1 We therefore propose that 2ME2 attenuates EAE through di
2 We therefore propose that a certain portion of the muscl
3 We therefore propose that a critical aspect of SCA1 path
4 We therefore propose that a developmental timing mechani
5 We therefore propose that a reason that current therapeu
6 We therefore propose that a simple steric exclusion mode
7 eptors are restricted to inhibitory signals;
we therefore propose that a single receptor can have dua
8 We therefore propose that a threshold level of intracell
9 We therefore propose that ABA signaling via inhibition o
10 We, therefore, propose that additional mechanism(s) exis
11 We therefore propose that AdoMet-induced conformational
12 We therefore propose that ADT-2 regulates body size both
13 We therefore propose that Akr1p negatively affects the a
14 We therefore propose that Akt increases coupling of gluc
15 We therefore propose that all accreditation bodies work
16 We therefore propose that although G4-forming sequences
17 We therefore propose that an early proinflammatory state
18 We therefore propose that an important function of nucle
19 We therefore propose that an initial event in stiffness
20 We therefore propose that an Mtw1 complex is required fo
21 We therefore propose that an ordered histone code can pr
22 We therefore propose that ATM and ATR activate an, as ye
23 We therefore propose that ATP is most likely to regulate
24 We therefore propose that autophagy may act, in part, as
25 We therefore propose that backbone rigidity is a fundame
26 We therefore propose that Bax auto-activation amplifies
27 We therefore propose that BBS may not be a single-gene r
28 We therefore propose that Best2 is a HCO3- channel that
29 We therefore propose that binding of p120ctn to microtub
30 We therefore propose that BMP signalling is modulated bo
31 We therefore propose that Bni1p may localize or alter th
32 We, therefore, propose that bone loss in postmenopausal
33 We therefore propose that both pre- and postsynaptic mec
34 We therefore propose that both Saci_0568 and Saci_0748 a
35 We therefore proposed that brief antecedent ischemia/rep
36 We therefore propose that Btn1p is required for tight re
37 We therefore propose that CAP binds the cleft between di
38 We therefore propose that carboxylic acid metabolites of
39 We therefore propose that caveolin may serve as an eNOS
40 We therefore propose that CcmI-1 is part of the CcmF-Ccm
41 We therefore proposed that CDT is a tripartite toxin com
42 We therefore propose that CENP-A induces geometric const
43 We, therefore, propose that CENP-E transports chromosome
44 We therefore propose that centrosome loss or a prolonged
45 We therefore propose that chromosome rigidity plays an e
46 We therefore propose that clinical-grade non-tagged reco
47 We therefore propose that coexpression of CD160 and 2B4
48 We therefore propose that cognitive shifting in primates
49 We therefore propose that combination therapy targeting
50 We therefore propose that contrary to the maladaptive mo
51 We therefore propose that Cox20 binding to Cox2 on the t
52 We therefore propose that CURT1 proteins modify thylakoi
53 We therefore propose that Cx26-mediated release of ATP i
54 We therefore propose that CYREN is a direct cell-cycle-d
55 We therefore propose that D6, which is upregulated in bo
56 We therefore propose that defective mitochondrial protei
57 We therefore propose that defects in cell polarity under
58 We therefore propose that determination of HDL capacity
59 We therefore propose that disordered methylation plays a
60 We therefore propose that distinct perceptual features o
61 We therefore propose that dMyD88 is the functional homol
62 We therefore propose that DNA is a sequence-specific all
63 We therefore propose that drive to the M4/6 neurons refl
64 We therefore propose that during coat assembly, PKC acti
65 We therefore propose that dynamic changes in GR stoichio
66 We therefore propose that dynein/dynactin complexes may
67 nosine, regulate the rundown of swimming and
we therefore proposed that ectonucleotidase proteins are
68 We therefore propose that endocannabinoid modulation of
69 We therefore propose that eplin dephosphorylation by hCD
70 We therefore propose that extreme acidophiles had to rep
71 We therefore propose that FADD haploinsufficiency is lik
72 We therefore propose that FBP and the PTB-like protein m
73 We, therefore, propose that FcgammaRIIb serves to inhibi
74 We, therefore, propose that for a DSB to cause cell cycl
75 We therefore propose that fusion may occur by a DSB-inde
76 We therefore propose that genes regulating brain size du
77 We therefore propose that GLP-1 and exendin-4 are capabl
78 We therefore propose that glutathione facilitates the se
79 We therefore propose that H3K36me3 is the opposing modif
80 We therefore propose that HA hydrogels, with their devel
81 We therefore propose that heterozygosity at the MTL locu
82 We therefore propose that Hir proteins contribute to sil
83 We therefore propose that Hsp70 can be used as a vehicle
84 We therefore propose that human IscA1 plays an important
85 We therefore propose that hydrophobic interactions betwe
86 We therefore propose that in addition to countering p53
87 We therefore propose that in addition to its photorecept
88 We therefore propose that in addition to its role as a m
89 We therefore propose that in addition to their functions
90 We therefore propose that in fusion cells, Shot may func
91 We therefore propose that,
in addition to cytokinin tran
92 We therefore propose that,
in addition to its well estab
93 We therefore propose that,
in REM sleep, endogenously ge
94 We therefore propose that increased DDAH activity and th
95 We therefore proposed that increased reactive oxygen spe
96 We therefore propose that increasing levels of Ikaros an
97 We therefore propose that individual Ypt/Rabs, in the co
98 We therefore propose that inflammation may directly caus
99 We therefore propose that intact P. falciparum-infected
100 We therefore propose that invadolysin plays a crucial ro
101 We therefore propose that invasion is a balance between
102 We therefore propose that Ipl1/Aurora and Ase1 constitut
103 We therefore propose that LET-711 inhibits the expressio
104 We therefore propose that leukemic cell survival depends
105 We therefore propose that Lhs1p is a molecular chaperone
106 We therefore propose that lipid rafts spatially organize
107 We therefore propose that local RNA molecules interactin
108 We therefore propose that localization of a synergistic
109 We therefore propose that mammalian RAD52 facilitates re
110 We therefore propose that MAP kinase is a key regulator
111 We therefore propose that MCA is a useful technique to s
112 We therefore propose that mIHF is a distinct member of a
113 We therefore propose that miR-122* is an important contr
114 We therefore propose that miRNA92a and the PTEN-PI3K-KLF
115 We therefore propose that mitochondrial codon reassignme
116 We therefore propose that mitochondrial dysfunction in p
117 We therefore propose that moire interference between gri
118 We therefore propose that monomethylated H4K20 is involv
119 We therefore propose that MSMEG_5437, MSMEG_6129 and MSM
120 We therefore proposed that mTORC1 drives HIF-1alpha synt
121 We therefore propose that multiple Lig enzymes are neede
122 We therefore propose that muscle amphiphysin is not invo
123 We therefore propose that mutations in the MPV17 gene be
124 We therefore propose that N-cad restricts the response o
125 We therefore propose that N-WASP acts as a positive regu
126 We therefore propose that Nek6 facilitates association o
127 We therefore propose that neural and kinematic variabili
128 We therefore propose that NGAL participates in the antib
129 We therefore proposed that nitric oxide contributes to s
130 We therefore propose that NO differentially regulates th
131 We therefore propose that Notch signaling activates at l
132 We therefore propose that nucleoid compaction is part of
133 We therefore propose that obesity is an FGF21-resistant
134 We therefore propose that oligomeric forms of defensin,
135 We therefore propose that oligomerization of IP-10 is re
136 We therefore propose that oropharyngeal T cells activate
137 We therefore propose that osmosensing relies on a confor
138 We, therefore, propose that oxidation serves as a switch
139 We therefore propose that PGI(2)-mediated upregulation o
140 We therefore propose that PGI2-mediated upregulation of
141 We therefore propose that phosphorylation alone may be s
142 We therefore propose that PKA phosphorylation of Ci is r
143 We therefore propose that polyphosphate is a natural cof
144 We therefore propose that PP1 activity silences the chec
145 We, therefore, propose that PRAS40 regulates mTORC1 kina
146 We therefore propose that Rac1 and Nck cause dissociatio
147 ally rescued the circadian phenotype in vivo
We therefore propose that RAS2 acts via a cAMP-dependent
148 We therefore propose that rather than acting as an antio
149 We therefore propose that Rb and p27 cooperate to suppre
150 We therefore propose that RCP functions primarily as a R
151 We therefore propose that repression of multiple PIP3 de
152 We therefore propose that reproductive traits should be
153 We therefore propose that resveratrol, genistein, and ba
154 We therefore propose that Rhino anchors a nuclear comple
155 We therefore propose that S2 is necessary for angiogenes
156 the NPL in organisms in other environments;
we therefore propose that sand-swimming could be an exce
157 We therefore propose that SAR1376, and perhaps other mem
158 We therefore propose that Sens acts as a binary switch t
159 We therefore propose that separate circuits within the m
160 We therefore propose that sequential degradation ensures
161 We therefore propose that Siva-1 or its SAH region can b
162 We therefore propose that small molecule enhancers of au
163 We therefore propose that sublimation from millimetre-si
164 p was not stably maintained or inherited and
we therefore propose that Sum1-1p-mediated long-range re
165 We therefore propose that SUMO serves as a mark for nonf
166 We therefore propose that surface detachment of membrane
167 We therefore propose that targeted anticancer therapies
168 We therefore propose that the 8q24 risk loci operate thr
169 We therefore propose that the ability of dynamin to alte
170 We therefore propose that the absence of primer at the P
171 We therefore propose that the ACC/Lys(DNP) pair can be c
172 We therefore propose that the activities of specific ele
173 We therefore propose that the AID/APOBEC family of deami
174 We therefore propose that the beta-carbolines harmane an
175 We therefore propose that the Bub3-BubR1 complex on brok
176 We therefore propose that the carboxy-terminal helicase
177 We therefore propose that the changes in higher order ch
178 We therefore propose that the core elements must product
179 ed to detect similar proteins in PLASMODIUM:
We therefore propose that the COWP family of proteins ha
180 We therefore propose that the current consensus of uPA-c
181 We therefore propose that the cystatin E/M suppressor ge
182 We therefore propose that the D351Y mutation allows weak
183 We therefore propose that the DBC1-SIRT1 interaction may
184 We therefore propose that the default paradigm be change
185 We therefore propose that the default role for DC in the
186 We therefore propose that the early embryo environment r
187 We therefore propose that the enhanced photoelectrochemi
188 We therefore propose that the fetal epicardium, in respo
189 We therefore propose that the function of the flip-flop
190 We therefore propose that the gene encoding LTW4 be call
191 We therefore propose that the HAT-7 cell line is a usefu
192 We therefore propose that the hydrophobic patch in the P
193 We therefore propose that the inability of DP thymocytes
194 We therefore propose that the induction of elevated TAG1
195 We therefore propose that the interplay between elastici
196 We therefore propose that the metabolic activation of HN
197 philin has a reduced affinity for membranes;
we therefore propose that the modulation of the membrane
198 We therefore propose that the motor neuron-to-oligodendr
199 We therefore propose that the MT-LOOP region is an inter
200 We therefore propose that the NarQ sensor-transmitter pr
201 We therefore propose that the nonconventional hydrogen b
202 We therefore propose that the ocean introduces a memory
203 We therefore propose that the outward-occluded state rep
204 We therefore propose that the overexpression of Glc7 reg
205 We therefore propose that the pathology characterizing M
206 We therefore propose that the pIV multimer undergoes a l
207 We therefore propose that the PKA requirement is upstrea
208 We therefore propose that the postmitotic nuclear envelo
209 We therefore propose that the previously demonstrated er
210 We therefore propose that the primary abnormality in TRM
211 We therefore propose that the production of TSP1 at late
212 We therefore propose that the R2 domain can interact wit
213 We therefore propose that the restriction of oscillation
214 We therefore propose that the role of ASIC1a in LTP and
215 We therefore propose that the rs2 gene product acts to r
216 We therefore propose that the Seven-up-initiated Imp/Syp
217 We therefore propose that the single epithelial cilium i
218 We therefore propose that the speciation of Zn in peat d
219 We therefore propose that the synergistic stimulation of
220 We therefore propose that the TGF-beta system is up-regu
221 We therefore propose that the TNFR-I-ERK1/2 pathway play
222 We therefore propose that the transcription-mediated evi
223 We therefore propose that the transcriptional dynamics o
224 We therefore propose that the well-known EphA4-dependent
225 We therefore propose that the ZLI is induced within the
226 We, therefore, propose that the reporter gene always act
227 We therefore propose that there are significant genetic
228 We therefore propose that these effects relate to the in
229 We therefore propose that these ER chaperones complex to
230 We, therefore, propose that they should be designated na
231 We therefore propose that this case of vitamin D resista
232 We therefore propose that this experimental paradigm cou
233 We therefore propose that this repression and MSCI itsel
234 We therefore propose that this simple, accurate PCR appr
235 We therefore propose that this virally derived mutation
236 We therefore proposed that TIF2.0 binding to AF1 changes
237 We therefore propose that Tim23p is imported into mitoch
238 We therefore propose that TRIM5alpha proteins have evolv
239 We therefore propose that tub may be associated with syn
240 We therefore propose that TULA-2 is the primary tyrosine
241 We therefore propose that UAP56 and Vasa function in a p
242 We therefore propose that ubiquitination of the EpoR cri
243 We therefore propose that under certain genetic conditio
244 We therefore propose that under normal conditions a seco
245 We therefore propose that unexpectedly pVHL, via the deg
246 We therefore propose that variants in CMIP and ATP2C2 ac
247 We therefore propose that VEGF acutely acts to increase
248 We therefore propose that Vps4 proteins cycle between so
249 We therefore propose that Wnt signaling to beta-catenin
250 We therefore propose that yeast kinetochore assembly req
251 We therefore propose that ylgG be redesignated as folQ.
252 We therefore propose that ZRANB3 maintains genomic stabi