (without a signific
42 We therefore conclude that cocaine enhances dopamine rel
43 We therefore conclude that column bleed contamination ha
44 We therefore conclude that conserved RNAPII-associated m
45 We therefore conclude that CORVET is a tethering complex
46 We therefore conclude that Cys-456 of ThiI is critical f
47 We therefore conclude that despite the hypoxic nature of
48 We, therefore, conclude that diazoxide-mediated precondi
49 pacity building and technique refinement and
we therefore conclude that diversification of our manage
50 We therefore conclude that DNA concentration is a widely
51 We therefore conclude that DNA-PK-dependent phosphorylat
52 We therefore conclude that DSCAM is not required for Net
53 We therefore conclude that E is indeed a cell wall synth
54 We therefore conclude that E2f4 is required to determine
55 We therefore conclude that Earth's dynamo would survive
56 We therefore conclude that eEF1A glutaminylation is a ye
57 We therefore conclude that endotoxin inhibits enterocyte
58 We therefore conclude that Eph/ephrin interactions guide
59 We therefore conclude that epigenetic regulation plays a
60 We therefore conclude that,
even in the highly unlikely
61 We therefore conclude that FANCC acts in concert with Hs
62 We therefore conclude that FcgammaR-mediated cell activa
63 We, therefore, conclude that following myocardial injury
64 We therefore conclude that for this class I+ class II-tr
65 We therefore conclude that formation of Cys106-sulfinic
66 We therefore conclude that FOXO3a plays an important rol
67 We, therefore, conclude that G6PD is a critical cytosoli
68 We therefore conclude that genomic drift plays an import
69 We therefore conclude that germline ENPP1 cysteine-speci
70 We, therefore, conclude that Glut3 is targeted to secret
71 We therefore conclude that Grg5 enhances Runx2 activity
72 We therefore conclude that H5 is specifically associated
73 We therefore conclude that HACE1 is not directly involve
74 We therefore conclude that HCV+ recipients can safely re
75 We therefore conclude that hemoglobin protects Plasmodiu
76 We therefore conclude that hepatic elimination and renal
77 We therefore conclude that hESCs are capable of generati
78 We therefore conclude that high-order epistasis profound
79 We therefore conclude that HIPK2 is a potential target f
80 We therefore conclude that HNF-4alpha is both essential
81 We, therefore, conclude that hpaECMs has the potential t
82 We therefore concluded that HPV31, and possibly human pa
83 We, therefore, conclude that Hsp90 and Hsc70 bind indepe
84 We therefore conclude that HuMUC18 is an important deter
85 We therefore conclude that hyperhomocysteinemia impairs
86 We therefore conclude that,
if it occurs at all, homolog
87 We therefore concluded that IgE and eosinophils are requ
88 We therefore conclude that IL-1alpha up-regulates cathep
89 We therefore conclude that in a mutant p53 background, p
90 rsely related to epithelial apoptosis scores
we therefore conclude that in patients with nonulcer dys
91 We, therefore, conclude that in these epithelial cells,
92 We therefore conclude that,
in neuronal cells of mice, t
93 We therefore conclude that inhibition of C5 cleavage dur
94 We therefore conclude that intrahost HIV-1 evolution in
95 We therefore conclude that IRF3 specifically regulates I
96 We therefore conclude that ischemia/reperfusion alters t
97 We therefore conclude that isogenicity of clones must be
98 We therefore conclude that kin may influence the progres
99 We therefore conclude that KM-1 represents a new class o
100 We therefore conclude that KSR is an essential upstream
101 We therefore conclude that lack of uPA is associated wit
102 We therefore conclude that lactation, rather than involv
103 We therefore conclude that large primary impacts into a
104 We therefore conclude that loss of functional capillarie
105 We therefore conclude that low rather than high NO bioav
106 We therefore conclude that M. avium subsp. hominissuis i
107 We therefore conclude that MAP1B is expressed in Schwann
108 We therefore conclude that matching for HLA-C should be
109 We, therefore, conclude that mavacamten acts on multiple
110 We therefore conclude that measurements of CT ratio and
111 We therefore conclude that metaxin is a mitochondrial pr
112 We therefore conclude that Mgp gene deletion is what we
113 We therefore conclude that micronuclei represent an impo
114 We therefore conclude that miR-21 is not essential for p
115 We therefore conclude that moist convection-similar to l
116 We therefore conclude that most adaptive mutations are r
117 We therefore conclude that Mrp14 promotes APP processing
118 We therefore conclude that MUC18 expression is increased
119 We therefore conclude that muscle spindles can act as "f
120 We therefore concluded that mutations like K34A disrupte
121 We therefore conclude that MYO9A deficiency may affect t
122 We therefore conclude that Myt1 inhibition of Cyclin A/C
123 We therefore conclude that natural selection is probably
124 We therefore conclude that neocortical plasticity mechan
125 We therefore conclude that NOM1 is a PP1 nucleolar targe
126 We, therefore, conclude that Nox4 is a positive transcri
127 We therefore conclude that NR3A modulates the NR1/NR3A p
128 sonance spectroscopy, and liver biochemistry
We therefore conclude that omega-3 fatty acid supplement
129 We therefore conclude that one or more cdks active from
130 We therefore conclude that optimal functioning of viral
131 We therefore conclude that other unidentified, possibly
132 We, therefore, conclude that P-selectin may anchor ULVWF
133 We therefore conclude that PB is a reliable source for t
134 We therefore conclude that perceived speed is not based
135 We therefore conclude that perturbation of PTEN/Akt sign
136 We therefore conclude that PI3K-ERK1/2 crosstalk plays a
137 We therefore conclude that PKGII is an activator of lung
138 We therefore conclude that PlaB is a virulence factor th
139 ivities as well as their oxidized state, and
we therefore conclude that PlaC likely forms at least on
140 We therefore conclude that PMAT can function as a polysp
141 We therefore conclude that polarization of Rac1 activati
142 We therefore conclude that populations of free charges a
143 We therefore conclude that pre-BCR specificity per se di
144 We therefore conclude that premature termination of host
145 We therefore conclude that proper maturation of cardiac
146 We therefore conclude that PTH1R signaling directly and
147 We therefore conclude that reconstitution of DNA replica
148 We therefore conclude that recurrent apnea produces abno
149 periments have confirmed this prediction and
we, therefore, conclude that relatively small anatomical
150 We therefore conclude that repression of this transcript
151 We therefore conclude that RGS5 is an endogenous regulat
152 We therefore conclude that RP3 activates classical NF-ka
153 We therefore conclude that S. typhimurium fliE is essent
154 We therefore conclude that selectively logged forests ar
155 We therefore conclude that severe ocean acidification mi
156 We therefore conclude that shelterin can protect chromos
157 We, therefore, conclude that signals generated by the co
158 We therefore conclude that singularity regulation by Cdc
159 We therefore conclude that slide selection based upon pr
160 We therefore conclude that Smad2 loss causes HGF upregul
161 We therefore conclude that some PMC, subcoeruleus, and A
162 We therefore conclude that sorcin retains ChREBP in the
163 We therefore conclude that SR is not a general property
164 We therefore conclude that Stat3 plays a crucial role in
165 We therefore conclude that stathmin is required for the
166 We therefore conclude that stimulation of eukaryotic hFE
167 We therefore conclude that stimulation of muscle proteol
168 We therefore conclude that substrate recognition by the
169 We therefore conclude that such a mechanism of hammerhea
170 In contrast to previous assumptions,
we therefore conclude that SYCP1 and SYCP3 form monophyl
171 We therefore conclude that synthetic anion carriers are
172 We therefore conclude that systemic nonselective beta-ad
173 We therefore concluded that targeting chemokines with an
174 We therefore conclude that tgp1(+) is regulated by trans
175 We therefore conclude that the activation of bcl-2 by NF
176 We therefore conclude that the attachment of cells to fi
177 We therefore conclude that the bending mechanics within
178 We therefore conclude that the carboxy terminus of proca
179 We therefore conclude that the correlation in Vbeta expr
180 We therefore conclude that the disparity in NO productio
181 We therefore conclude that the dominant and homogeneous
182 We therefore conclude that the DR3-TL1A pathway regulate
183 We therefore conclude that the endosomal pool of PI3P, g
184 We therefore conclude that the FA/BRCA pathway, while be
185 We therefore conclude that the FRIZZLED2 mutation is a d
186 We therefore conclude that the gain of chromosome 3q tha
187 We therefore conclude that the gata1/pu.1 paradigm for l
188 We therefore conclude that the general applicability of
189 We therefore conclude that the genetic structure of B. b
190 We therefore conclude that the human renin promoter can
191 We therefore conclude that the IIS induces a rapid incre
192 We therefore conclude that the kinetics of V(O(2)) durin
193 We therefore conclude that the Ku80 carboxy terminus is
194 We therefore conclude that the laboratory CXCR4-tropic H
195 We therefore conclude that the mechanics of the bilayer
196 We therefore conclude that the milder reproductive pheno
197 We therefore conclude that the mutant enzyme facilitates
198 We therefore conclude that the NDV-GFP virus could be us
199 We therefore conclude that the Ni hyperaccumulation phen
200 We therefore conclude that the nuclear export of mVP2 is
201 We therefore conclude that the ORF7b protein is not only
202 We therefore conclude that the p2y receptors target only
203 We therefore conclude that the PARP isoforms play non-re
204 We therefore conclude that the phenomenon of male-killin
205 We therefore conclude that the predominant background K-
206 We therefore conclude that the presence of the oxidized
207 We therefore conclude that the pseudogap competes with t
208 We therefore conclude that the RCN1 protein affects over
209 We therefore conclude that the relative size of the floc
210 We therefore conclude that the SDF-1/CXCR4 axis both pre
211 We therefore conclude that the short form of STAT92E is
212 We therefore conclude that the sorting of beta-actin mRN
213 We therefore conclude that the STCs are proteinase inhib
214 We therefore conclude that the strategy humans play is n
215 We therefore conclude that the super-repressor phenotype
216 We therefore conclude that the synapse elimination proce
217 We therefore conclude that the thioredoxin of A. actinom
218 We therefore conclude that the tripartite leader sequenc
219 We therefore conclude that the two defects are caused by
220 We therefore conclude that the two neurotransmitters may
221 We therefore conclude that the U(L)17 gene products are
222 iella pneumoniae, and Pseudomonas aeruginosa
We therefore conclude that the underlying mechanism of e
223 We therefore conclude that the variability of the PrP(Sc
224 We, therefore, conclude that the mode of action of inosi
225 We, therefore, concluded that the interactions of delta-
226 We therefore conclude that therapeutic DCs function as A
227 We therefore conclude that there are two potential epito
228 We therefore conclude that there is a specific associati
229 We therefore conclude that there is no protein with a DN
230 We therefore conclude that these cysteines play a critic
231 We therefore conclude that these emitting species must o
232 We therefore conclude that these siRNAs interfere with t
233 We therefore conclude that these SNPs are not the etiolo
234 We therefore conclude that this beta-sheet structure is
235 We therefore conclude that this newly cloned mouse Muc5b
236 We therefore conclude that this preparation is not suita
237 We therefore conclude that this signal ensures surface t
238 We therefore conclude that,
through the post-transcripti
239 We therefore conclude that Tnfsf4 underlies Ath1 in mice
240 We therefore conclude that translocation of TMPRSS2-ERG
241 We therefore conclude that triptolide causes global tran
242 We therefore conclude that two mechanisms contribute to
243 We therefore conclude that unless super-inhibitory doses
244 We therefore conclude that viable Chlamydia pneumoniae o
245 We therefore conclude that vinculin conformation, as mod
246 We therefore conclude that weak poly(A) sites, regardles
247 We therefore conclude that whitening of natural inputs c
248 We, therefore, conclude that WRN stimulates (CTG)(n) HPR
249 We therefore conclude that Xenopus oocytes may be a usef